Short term triiodo-L-thyronine treatment inhibits cardiac myocyte apoptosis in border area after myocardial infarction in rats. - Wikidata - wikimedia - TriplyDB

Short term triiodo-L-thyronine treatment inhibits cardiac myocyte apoptosis in border area after myocardial infarction in rats.

Short term triiodo-L-thyronine treatment inhibits cardiac myocyte apoptosis in border area after myocardial infarction in rats.

http://www.wikidata.org/entity/Q36449314

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Hypothyroidism and its rapid correction alter cardiac remodeling Safe Oral Triiodo-L-Thyronine Therapy Protects from Post-Infarct Cardiac Dysfunction and Arrhythmias without Cardiovascular Adverse Effects Supplementation of T3 recovers hypothyroid rat liver cells from oxidatively damaged inner mitochondrial membrane leading to apoptosis Regulation of gene expression with thyroid hormone in rats with myocardial infarction Longstanding hyperthyroidism is associated with normal or enhanced intrinsic cardiomyocyte function despite decline in global cardiac function.Thyroid hormone reverses aging-induced myocardial fatty acid oxidation defects and improves the response to acutely increased afterload.Post-myocardial infarction left ventricular myocyte remodeling: are there gender differences in rats?Evaluation of Cardioprotective Effect of 3,5,3'-Tri-iodo-L-thyronine in Isoproterenol-Induced Cardiotoxicity Thyroid hormone induces sprouting angiogenesis in adult heart of hypothyroid mice through the PDGF-Akt pathway.Improvement of left ventricular remodeling after myocardial infarction with eight weeks L-thyroxine treatment in rats Effects of valsartan on ventricular arrhythmia induced by programmed electrical stimulation in rats with myocardial infarction.Myocardial infarction does not affect fatty-acid profiles in rats.Early long-term L-T3 replacement rescues mitochondria and prevents ischemic cardiac remodelling in rats.Signaling mechanisms in thyroid hormone-induced cardiac hypertrophy.New insights into the role of thyroid hormone in cardiac remodeling: time to reconsider?Acute myocardial infarction and thyroid function: new pathophysiological and therapeutic perspectives.Thyroid hormone and cardiac repair/regeneration: from Prometheus myth to reality?The emerging role of TRα1 in cardiac repair: potential therapeutic implications.Recurrence of differentiated thyroid carcinoma during full TSH suppression: is the tumor now thyroid hormone dependent?Restoration of thyroid hormone balance: a game changer in the treatment of heart failure?Role of thyroid hormones in ventricular remodeling.Translating thyroid hormone effects into clinical practice: the relevance of thyroid hormone receptor α1 in cardiac repair.Mitochondria as key targets of cardioprotection in cardiac ischemic disease: role of thyroid hormone triiodothyronine.Association of serum HMGB2 level with MACE at 1 mo of myocardial infarction: Aggravation of myocardial ischemic injury in rats by HMGB2 via ROS.Cardioprotection and thyroid hormones.Thyroid Hormone and Cardioprotection.New insights into mechanisms of cardioprotection mediated by thyroid hormones.Early and short-term triiodothyronine supplementation prevents adverse post-ischemic cardiac remodeling: role of transforming growth factor-β1 and anti-fibrotic miRNA signaling.Thyroid hormone and tissue repair: new tricks for an old hormone?Thyroid hormone and cardiac disease: from basic concepts to clinical application.Modified Low-Dose Triiodo-L-thyronine Therapy Safely Improves Function Following Myocardial Ischemia-Reperfusion Injury.Roles and controls of mTOR in the heart. Focus on "mTOR attenuates the inflammatory response in cardiomyocytes and prevents cardiac dysfunction in pathological hypertrophy".Low T3 State Is Correlated with Cardiac Mitochondrial Impairments after Ischemia Reperfusion Injury: Evidence from a Proteomic Approach.Inhibition of thyroid hormone receptor α1 impairs post-ischemic cardiac performance after myocardial infarction in mice.Cardiac ischemia/reperfusion injury is inversely affected by thyroid hormones excess or deficiency in male Wistar rats.Impact of thyroid hormone administration on fluid requirements and hepatic injury markers in hemorrhagic shock due to liver trauma.Dose-dependent effects of thyroid hormone on post-ischemic cardiac performance: potential involvement of Akt and ERK signalings.Changes in left ventricular function and remodeling after myocardial infarction in hypothyroid rats.

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P2860

Short term triiodo-L-thyronine treatment inhibits cardiac myocyte apoptosis in border area after myocardial infarction in rats.

http://www.wikidata.org/entity/Q36449314

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Short term triiodo-L-thyronine ...... myocardial infarction in rats.

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Short term triiodo-L-thyronine ...... myocardial infarction in rats.

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Short term triiodo-L-thyronine ...... myocardial infarction in rats.

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Short term triiodo-L-thyronine ...... myocardial infarction in rats.

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A Martin Gerdes

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Jinghai Chen

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Qiangrong Liang

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Rebecca A Redetzke

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Satoru Kobayashi

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Suleman Said

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Yue-Feng Chen

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P2860

Growth retardation and increased apoptosis in mice with homozygous disruption of the Akt1 gene

Control of stress-dependent cardiac growth and gene expression by a microRNA

Thyroid hormone regulation of apoptosis induced by retinoic acid in promyeloleukemic HL-60 cells: studies with retinoic acid receptor-specific and retinoid x receptor-specific ligands.

Progressive left ventricular remodeling and apoptosis late after myocardial infarction in mouse heart.

Cardiac-specific elevations in thyroid hormone enhance contractility and prevent pressure overload-induced cardiac dysfunction.

Thyronin treatment in adult and pediatric heart surgery: clinical experience and review of the literature.

Akt and PI 3-kinase signaling in cardiomyocyte hypertrophy and survival.

The role of phosphoinositide-3 kinase and PTEN in cardiovascular physiology and disease.

Left ventricular performance and remodeling in rabbits after myocardial infarction. Effects of a thyroid hormone analogue.

Akt2 regulates cardiac metabolism and cardiomyocyte survival.

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