Mucin (Muc) expression during pancreatic cancer progression in spontaneous mouse model: potential implications for diagnosis and therapy.
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Novel agents for advanced pancreatic cancerMucins in neoplasms of pancreas, ampulla of Vater and biliary systemNovel pancreatic cancer cell lines derived from genetically engineered mouse models of spontaneous pancreatic adenocarcinoma: applications in diagnosis and therapyNovel role of pancreatic differentiation 2 in facilitating self-renewal and drug resistance of pancreatic cancer stem cells.PD2/Paf1 depletion in pancreatic acinar cells promotes acinar-to-ductal metaplasia.Nuclear receptors and pathogenesis of pancreatic cancer.Spatiotemporal proteomic analyses during pancreas cancer progression identifies serine/threonine stress kinase 4 (STK4) as a novel candidate biomarker for early stage disease.Constitutively active Akt1 cooperates with KRas(G12D) to accelerate in vivo pancreatic tumor onset and progression.Targeting EGF-receptor(s) - STAT1 axis attenuates tumor growth and metastasis through downregulation of MUC4 mucin in human pancreatic cancerNCOA3-mediated upregulation of mucin expression via transcriptional and post-translational changes during the development of pancreatic cancer.Genetically engineered mucin mouse models for inflammation and cancer.Novel HER3/MUC4 oncogenic signaling aggravates the tumorigenic phenotypes of pancreatic cancer cellsQuantitative Proteomic Analysis of Differentially Expressed Protein Profiles Involved in Pancreatic Ductal AdenocarcinomaChanges in microRNA (miRNA) expression during pancreatic cancer development and progression in a genetically engineered KrasG12D;Pdx1-Cre mouse (KC) modelIntegrated glycomic analysis of ovarian cancer side population cells.Potential applications of nanotechnology for the diagnosis and treatment of pancreatic cancerOptimum chemotherapy in the management of metastatic pancreatic cancerMUC4 mucin- a therapeutic target for pancreatic ductal adenocarcinoma.MUC5AC interactions with integrin β4 enhances the migration of lung cancer cells through FAK signaling.Carboxyl-terminal domain of MUC16 imparts tumorigenic and metastatic functions through nuclear translocation of JAK2 to pancreatic cancer cells.ATM-deficiency increases genomic instability and metastatic potential in a mouse model of pancreatic cancerHypoxia-induced oxidative stress promotes MUC4 degradation via autophagy to enhance pancreatic cancer cells survivalBile acids-mediated overexpression of MUC4 via FAK-dependent c-Jun activation in pancreatic cancer.Apolipoprotein A-I mimetic peptide 4F suppresses tumor-associated macrophages and pancreatic cancer progression.Ramifications of Secreted Mucin MUC5AC in Malignant Journey: A Holistic View.
P2860
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P2860
Mucin (Muc) expression during pancreatic cancer progression in spontaneous mouse model: potential implications for diagnosis and therapy.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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name
Mucin (Muc) expression during ...... ons for diagnosis and therapy.
@ast
Mucin (Muc) expression during ...... ons for diagnosis and therapy.
@en
type
label
Mucin (Muc) expression during ...... ons for diagnosis and therapy.
@ast
Mucin (Muc) expression during ...... ons for diagnosis and therapy.
@en
prefLabel
Mucin (Muc) expression during ...... ons for diagnosis and therapy.
@ast
Mucin (Muc) expression during ...... ons for diagnosis and therapy.
@en
P2093
P2860
P356
P1476
Mucin (Muc) expression during ...... ions for diagnosis and therapy
@en
P2093
Dhanya Haridas
Maneesh Jain
María P Torres
Michael Baine
Moorthy P Ponnusamy
Muzafar A Macha
Parama Dey
Satyanarayana Rachagani
Sonny L Johansson
Sukhwinder Kaur
P2860
P2888
P356
10.1186/1756-8722-5-68
P577
2012-10-26T00:00:00Z
P5875
P6179
1026144786