Activation of 5-HT2A receptors upregulates the function of the neuronal K-Cl cotransporter KCC2. - Wikidata - wikimedia - TriplyDB

Activation of 5-HT2A receptors upregulates the function of the neuronal K-Cl cotransporter KCC2.

Activation of 5-HT2A receptors upregulates the function of the neuronal K-Cl cotransporter KCC2.

http://www.wikidata.org/entity/Q36512082

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Development and regulation of chloride homeostasis in the central nervous system Recovery of neuronal and network excitability after spinal cord injury and implications for spasticity Modulation of neuronal activity by phosphorylation of the K-Cl cotransporter KCC2 Serotonergic modulation of post-synaptic inhibition and locomotor alternating pattern in the spinal cord Current view on the functional regulation of the neuronal K(+)-Cl(-) cotransporter KCC2 Overexpression of BDNF increases excitability of the lumbar spinal network and leads to robust early locomotor recovery in completely spinalized rats Serotonin, dopamine and noradrenaline adjust actions of myelinated afferents via modulation of presynaptic inhibition in the mouse spinal cord Synaptic Inhibition in Avian Interaural Level Difference Sound Localizing Neurons Reactive Neurogenesis and Down-Regulation of the Potassium-Chloride Cotransporter KCC2 in the Cochlear Nuclei after Cochlear Deafferentation.5-HT₂ and 5-HT₇ receptor agonists facilitate plantar stepping in chronic spinal rats through actions on different populations of spinal neurons.Chloride extrusion enhancers as novel therapeutics for neurological diseases Daily intermittent hypoxia enhances walking after chronic spinal cord injury: a randomized trial.Down-regulation of KCC2 expression and phosphorylation in motoneurons, and increases the number of in primary afferent projections to motoneurons in mice with post-stroke spasticity.Cation-chloride cotransporters in neuronal development, plasticity and disease Engagement of the GABA to KCC2 signaling pathway contributes to the analgesic effects of A3AR agonists in neuropathic pain Serotonin receptor and KCC2 gene expression in lumbar flexor and extensor motoneurons posttransection with and without passive cycling.Proteomics: in pursuit of effective traumatic brain injury therapeutics.GABAergic control of depression-related brain states Endocannabinoids blunt the augmentation of synaptic transmission by serotonin 2A receptors in the nucleus tractus solitarii (nTS).Intramuscular Neurotrophin-3 normalizes low threshold spinal reflexes, reduces spasms and improves mobility after bilateral corticospinal tract injury in rats.Inactivity-induced respiratory plasticity: protecting the drive to breathe in disorders that reduce respiratory neural activity Differential effects on KCC2 expression and spasticity of ALS and traumatic injuries to motoneurons.GABAA receptor-mediated tonic depolarization in developing neural circuits.K-Cl cotransporter KCC2--a moonlighting protein in excitatory and inhibitory synapse development and function.Depolarizing GABA and developmental epilepsies.Preclinical models of muscle spasticity: valuable tools in the development of novel treatment for neurological diseases and conditions.Regulation of neuronal chloride homeostasis by neuromodulators.Deletion of the Fractalkine Receptor, CX3CR1, Improves Endogenous Repair, Axon Sprouting, and Synaptogenesis after Spinal Cord Injury in Mice.Rehabilitation Strategies after Spinal Cord Injury: Inquiry into the Mechanisms of Success and Failure.The Role of K(+)-Cl(-)-Cotransporter-2 in Neuropathic Pain.The Timing of the Excitatory-to-Inhibitory GABA Switch Is Regulated by the Oxytocin Receptor via KCC2 Developmental Expression Patterns of KCC2 and Functionally Associated Molecules in the Human Brain.The membrane trafficking and functionality of the K+-Cl- co-transporter KCC2 is regulated by TGF-β2.The 5-HT2A receptor potassium-chloride cotransporter 2 signaling pathway in a rat incision pain model.Inter- and intralimb adaptations to a sensory perturbation during activation of the serotonin system after a low spinal cord transection in neonatal rats Phospholipase C-related but catalytically inactive protein modulates pain behavior in a neuropathic pain model in mice Undirected compensatory plasticity contributes to neuronal dysfunction after severe spinal cord injury.Spinal Disinhibition in Experimental and Clinical Painful Diabetic Neuropathy.Neuroprotection by Propofol Post-Conditioning: Focus on PKMζ/KCC2 Pathway Activity.Retracing your footsteps: developmental insights to spinal network plasticity following injury.

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Activation of 5-HT2A receptors upregulates the function of the neuronal K-Cl cotransporter KCC2.

http://www.wikidata.org/entity/Q36512082

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name

Activation of 5-HT2A receptors ...... ronal K-Cl cotransporter KCC2.

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Activation of 5-HT2A receptors ...... ronal K-Cl cotransporter KCC2.

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type

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Activation of 5-HT2A receptors ...... ronal K-Cl cotransporter KCC2.

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Activation of 5-HT2A receptors ...... ronal K-Cl cotransporter KCC2.

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Activation of 5-HT2A receptors ...... ronal K-Cl cotransporter KCC2.

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Activation of 5-HT2A receptors ...... ronal K-Cl cotransporter KCC2.

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Proceedings of the National Academy of Sciences of the United States of America

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Activation of 5-HT2A receptors ...... ronal K-Cl cotransporter KCC2.

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Cécile Brocard

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Dorothée Buttigieg

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Georg Haase

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Hélène Bras

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Karina Sadlaoud

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Laurent Vinay

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Pascale Boulenguez

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Rémi Bos

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Sylvie Liabeuf

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P2860

The motor infrastructure: from ion channels to neuronal networks

NMDA receptor activity downregulates KCC2 resulting in depolarizing GABAA receptor-mediated currents

1-Aminomethylbenzocycloalkanes: conformationally restricted hallucinogenic phenethylamine analogues as functionally selective 5-HT2A receptor agonists

Molecular characterization of a putative K-Cl cotransporter in rat brain. A neuronal-specific isoform

Serotonin 5-HT(2) receptor activation induces a long-lasting amplification of spinal reflex actions in the rat

5-HT(2A) and 5-HT(2C) receptors exert opposing effects on locomotor activity in mice

Recovery of motoneuron and locomotor function after spinal cord injury depends on constitutive activity in 5-HT2C receptors.

The 5-HT2A receptor is widely distributed in the rat spinal cord and mainly localized at the plasma membrane of postsynaptic neurons.

Small-molecule screen identifies inhibitors of the neuronal K-Cl cotransporter KCC2

Neuroanatomic relationships between the GABAergic and serotonergic systems in the developing human medulla.

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1

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110

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233941924

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23248270

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3538195

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