Activation of D1 dopamine receptors induces emergence from isoflurane general anesthesia.
about
Electroencephalographic variation during end maintenance and emergence from surgical anesthesiaNorepinephrine is required to promote wakefulness and for hypocretin-induced arousal in zebrafish.Electrical stimulation of the ventral tegmental area induces reanimation from general anesthesia.α2-Adrenergic stimulation of the ventrolateral preoptic nucleus destabilizes the anesthetic state.Dextroamphetamine (but Not Atomoxetine) Induces Reanimation from General Anesthesia: Implications for the Roles of Dopamine and Norepinephrine in Active Emergence.The Role of Dopaminergic VTA Neurons in General Anesthesia.Ageing delays emergence from general anaesthesia in rats by increasing anaesthetic sensitivity in the brainOptogenetic activation of dopamine neurons in the ventral tegmental area induces reanimation from general anesthesia.Electrophysiological characterization of entopeduncular nucleus neurons in anesthetized and freely moving rats.Caffeine accelerates recovery from general anesthesia.Effects of γ-Aminobutyric Acid Type A Receptor Modulation by Flumazenil on Emergence from General AnesthesiaThe Neurobiology of Anesthetic Emergence.Disconnecting Consciousness: Is There a Common Anesthetic End Point?A clinical review of inhalation anesthesia with sevoflurane: from early research to emerging topics.A Novel Strategy to Reverse General Anesthesia by Scavenging with the Acyclic Cucurbit[n]uril-type Molecular Container Calabadion 2Physostigmine and Methylphenidate Induce Distinct Arousal States During Isoflurane General Anesthesia in Rats.Explaining general anesthesia: a two-step hypothesis linking sleep circuits and the synaptic release machinery.Effects of pair bonding on dopamine D1 receptors in monogamous male titi monkeys (Callicebus cupreus).Caffeine accelerates recovery from general anesthesia via multiple pathways.Paradoxical Emergence: Administration of Subanesthetic Ketamine during Isoflurane Anesthesia Induces Burst Suppression but Accelerates Recovery.Selective blockade of N-methyl-D-aspartate channels in combination with dopamine receptor antagonism induces loss of the righting reflex in mice, but not immobility.Towards a better understanding of anesthesia emergence mechanisms: Research and clinical implications
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P2860
Activation of D1 dopamine receptors induces emergence from isoflurane general anesthesia.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
@zh-hans
2013年学术文章
@zh-my
2013年学术文章
@zh-sg
2013年學術文章
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2013年學術文章
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2013年學術文章
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name
Activation of D1 dopamine receptors induces emergence from isoflurane general anesthesia.
@ast
Activation of D1 dopamine receptors induces emergence from isoflurane general anesthesia.
@en
type
label
Activation of D1 dopamine receptors induces emergence from isoflurane general anesthesia.
@ast
Activation of D1 dopamine receptors induces emergence from isoflurane general anesthesia.
@en
prefLabel
Activation of D1 dopamine receptors induces emergence from isoflurane general anesthesia.
@ast
Activation of D1 dopamine receptors induces emergence from isoflurane general anesthesia.
@en
P2093
P2860
P1433
P1476
Activation of D1 dopamine receptors induces emergence from isoflurane general anesthesia.
@en
P2093
Emery N Brown
Jessica J Chemali
Norman E Taylor
P2860
P356
10.1097/ALN.0B013E318278C896
P407
P577
2013-01-01T00:00:00Z