Mast cell tryptase may modulate endothelial cell phenotype in healing myocardial infarcts. - Wikidata - wikimedia - TriplyDB

Mast cell tryptase may modulate endothelial cell phenotype in healing myocardial infarcts.

Mast cell tryptase may modulate endothelial cell phenotype in healing myocardial infarcts.

http://www.wikidata.org/entity/Q36509613

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Jab1, a novel protease-activated receptor-2 (PAR-2)-interacting protein, is involved in PAR-2-induced activation of activator protein-1 Tissue factor, protease activated receptors and pathologic heart remodelling Inflammation and Its Correlates in Regenerative Wound Healing: An Alternate Perspective CUX1 transcription factor is a downstream effector of the proteinase-activated receptor 2 (PAR2)Evidence for Mechanisms Underlying the Functional Benefits of a Myocardial Matrix Hydrogel for Post-MI Treatment.Cardiac mast cells: the centrepiece in adverse myocardial remodelling.Chemokines CCL3/MIP1α, CCL5/RANTES and CCL18/PARC are independent risk predictors of short-term mortality in patients with acute coronary syndromes.Mast cell tryptases and chymases in inflammation and host defense.Protease activated receptor-2 contributes to heart failure.Protease-activated receptors and myocardial infarction The emerging prominence of the cardiac mast cell as a potent mediator of adverse myocardial remodeling Tryptase activates isolated adult cardiac fibroblasts via protease activated receptor-2 (PAR-2).Fibroblasts in post-infarction inflammation and cardiac repair.Pathogenic role of cardiac mast cell activation/degranulation,TNF-alpha, and cell death in acute drug-related fatalities Mast cells regulate myofilament calcium sensitization and heart function after myocardial infarction.The immune system and cardiac repair.The pathogenesis of cardiac fibrosis.Estrogen modulates the influence of cardiac inflammatory cells on function of cardiac fibroblasts.Ischemia/reperfusion injury: The role of immune cells Targeting the chemokines in cardiac repair.The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis.Tryptase-positive mast cells and angiogenesis in keloids: a new possible post-surgical target for prevention.Fibroblasts and the extracellular matrix in right ventricular disease.Mast Cells: Key Contributors to Cardiac Fibrosis.Developmental changes of mast cell populations in the cerebral meninges of the rat.The Macrophage in Cardiac Homeostasis and Disease: JACC Macrophage in CVD Series (Part 4)

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Mast cell tryptase may modulate endothelial cell phenotype in healing myocardial infarcts.

http://www.wikidata.org/entity/Q36509613

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2005年論文

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2005年论文

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Mast cell tryptase may modulat ...... n healing myocardial infarcts.

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Mast cell tryptase may modulat ...... n healing myocardial infarcts.

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Mast cell tryptase may modulat ...... n healing myocardial infarcts.

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Mast cell tryptase may modulat ...... n healing myocardial infarcts.

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The Journal of Pathology

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Mast cell tryptase may modulat ...... n healing myocardial infarcts.

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Daniela Kraemer

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George H Caughey

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Guofeng Ren

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Leonardo Mendoza

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Lloyd H Michael

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Mark L Entman

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Porur Somasundaram

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P2860

Proliferative action of mast-cell tryptase is mediated by PAR2, COX2, prostaglandins, and PPARgamma : Possible relevance to human fibrotic disorders

Human interferon-inducible protein 10 is a potent inhibitor of angiogenesis in vivo

Chemokines and disease

Protease-activated receptor-2 stimulates angiogenesis and accelerates hemodynamic recovery in a mouse model of hindlimb ischemia.

Mast cells and their mediators in cutaneous wound healing--active participants or innocent bystanders?

Mast cells in innate immunity.

Interleukin-8 gene induction in the myocardium after ischemia and reperfusion in vivo.

Interactions of mast cell tryptase with thrombin receptors and PAR-2.

The inflammatory response in myocardial infarction.

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102-111

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scholarly article

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10.1002/PATH.1690

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1

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205

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Nikolaos G Frangogiannis

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2005-01-01T00:00:00Z

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8142325

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15586361

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