Absence of AMPKα2 accelerates cellular senescence via p16 induction in mouse embryonic fibroblasts.
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AMP-activated protein kinase reduces inflammatory responses and cellular senescence in pulmonary emphysemaAMPKα1 deletion in fibroblasts promotes tumorigenesis in athymic nude mice by p52-mediated elevation of erythropoietin and CDK2.AMPK and HIF signaling pathways regulate both longevity and cancer growth: the good news and the bad news about survival mechanisms.Molecular pathogenesis in chronic obstructive pulmonary disease and therapeutic potential by targeting AMP-activated protein kinase.Activation of AMP-activated protein kinase by metformin ablates angiotensin II-induced endoplasmic reticulum stress and hypertension in mice in vivo.Protective Effect of Hyperbaric Oxygen on Cognitive Impairment Induced by D-Galactose in Mice.
P2860
Absence of AMPKα2 accelerates cellular senescence via p16 induction in mouse embryonic fibroblasts.
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2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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name
Absence of AMPKα2 accelerates ...... n mouse embryonic fibroblasts.
@ast
Absence of AMPKα2 accelerates ...... n mouse embryonic fibroblasts.
@en
type
label
Absence of AMPKα2 accelerates ...... n mouse embryonic fibroblasts.
@ast
Absence of AMPKα2 accelerates ...... n mouse embryonic fibroblasts.
@en
prefLabel
Absence of AMPKα2 accelerates ...... n mouse embryonic fibroblasts.
@ast
Absence of AMPKα2 accelerates ...... n mouse embryonic fibroblasts.
@en
P2093
P2860
P1476
Absence of AMPKα2 accelerates ...... n mouse embryonic fibroblasts.
@en
P2093
Imoh Sunday Okon
Ming-Hui Zou
P2860
P356
10.1016/J.BIOCEL.2015.12.010
P577
2015-12-21T00:00:00Z