Multiple NF-κB sites in HIV-1 subtype C long terminal repeat confer superior magnitude of transcription and thereby the enhanced viral predominance.
about
Targeting NF-κB signaling with protein kinase C agonists as an emerging strategy for combating HIV latencyTriple NF-kB binding sites and LTR sequence similarities in HIV-1C isolates irrespective of helminth co-infectionPhylogenetic characterization of six full-length HIV-1 subtype C molecular clones from three patients: identification of rare subtype C strains containing two NF-κB motifs in the long terminal repeatPhenotypic properties of transmitted founder HIV-1A high throughput RNAi screen reveals determinants of HIV-1 activity in host kinasesReactivation of latent HIV-1 by new semi-synthetic ingenol esters.The PTAP sequence duplication in HIV-1 subtype C Gag p6 in drug-naive subjects of India and South Africa.tat Exon 1 exhibits functional diversity during HIV-1 subtype C primary infection.Heat Shock Factor 1 Mediates Latent HIV Reactivation.Clade C HIV-1 isolates circulating in Southern Africa exhibit a greater frequency of dicysteine motif-containing Tat variants than those in Southeast Asia and cause increased neurovirulence.Functional Incompatibility between the Generic NF-κB Motif and a Subtype-Specific Sp1III Element Drives the Formation of the HIV-1 Subtype C Viral PromoterInterference with nuclear factor kappaB signaling pathway by pathogen-encoded proteases: global and selective inhibition.Viral Decay Dynamics and Mathematical Modeling of Treatment Response: Evidence of Lower in vivo Fitness of HIV-1 Subtype CViral and cellular factors underlying neuropathogenesis in HIV associated neurocognitive disorders (HAND)Subtype-Specific Differences in Gag-Protease-Driven Replication Capacity Are Consistent with Intersubtype Differences in HIV-1 Disease Progression.Primate Lentiviruses Modulate NF-κB Activity by Multiple Mechanisms to Fine-Tune Viral and Cellular Gene Expression.Genetic diversity and proviral DNA load in different neural compartments of HIV-1 subtype C infection.HIV Tat protein: Is Tat-C much trickier than Tat-B?Genetic diversity in HIV-1 subtype C LTR from Brazil and Mozambique generates new transcription factor-binding sites.The biological characteristics of predominant strains of HIV-1 genotype: modeling of HIV-1 infection among men who have sex with men.Reply to Neogi et al.Distinctive variation in the U3R region of the 5' Long Terminal Repeat from diverse HIV-1 strains.Evolution of Neutralization Response in HIV-1 Subtype C-Infected Individuals Exhibiting Broad Cross-Clade Neutralization of HIV-1 Strains.Cellular RelB interacts with the transactivator Tat and enhance HIV-1 expression
P2860
Q27026631-7D448D9B-A645-469E-8405-D8616281CB8FQ33580261-AA15ED56-B1D2-4B6D-BE02-F8A148745587Q33713838-9BB07FC2-F711-4C4A-8F88-62A4E8E9FAE0Q34336058-56FFFACB-2AE5-4188-AEB4-48112DD991B2Q35195281-6E42EFE8-E85B-4D65-BB99-8E27344E133BQ35252396-2AC6103B-A381-4B85-A311-76F410E89B4EQ36259014-8233CD26-3966-44B3-9808-0388F44BA558Q36827396-BD5C8594-6381-4D7E-9893-FF27F5B48817Q36910965-6DF08C85-C487-4244-9E90-C69D11CAA2F1Q36940756-2A9ED7A9-E6FB-405C-B704-02E4A57DA6E1Q37174431-EAA41C59-A683-4A31-B219-B9BB078AEAC8Q37214160-A24BAA86-8D7B-43D6-8D5A-A06AFABABC9BQ37519818-D3FFD5C6-1BCE-4934-A5E2-B8AEFF1FAAA3Q38217388-5AE230E8-54ED-415F-A74D-D01B9D6993E0Q38706845-F48F62B7-AB89-48CD-93D6-5115497FBA55Q38928538-CA6D4E62-6557-4605-A0CA-9393045401CAQ39031641-48585CFC-7D70-4331-A6CE-23453E94290AQ41329286-A373BC5B-5D1A-4913-8567-5C43ED8A0AB8Q41463492-69C5F4E0-9029-4ED9-8ED0-0A6F0060935AQ41500668-961105CE-E7F9-4CC9-B6F6-BE9A2FEB191AQ42537511-D3975676-38B2-4F37-A19D-CAD36EF04A84Q53692218-0088C73E-5E0A-4AF9-9420-C12615B5E2FAQ55359818-D53FD772-6684-4B64-839B-9D2A562E8ED1Q58706821-4752ABA3-4AFD-4DAE-804E-76857962149F
P2860
Multiple NF-κB sites in HIV-1 subtype C long terminal repeat confer superior magnitude of transcription and thereby the enhanced viral predominance.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
Multiple NF-κB sites in HIV-1 ...... e enhanced viral predominance.
@ast
Multiple NF-κB sites in HIV-1 ...... e enhanced viral predominance.
@en
type
label
Multiple NF-κB sites in HIV-1 ...... e enhanced viral predominance.
@ast
Multiple NF-κB sites in HIV-1 ...... e enhanced viral predominance.
@en
prefLabel
Multiple NF-κB sites in HIV-1 ...... e enhanced viral predominance.
@ast
Multiple NF-κB sites in HIV-1 ...... e enhanced viral predominance.
@en
P2093
P2860
P50
P356
P1476
Multiple NF-κB sites in HIV-1 ...... e enhanced viral predominance.
@en
P2093
Anil Babu Mukthey
Anita Mahadevan
Anjali Verma
Kadappa Shivappa Satish
Kuan-Teh Jeang
Madhu Vajpayee
Narayana Cheedarla
Nirmala Rajagopalan
Pachamuthu Balakrishnan
Raghavendra Bhatt
P2860
P304
44714-44735
P356
10.1074/JBC.M112.397158
P407
P577
2012-11-06T00:00:00Z