The LMP1 oncogene of EBV activates PERK and the unfolded protein response to drive its own synthesis. - Wikidata - wikimedia - TriplyDB

The LMP1 oncogene of EBV activates PERK and the unfolded protein response to drive its own synthesis.

The LMP1 oncogene of EBV activates PERK and the unfolded protein response to drive its own synthesis.

http://www.wikidata.org/entity/Q36445900

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Hepatitis C Virus Infection Induces Autophagy as a Prosurvival Mechanism to Alleviate Hepatic ER-Stress Response Oncogenes and RNA splicing of human tumor viruses Viruses and autophagy Arms Race between Enveloped Viruses and the Host ERAD Machinery Modulation of the Translational Landscape During Herpesvirus Infection Mitochondria in the spotlight of aging and idiopathic pulmonary fibrosis Augmented latent membrane protein 1 expression from Epstein-Barr virus episomes with minimal terminal repeats Micro RNAs of Epstein-Barr virus promote cell cycle progression and prevent apoptosis of primary human B cells.Either ZEB1 or ZEB2/SIP1 can play a central role in regulating the Epstein-Barr virus latent-lytic switch in a cell-type-specific manner.Aggressive large B-cell lymphoma with plasma cell differentiation: immunohistochemical characterization of plasmablastic lymphoma and diffuse large B-cell lymphoma with partial plasmablastic phenotype The latent membrane protein 1 (LMP1) oncogene of Epstein-Barr virus can simultaneously induce and inhibit apoptosis in B cells.Viral oncogenes, noncoding RNAs, and RNA splicing in human tumor viruses The p17 nonstructural protein of avian reovirus triggers autophagy enhancing virus replication via activation of phosphatase and tensin deleted on chromosome 10 (PTEN) and AMP-activated protein kinase (AMPK), as well as dsRNA-dependent protein kinas Lymphomas differ in their dependence on Epstein-Barr virus Amino acid residues 489-503 of dihydropyridine receptor (DHPR) β1a subunit are critical for structural communication between the skeletal muscle DHPR complex and type 1 ryanodine receptor Cytomegalovirus downregulates IRE1 to repress the unfolded protein response.LMP1 association with CD63 in endosomes and secretion via exosomes limits constitutive NF-κB activation Bortezomib induction of C/EBPβ mediates Epstein-Barr virus lytic activation in Burkitt lymphoma.Evaluation of Epstein-Barr virus latent membrane protein 2 specific T-cell receptors driven by T-cell specific promoters using lentiviral vector LMP1-deficient Epstein-Barr virus mutant requires T cells for lymphomagenesis.Herpesviruses and autophagy: catch me if you can!The role of autophagy in intracellular pathogen nutrient acquisition.Murine cytomegalovirus targets transcription factor ATF4 to exploit the unfolded-protein response An Epstein-Barr Virus (EBV) mutant with enhanced BZLF1 expression causes lymphomas with abortive lytic EBV infection in a humanized mouse model.Metabolic stress is a barrier to Epstein-Barr virus-mediated B-cell immortalization.Epstein-Barr virus maintains lymphomas via its miRNAs.Fluorescence Resonance Energy Transfer-based Structural Analysis of the Dihydropyridine Receptor α1S Subunit Reveals Conformational Differences Induced by Binding of the β1a Subunit.The unfolded protein response and autophagy: herpesviruses rule!Phosphorylation sites of Epstein-Barr virus EBNA1 regulate its function Modulation of autophagy-like processes by tumor viruses.How human tumor viruses make use of autophagy.Autophagy in viral replication and pathogenesis.When autophagy meets viruses: a double-edged sword with functions in defense and offense.Epstein-barr virus and the pathogenesis of T and NK lymphoma: a mystery unsolved.Modulation of the autophagy pathway by human tumor viruses.A new paradigm: innate immune sensing of viruses via the unfolded protein response.Cellular unfolded protein response against viruses used in gene therapy The expanding roles of endoplasmic reticulum stress in virus replication and pathogenesis.Nuclear factor-kappaB binds to the Epstein-Barr Virus LMP1 promoter and upregulates its expression The p38 signaling pathway upregulates expression of the Epstein-Barr virus LMP1 oncogene.

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The LMP1 oncogene of EBV activates PERK and the unfolded protein response to drive its own synthesis.

http://www.wikidata.org/entity/Q36445900

description

2007 nî lūn-bûn

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P2860

Mammalian transcription factor ATF6 is synthesized as a transmembrane protein and activated by proteolysis in response to endoplasmic reticulum stress

Plasma cell differentiation requires the transcription factor XBP-1

Mammalian eukaryotic initiation factor 2 alpha kinases functionally substitute for GCN2 protein kinase in the GCN4 translational control mechanism of yeast

TRAF5, a novel tumor necrosis factor receptor-associated factor family protein, mediates CD40 signaling

The Epstein-Barr virus oncogene product latent membrane protein 1 engages the tumor necrosis factor receptor-associated death domain protein to mediate B lymphocyte growth transformation and activate NF-kappaB

EBNA-1, a bifunctional transcriptional activator

The EBV nuclear antigen 1 (EBNA1) enhances B cell immortalization several thousandfold

Human cytomegalovirus infection activates and regulates the unfolded protein response.

The luminal domain of ATF6 senses endoplasmic reticulum (ER) stress and causes translocation of ATF6 from the ER to the Golgi

Protein translation and folding are coupled by an endoplasmic-reticulum-resident kinase

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