Role of apoptosis in the pathogenesis of COPD and pulmonary emphysema - Wikidata - wikimedia - TriplyDB

Role of apoptosis in the pathogenesis of COPD and pulmonary emphysema

Role of apoptosis in the pathogenesis of COPD and pulmonary emphysema

http://www.wikidata.org/entity/Q36434963

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The effects of carbon nanotubes on lung and dermal cellular behaviors Nitrosative stress inhibits the aminophospholipid translocase resulting in phosphatidylserine externalization and macrophage engulfment Bronchoscopic lung volume reduction procedures for chronic obstructive pulmonary disease Pathogenic triad in COPD: oxidative stress, protease-antiprotease imbalance, and inflammation COPD immunopathology The role of airway macrophages in apoptotic cell clearance following acute and chronic lung inflammation Impact of COPD and emphysema on survival of patients with lung cancer: A meta-analysis of observational studies Chronic obstructive pulmonary disease Impaired flow-mediated dilation is associated with low pulmonary function and emphysema in ex-smokers: the Emphysema and Cancer Action Project (EMCAP) Study Lung injury after cigarette smoking is particle related Molecular and cellular mechanism of lung injuries due to exposure to sulfur mustard: a review Scale dependence of structure-function relationship in the emphysematous mouse lung.Utilization of the 1,2,3,5-thiatriazolidin-3-one 1,1-dioxide scaffold in the design of potential inhibitors of human neutrophil proteinase 3.Flavored little cigar smoke induces cytotoxicity and apoptosis in airway epithelia.Hypoxia-induced deoxycytidine kinase expression contributes to apoptosis in chronic lung disease.Mechanism-based inhibitors of serine proteases with high selectivity through optimization of S' subsite binding.Mechanistic understanding of nanoparticles' interactions with extracellular matrix: the cell and immune system Deregulation of apoptosis mediators' p53 and bcl2 in lung tissue of COPD patients.Macrophage migration inhibitory factor is a novel determinant of cigarette smoke-induced lung damage.Percent emphysema, airflow obstruction, and impaired left ventricular filling.Circulating hematopoietic progenitor cells are decreased in COPD.Human CD56+ cytotoxic lung lymphocytes kill autologous lung cells in chronic obstructive pulmonary disease Increased expression of beta-defensin 1 (DEFB1) in chronic obstructive pulmonary disease.Inhibitors of human neutrophil elastase based on a highly functionalized N-amino-4-imidazolidinone scaffold Granzymes in age-related cardiovascular and pulmonary diseases.Lung injury and cancer: Mechanistic insights into ceramide and EGFR signaling under cigarette smoke.Effects of structure on inhibitory activity in a series of mechanism-based inhibitors of human neutrophil elastase.Altered gene expression in blood and sputum in COPD frequent exacerbators in the ECLIPSE cohort.Longitudinal study of spatially heterogeneous emphysema progression in current smokers with chronic obstructive pulmonary disease Lung injury and lung cancer caused by cigarette smoke-induced oxidative stress: Molecular mechanisms and therapeutic opportunities involving the ceramide-generating machinery and epidermal growth factor receptor.Histone deacetylase (HDAC) 1 controls the expression of beta defensin 1 in human lung epithelial cells.Resveratrol exerts an anti-apoptotic effect on human bronchial epithelial cells undergoing cigarette smoke exposure.Changes in airway histone deacetylase2 in smokers and COPD with inhaled corticosteroids: a randomized controlled trial p53 mediates cigarette smoke-induced apoptosis of pulmonary endothelial cells: inhibitory effects of macrophage migration inhibitor factor Neutral sphingomyelinase 2: a novel target in cigarette smoke-induced apoptosis and lung injury.Retinoic acid receptor alpha: One of plasma biomarkers associated with exacerbation of chronic obstructive pulmonary disease.Oxidative DNA damage in lung tissue from patients with COPD is clustered in functionally significant sequences.Non-essential role for TLR2 and its signaling adaptor Mal/TIRAP in preserving normal lung architecture in mice Critical role of CFTR-dependent lipid rafts in cigarette smoke-induced lung epithelial injury.Expression and methylation of mitochondrial transcription factor a in chronic obstructive pulmonary disease patients with lung cancer.

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Role of apoptosis in the pathogenesis of COPD and pulmonary emphysema

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2006年學術文章

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2006年學術文章

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Role of apoptosis in the pathogenesis of COPD and pulmonary emphysema

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Role of apoptosis in the pathogenesis of COPD and pulmonary emphysema

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type

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Role of apoptosis in the pathogenesis of COPD and pulmonary emphysema

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Role of apoptosis in the pathogenesis of COPD and pulmonary emphysema

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Role of apoptosis in the pathogenesis of COPD and pulmonary emphysema

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Role of apoptosis in the pathogenesis of COPD and pulmonary emphysema

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Respiratory Research

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Role of apoptosis in the pathogenesis of COPD and pulmonary emphysema

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P2093

Guy F Joos

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Guy G Brusselle

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Ingel K Demedts

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Tine Demoor

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P2860

Ordering the cytochrome c-initiated caspase cascade: hierarchical activation of caspases-2, -3, -6, -7, -8, and -10 in a caspase-9-dependent manner

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

Growth factors can influence cell growth and survival through effects on glucose metabolism

Elastase-mediated phosphatidylserine receptor cleavage impairs apoptotic cell clearance in cystic fibrosis and bronchiectasis

Caspases are activated in a branched protease cascade and control distinct downstream processes in Fas-induced apoptosis

Marked alveolar apoptosis/proliferation imbalance in end-stage emphysema

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apoptotic process

chronic obstructive pulmonary disease

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