Glucocorticoid receptors recruit the CaMKIIα-BDNF-CREB pathways to mediate memory consolidation. - Wikidata - wikimedia - TriplyDB

Glucocorticoid receptors recruit the CaMKIIα-BDNF-CREB pathways to mediate memory consolidation.

Glucocorticoid receptors recruit the CaMKIIα-BDNF-CREB pathways to mediate memory consolidation.

http://www.wikidata.org/entity/Q36431262

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Linking Mitochondria to Synapses: New Insights for Stress-Related Neuropsychiatric Disorders The Role of Lactate-Mediated Metabolic Coupling between Astrocytes and Neurons in Long-Term Memory Formation Protein degradation and protein synthesis in long-term memory formation.The neurobiological bases of memory formation: from physiological conditions to psychopathology Tinospora cordifolia ameliorates anxiety-like behavior and improves cognitive functions in acute sleep deprived rats Down-regulation of serum gonadotropins but not estrogen replacement improves cognition in aged-ovariectomized 3xTg AD female mice Infantile amnesia reflects a developmental critical period for hippocampal learning.Administration of a Histone Deacetylase Inhibitor into the Basolateral Amygdala Enhances Memory Consolidation, Delays Extinction, and Increases Hippocampal BDNF Levels.Additive gene-environment effects on hippocampal structure in healthy humans.A positive autoregulatory BDNF feedback loop via C/EBPβ mediates hippocampal memory consolidation.Physical exercise ameliorates the reduction of neural stem cell, cell proliferation and neuroblast differentiation in senescent mice induced by D-galactose Challenging oneself intermittently to improve health.Membrane-initiated non-genomic signaling by estrogens in the hypothalamus: cross-talk with glucocorticoids with implications for behavior.Minocycline upregulates cyclic AMP response element binding protein and brain-derived neurotrophic factor in the hippocampus of cerebral ischemia rats and improves behavioral deficits Glucocorticoid actions on synapses, circuits, and behavior: implications for the energetics of stress.SKF83959 produces antidepressant effects in a chronic social defeat stress model of depression through BDNF-TrkB pathway.Adrenal fasciculata cells express T-type and rapidly and slowly activating L-type Ca2+ channels that regulate cortisol secretion VGF and Its C-Terminal Peptide TLQP-62 Regulate Memory Formation in Hippocampus via a BDNF-TrkB-Dependent Mechanism.The mitochondrial uncoupler DNP triggers brain cell mTOR signaling network reprogramming and CREB pathway up-regulation Translational Approaches Targeting Reconsolidation.Glucocorticoid Homeostasis in the Dentate Gyrus Is Essential for Opiate Withdrawal-Associated Memories.Combination of early and delayed ischemic postconditioning enhances brain-derived neurotrophic factor production by upregulating the ERK-CREB pathway in rats with focal ischemia.From Memory Impairment to Posttraumatic Stress Disorder-Like Phenotypes: The Critical Role of an Unpredictable Second Traumatic Experience.Neurotrophic-priming of glucocorticoid receptor signaling is essential for neuronal plasticity to stress and antidepressant treatment mTOR is essential for corticosteroid effects on hippocampal AMPA receptor function and fear memory CaMKII regulates proteasome phosphorylation and activity and promotes memory destabilization following retrieval.Phosphatidylinositol 3-Kinase Couples Localised Calcium Influx to Activation of Akt in Central Nerve Terminals Interindividual Variability in Stress Susceptibility: A Role for Epigenetic Mechanisms in PTSD.CCAAT enhancer binding protein δ plays an essential role in memory consolidation and reconsolidation The Omega-3 Polyunsaturated Fatty Acid Docosahexaenoic Acid (DHA) Reverses Corticosterone-Induced Changes in Cortical Neurons Ca2+ and K+ channels of normal human adrenal zona fasciculata cells: properties and modulation by ACTH and AngII.The role of the glucocorticoids in developing resilience to stress and addiction.Astrocytic β2-adrenergic receptors mediate hippocampal long-term memory consolidation.Pathways to neurodegeneration: mechanistic insights from GWAS in Alzheimer's disease, Parkinson's disease, and related disorders Downregulation of miR-219 enhances brain-derived neurotrophic factor production in mouse dorsal root ganglia to mediate morphine analgesic tolerance by upregulating CaMKIIγ.Deletion of Neurotrophin Signaling through the Glucocorticoid Receptor Pathway Causes Tau Neuropathology.Changes in mitochondrial function are pivotal in neurodegenerative and psychiatric disorders: how important is BDNF?Stress and glucocorticoid receptor-dependent mechanisms in long-term memory: from adaptive responses to psychopathologies BDNF Variants May Modulate Long-Term Visual Memory Performance in a Healthy Cohort.Growth factor signaling and memory formation: temporal and spatial integration of a molecular network.

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Glucocorticoid receptors recruit the CaMKIIα-BDNF-CREB pathways to mediate memory consolidation.

http://www.wikidata.org/entity/Q36431262

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2012 nî lūn-bûn

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Glucocorticoid receptors recru ...... mediate memory consolidation.

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Glucocorticoid receptors recru ...... mediate memory consolidation.

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Glucocorticoid receptors recru ...... mediate memory consolidation.

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Glucocorticoid receptors recru ...... mediate memory consolidation.

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Glucocorticoid receptors recru ...... mediate memory consolidation.

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Glucocorticoid receptors recru ...... mediate memory consolidation.

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Cristina M Alberini

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Dhananjay Bambah-Mukku

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Dillon Y Chen

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Gabriella Pollonini

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P2860

A phospho-switch controls the dynamic association of synapsins with synaptic vesicles

The molecular basis of CaMKII function in synaptic and behavioural memory

Physiology and neurobiology of stress and adaptation: central role of the brain

Persistence of long-term memory storage requires a late protein synthesis- and BDNF- dependent phase in the hippocampus

A critical role for IGF-II in memory consolidation and enhancement.

Transcription factors in long-term memory and synaptic plasticity

BDNF-triggered events in the rat hippocampus are required for both short- and long-term memory formation

MuSK expressed in the brain mediates cholinergic responses, synaptic plasticity, and memory formation

Glucocorticoids and hippocampal atrophy in neuropsychiatric disorders.

The stress hormone corticosterone increases synaptic alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptors via serum- and glucocorticoid-inducible kinase (SGK) regulation of the GDI-Rab4 complex

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12

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3509234

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