RAGE signaling sustains inflammation and promotes tumor development.
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S100A9 interaction with TLR4 promotes tumor growthThe secreted protein S100A7 (psoriasin) is induced by telomere dysfunction in human keratinocytes independently of a DNA damage response and cell cycle regulatorsReceptor for advanced glycation end products and its involvement in inflammatory diseasesGene from a psoriasis susceptibility locus primes the skin for inflammationProinflammatory S100 proteins regulate the accumulation of myeloid-derived suppressor cellsRAGE, carboxylated glycans and S100A8/A9 play essential roles in colitis-associated carcinogenesisAggressiveness Niche: Can It Be the Foster Ground for Cancer Metastasis Precursors?Molecular signaling involving intrinsically disordered proteins in prostate cancerInvolvement of HMGB1 in Resistance to Tumor Vessel-Targeted, Monoclonal Antibody-Based ImmunotherapyPAMPs and DAMPs: signal 0s that spur autophagy and immunityTumour hypoxia promotes melanoma growth and metastasis via High Mobility Group Box-1 and M2-like macrophagesDamage-associated molecular patterns in cancer: a double-edged swordImmature myeloid cells directly contribute to skin tumor development by recruiting IL-17-producing CD4+ T cellsIL-17 mediated inflammation promotes tumor growth and progression in the skinCommon interactions between S100A4 and S100A9 defined by a novel chemical probeDeficiency of the Kruppel-like factor KLF4 correlates with increased cell proliferation and enhanced skin tumorigenesisGlyoxalase 1-419C>A variant is associated with oxidative stress: implications in prostate cancer progressionTopical application of a platelet activating factor receptor agonist suppresses phorbol ester-induced acute and chronic inflammation and has cancer chemopreventive activity in mouse skinS100A8/A9 stimulates keratinocyte proliferation in the development of squamous cell carcinoma of the skin via the receptor for advanced glycation-end productsBlocking the Interactions between Calcium-Bound S100A12 Protein and the V Domain of RAGE Using TranilastZinc replenishment reverses overexpression of the proinflammatory mediator S100A8 and esophageal preneoplasia in the ratTIRAP, an adaptor protein for TLR2/4, transduces a signal from RAGE phosphorylated upon ligand bindingThe multiple faces of RAGE--opportunities for therapeutic intervention in aging and chronic diseaseCommercial processed soy-based food product contains glycated and glycoxidated lunasin proteoformsApoptotic cell recognition receptors and scavenger receptors.Proteomic and pathway analyses reveal a network of inflammatory genes associated with differences in skin tumor promotion susceptibility in DBA/2 and C57BL/6 mice.Disturbances in metabolic, transport and structural genes in experimental colonic inflammation in the rat: a longitudinal genomic analysis.The receptor for advanced glycation end products promotes pancreatic carcinogenesis and accumulation of myeloid-derived suppressor cells.Identification of the Rage-dependent gene regulatory network in a mouse model of skin inflammation.Deletion of bone marrow-derived receptor for advanced glycation end products inhibits atherosclerotic plaque progression.The danger signal S100B integrates pathogen- and danger-sensing pathways to restrain inflammationTLR4-mediated skin carcinogenesis is dependent on immune and radioresistant cells.CD14(+)S100A9(+) monocytic myeloid-derived suppressor cells and their clinical relevance in non-small cell lung cancerAlternatively spliced RAGEv1 inhibits tumorigenesis through suppression of JNK signalingS100A9 a new marker for monocytic human myeloid-derived suppressor cells.Opposing function of MYBBP1A in proliferation and migration of head and neck squamous cell carcinoma cells.Longistatin in tick saliva blocks advanced glycation end-product receptor activation.HMGB1 enhances immune suppression by facilitating the differentiation and suppressive activity of myeloid-derived suppressor cellsThe association of HMGB1 expression with clinicopathological significance and prognosis in hepatocellular carcinoma: a meta-analysis and literature review.RAGE mediates S100A7-induced breast cancer growth and metastasis by modulating the tumor microenvironment
P2860
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P2860
RAGE signaling sustains inflammation and promotes tumor development.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
RAGE signaling sustains inflammation and promotes tumor development.
@ast
RAGE signaling sustains inflammation and promotes tumor development.
@en
type
label
RAGE signaling sustains inflammation and promotes tumor development.
@ast
RAGE signaling sustains inflammation and promotes tumor development.
@en
prefLabel
RAGE signaling sustains inflammation and promotes tumor development.
@ast
RAGE signaling sustains inflammation and promotes tumor development.
@en
P2093
P2860
P356
P1476
RAGE signaling sustains inflammation and promotes tumor development.
@en
P2093
Alexander Enk
Angelika Bierhaus
Astrid Riehl
Bernd Arnold
Gerhard Fürstenberger
Jochen Hess
Julia Németh
Karin Müller-Decker
Moritz Durchdewald
Peter Angel
P2860
P304
P356
10.1084/JEM.20070679
P407
P577
2008-01-21T00:00:00Z