Interleukin-23 may contribute to the pathogenesis of lumbar disc herniation through the IL-23/IL-17 pathway
about
Asthma and early herniated intervertebral disc disease.An Animal Model of Modic Changes by Embedding Autogenous Nucleus Pulposus inside Subchondral Bone of Lumbar Vertebrae.Involvement of IL-17 in Secondary Brain Injury After a Traumatic Brain Injury in Rats.IL-33/ST2 signaling contributes to radicular pain by modulating MAPK and NF-κB activation and inflammatory mediator expression in the spinal cord in rat models of noncompressive lumber disk herniation.
P2860
Interleukin-23 may contribute to the pathogenesis of lumbar disc herniation through the IL-23/IL-17 pathway
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年学术文章
@wuu
2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
@zh-sg
2016年學術文章
@yue
2016年學術文章
@zh
2016年學術文章
@zh-hant
name
Interleukin-23 may contribute ...... hrough the IL-23/IL-17 pathway
@ast
Interleukin-23 may contribute ...... hrough the IL-23/IL-17 pathway
@en
type
label
Interleukin-23 may contribute ...... hrough the IL-23/IL-17 pathway
@ast
Interleukin-23 may contribute ...... hrough the IL-23/IL-17 pathway
@en
prefLabel
Interleukin-23 may contribute ...... hrough the IL-23/IL-17 pathway
@ast
Interleukin-23 may contribute ...... hrough the IL-23/IL-17 pathway
@en
P2093
P2860
P1476
Interleukin-23 may contribute ...... hrough the IL-23/IL-17 pathway
@en
P2093
Hongqiang Jiang
Jianxiong Ma
Pengfei Li
Xinlong Ma
P2860
P2888
P356
10.1186/S13018-016-0343-8
P577
2016-01-16T00:00:00Z
P5875
P6179
1026277988