The Mad side of the Max network: antagonizing the function of Myc and more.
about
Role of MXD3 in proliferation of DAOY human medulloblastoma cellsSin3b interacts with Myc and decreases Myc levelsEpstein-Barr virus nuclear antigen 3C interacts with and enhances the stability of the c-Myc oncoproteinMultiple basic helix-loop-helix proteins regulate expression of the ENO1 gene of Saccharomyces cerevisiae.Deep sequencing of MYC DNA-binding sites in Burkitt lymphomaCausal reasoning identifies mechanisms of sensitivity for a novel AKT kinase inhibitor, GSK690693.Distinct and temporal roles of nucleosomal remodeling and histone deacetylation in the repression of the hTERT gene.TGFβ1 enhances MAD1 expression and stimulates promoter-bound Pol II phosphorylation: basic functions of C/EBP, SP and SMAD3 transcription factorsMYC Deregulation in Primary Human Cancers.The Max b-HLH-LZ can transduce into cells and inhibit c-Myc transcriptional activities.Sensitivity of malignant peripheral nerve sheath tumor cells to TRAIL is augmented by loss of NF1 through modulation of MYC/MAD and is potentiated by curcumin through induction of ROS.Reverse engineering the neuroblastoma regulatory network uncovers MAX as one of the master regulators of tumor progression.Evolution of the Max and Mlx networks in animals.A C. elegans Myc-like network cooperates with semaphorin and Wnt signaling pathways to control cell migration.Mxi1-0 regulates the growth of human umbilical vein endothelial cells through extracellular signal-regulated kinase 1/2 (ERK1/2) and interleukin-8 (IL-8)-dependent pathways.Drosophila growth and development in the absence of dMyc and dMnt.Activation of PI3K/Akt and MAPK pathways regulates Myc-mediated transcription by phosphorylating and promoting the degradation of Mad1.The tumor suppressor activity of IKKalpha in stratified epithelia is exerted in part via the TGF-beta antiproliferative pathwayMyc's broad reach.TCP10L synergizes with MAD1 in transcriptional suppression and cell cycle arrest through mutual interaction.The suppression of MAD1 by AKT-mediated phosphorylation activates MAD1 target genes transcription.Myc: the beauty and the beast.Combining histone deacetylase inhibitor vorinostat with aurora kinase inhibitors enhances lymphoma cell killing with repression of c-Myc, hTERT, and microRNA levels.Regulation of the MAD1 promoter by G-CSF.A novel role of the Mad family member Mad3 in cerebellar granule neuron precursor proliferation.MicroRNA 22 regulates cell cycle length in cerebellar granular neuron precursors.Cell-restricted immortalization by human papillomavirus correlates with telomerase activation and engagement of the hTERT promoter by Myc.AS601245, an Anti-Inflammatory JNK Inhibitor, and Clofibrate Have a Synergistic Effect in Inducing Cell Responses and in Affecting the Gene Expression Profile in CaCo-2 Colon Cancer Cells.One Decade Later: What has Gene Expression Profiling Told us About Neuronal Cell Types, Brain Function and Disease?
P2860
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P2860
The Mad side of the Max network: antagonizing the function of Myc and more.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
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2006年學術文章
@zh-hant
name
The Mad side of the Max network: antagonizing the function of Myc and more.
@ast
The Mad side of the Max network: antagonizing the function of Myc and more.
@en
type
label
The Mad side of the Max network: antagonizing the function of Myc and more.
@ast
The Mad side of the Max network: antagonizing the function of Myc and more.
@en
prefLabel
The Mad side of the Max network: antagonizing the function of Myc and more.
@ast
The Mad side of the Max network: antagonizing the function of Myc and more.
@en
P356
P1476
The Mad side of the Max network: antagonizing the function of Myc and more
@en
P2093
S Rottmann
P304
P356
10.1007/3-540-32952-8_4
P577
2006-01-01T00:00:00Z