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Genetic deletion of CD36 enhances injury after acute neonatal stroke

Genetic deletion of CD36 enhances injury after acute neonatal stroke

http://www.wikidata.org/entity/Q36515336

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Mechanisms of perinatal arterial ischemic stroke Pathophysiology and neuroprotection of global and focal perinatal brain injury: lessons from animal models Barrier mechanisms in neonatal stroke Microglial Cells Prevent Hemorrhage in Neonatal Focal Arterial Stroke.Astrocytic transforming growth factor-beta signaling reduces subacute neuroinflammation after stroke in mice Cancer stem cell-specific scavenger receptor CD36 drives glioblastoma progression.CD36 deletion improves recovery from spinal cord injury.Neurobiology of microglial action in CNS injuries: receptor-mediated signaling mechanisms and functional roles Cleaning up after ICH: the role of Nrf2 in modulating microglia function and hematoma clearance Functional differences between microglia and monocytes after ischemic stroke.Neuronal Interleukin-4 as a Modulator of Microglial Pathways and Ischemic Brain Damage Modeling Ischemia in the Immature Brain: How Translational Are Animal Models?Endothelial CD36 Contributes to Postischemic Brain Injury by Promoting Neutrophil Activation via CSF3.The role of inflammation in perinatal brain injury.PPARγ-induced upregulation of CD36 enhances hematoma resolution and attenuates long-term neurological deficits after germinal matrix hemorrhage in neonatal rats.Nanomedicine in cerebral palsy.Microglia and Monocyte-Derived Macrophages in Stroke Fatty acid transporting proteins: Roles in brain development, aging, and stroke.Strategies to increase the activity of microglia as efficient protectors of the brain against infections.Live imaging of the innate immune response in neonates reveals differential TLR2 dependent activation patterns in sterile inflammation and infection.Lack of the scavenger receptor CD36 alters microglial phenotypes after neonatal stroke Cell Surface CD36 Protein in Monocyte/Macrophage Contributes to Phagocytosis during the Resolution Phase of Ischemic Stroke in Mice.Commentary on Myers et al.: growing role of the innate immunity receptor CD36 in central nervous system diseases.Sulfosuccinimidyl oleate sodium is neuroprotective and alleviates stroke-induced neuroinflammation.Uptake of dendrimer-drug by different cell types in the hippocampus after hypoxic-ischemic insult in neonatal mice: Effects of injury, microglial activation and hypothermia.Aged Mouse Cortical Microglia Display an Activation Profile Suggesting Immunotolerogenic Functions.Control of occlusion of middle cerebral artery in perinatal and neonatal mice with magnetic force

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Genetic deletion of CD36 enhances injury after acute neonatal stroke

http://www.wikidata.org/entity/Q36515336

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

2012年论文

@zh

2012年论文

@zh-cn

name

Genetic deletion of CD36 enhances injury after acute neonatal stroke

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Genetic deletion of CD36 enhances injury after acute neonatal stroke

@en

type

label

Genetic deletion of CD36 enhances injury after acute neonatal stroke

@ast

Genetic deletion of CD36 enhances injury after acute neonatal stroke

@en

prefLabel

Genetic deletion of CD36 enhances injury after acute neonatal stroke

@ast

Genetic deletion of CD36 enhances injury after acute neonatal stroke

@en

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Annals of Neurology

P1476

Genetic deletion of CD36 enhances injury after acute neonatal stroke

@en

P2093

Joel V Faustino

http://www.w3.org/2001/XMLSchema#string

Michael F Wendland

http://www.w3.org/2001/XMLSchema#string

Moon-Sook Woo

http://www.w3.org/2001/XMLSchema#string

Nikita Derugin

http://www.w3.org/2001/XMLSchema#string

Ping Zhou

http://www.w3.org/2001/XMLSchema#string

Xia Wang

http://www.w3.org/2001/XMLSchema#string

Zinaida S Vexler

http://www.w3.org/2001/XMLSchema#string

P2860

CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer

Myristylation and palmitylation of Src family members: the fats of the matter

Immature dendritic cells phagocytose apoptotic cells via alphavbeta5 and CD36, and cross-present antigens to cytotoxic T lymphocytes

CD36 gene transfer confers capacity for phagocytosis of cells undergoing apoptosis

Src family kinases: regulation of their activities, levels and identification of new pathways.

Microglial cells contribute to endogenous brain defenses after acute neonatal focal stroke

Engulfment of apoptotic cells: signals for a good meal.

The association between CD36 and Lyn protein tyrosine kinase is mediated by lipid.

CD36/fatty acid translocase, an inflammatory mediator, is involved in hyperlipidemia-induced exacerbation in ischemic brain injury.

Key role of CD36 in Toll-like receptor 2 signaling in cerebral ischemia.

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961-970

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scholarly article

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10.1002/ANA.23727

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6

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72

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Costantino Iadecola

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2012-12-01T00:00:00Z

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234032206

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23280844

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3539222

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