Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia. - Wikidata - wikimedia - TriplyDB

Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.

Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.

http://www.wikidata.org/entity/Q36483559

about

Non-autonomous overgrowth by oncogenic niche cells: Cellular cooperation and competition in tumorigenesis Aneuploidy causes premature differentiation of neural and intestinal stem cells Sterile inflammation in Drosophila miR-8 modulates cytoskeletal regulators to influence cell survival and epithelial organization in Drosophila wings.Tumor suppressor roles of CENP-E and Nsl1 in Drosophila epithelial tissues.Aneuploidy and chromosomal instability in cancer: a jackpot to chaos.Centrosomes are multifunctional regulators of genome stability Epithelial neoplasia in Drosophila entails switch to primitive cell states Centrosomal kinase Nek2 cooperates with oncogenic pathways to promote metastasis JNK signaling is needed to tolerate chromosomal instability Aneuploidy, cell delamination and tumorigenesis in Drosophila epithelia.Aneuploidy: implications for protein homeostasis and disease An essential step of kinetochore formation controlled by the SNARE protein Snap29.Centrosome and spindle assembly checkpoint loss leads to neural apoptosis and reduced brain size.Moesin is a major regulator of centrosome behavior in epithelial cells with extra centrosomes.Is aneuploidy deciding your fate?Phosphoenolpyruvate Carboxykinase Maintains Glycolysis-driven Growth in Drosophila Tumors.A deterministic analysis of genome integrity during neoplastic growth in Drosophila.Acentrosomal Drosophila epithelial cells exhibit abnormal cell division, leading to cell death and compensatory proliferation.Evidence for a novel function of Awd in maintenance of genomic stability.Feedback amplification loop drives malignant growth in epithelial tissues.CIN and Aneuploidy: Different Concepts, Different Consequences.Protein kinase C zeta suppresses low- or high-grade colorectal cancer (CRC) phenotypes by interphase centrosome anchoring.Modelling Cooperative Tumorigenesis in Drosophila.Contributions of DNA repair, cell cycle checkpoints and cell death to suppressing the DNA damage-induced tumorigenic behavior of Drosophila epithelial cells.Studying tumor growth in Drosophila using the tissue allograft method.The asymmetrically segregating lncRNA cherub is required for transforming stem cells into malignant cells.Drosophila Larval Brain Neoplasms Present Tumour-Type Dependent Genome Instability.

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P2860

Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.

http://www.wikidata.org/entity/Q36483559

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

@zh-my

2012年学术文章

@zh-sg

2012年學術文章

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2012年學術文章

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2012年學術文章

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name

Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.

@ast

Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.

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type

label

Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.

@ast

Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.

@en

prefLabel

Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.

@ast

Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia.

@en

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PNAS.1206675109

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Aneuploidy-induced delaminating cells drive tumorigenesis in Drosophila epithelia

@en

P2093

Andrés Dekanty

http://www.w3.org/2001/XMLSchema#string

Herbert Auer

http://www.w3.org/2001/XMLSchema#string

Lara Barrio

http://www.w3.org/2001/XMLSchema#string

P2860

Hallmarks of Cancer: The Next Generation

The DNA-damage response in human biology and disease

The spindle-assembly checkpoint in space and time

Genomic instability--an evolving hallmark of cancer

Identification of aneuploidy-tolerating mutations

Wingless promotes proliferative growth in a gradient-independent manner

Proliferation of aneuploid human cells is limited by a p53-dependent mechanism.

Generating chromosome instability through the simultaneous deletion of Mad2 and p53.

Molecular cloning of sequences from wingless, a segment polarity gene in Drosophila: the spatial distribution of a transcript in embryos.

The Cdc42/Par6/aPKC polarity complex regulates apoptosis-induced compensatory proliferation in epithelia.

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20549-20554

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scholarly article

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10.1073/PNAS.1206675109

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50

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109

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Mariana Muzzopappa

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2012-11-26T00:00:00Z

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233775176

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23184991

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3528526

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