Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.
about
Non-autonomous overgrowth by oncogenic niche cells: Cellular cooperation and competition in tumorigenesisAneuploidy causes premature differentiation of neural and intestinal stem cellsSterile inflammation in DrosophilamiR-8 modulates cytoskeletal regulators to influence cell survival and epithelial organization in Drosophila wings.Tumor suppressor roles of CENP-E and Nsl1 in Drosophila epithelial tissues.Aneuploidy and chromosomal instability in cancer: a jackpot to chaos.Centrosomes are multifunctional regulators of genome stabilityEpithelial neoplasia in Drosophila entails switch to primitive cell statesCentrosomal kinase Nek2 cooperates with oncogenic pathways to promote metastasisJNK signaling is needed to tolerate chromosomal instabilityAneuploidy, cell delamination and tumorigenesis in Drosophila epithelia.Aneuploidy: implications for protein homeostasis and diseaseAn essential step of kinetochore formation controlled by the SNARE protein Snap29.Centrosome and spindle assembly checkpoint loss leads to neural apoptosis and reduced brain size.Moesin is a major regulator of centrosome behavior in epithelial cells with extra centrosomes.Is aneuploidy deciding your fate?Phosphoenolpyruvate Carboxykinase Maintains Glycolysis-driven Growth in Drosophila Tumors.A deterministic analysis of genome integrity during neoplastic growth in Drosophila.Acentrosomal Drosophila epithelial cells exhibit abnormal cell division, leading to cell death and compensatory proliferation.Evidence for a novel function of Awd in maintenance of genomic stability.Feedback amplification loop drives malignant growth in epithelial tissues.CIN and Aneuploidy: Different Concepts, Different Consequences.Protein kinase C zeta suppresses low- or high-grade colorectal cancer (CRC) phenotypes by interphase centrosome anchoring.Modelling Cooperative Tumorigenesis in Drosophila.Contributions of DNA repair, cell cycle checkpoints and cell death to suppressing the DNA damage-induced tumorigenic behavior of Drosophila epithelial cells.Studying tumor growth in Drosophila using the tissue allograft method.The asymmetrically segregating lncRNA cherub is required for transforming stem cells into malignant cells.Drosophila Larval Brain Neoplasms Present Tumour-Type Dependent Genome Instability.
P2860
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P2860
Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年學術文章
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name
Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.
@ast
Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.
@en
type
label
Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.
@ast
Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.
@en
prefLabel
Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.
@ast
Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.
@en
P2093
P2860
P356
P1476
Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia
@en
P2093
Andrés Dekanty
Herbert Auer
Lara Barrio
P2860
P304
20549-20554
P356
10.1073/PNAS.1206675109
P407
P577
2012-11-26T00:00:00Z