Potassium channel dysfunction in fibroblasts identifies patients with Alzheimer disease
about
Models of beta-amyloid ion channels in the membrane suggest that channel formation in the bilayer is a dynamic process.Disease and region-related cardiac fibroblast potassium current variations and potential functional significance.Altered proteolysis in fibroblasts of Alzheimer patients with predictive implications for subjects at risk of diseaseDipeptidyl peptidase 10 (DPP10(789)): a voltage gated potassium channel associated protein is abnormally expressed in Alzheimer's and other neurodegenerative diseases.Alzheimer and beta-amyloid-treated fibroblasts demonstrate a decrease in a memory-associated GTP-binding protein, Cp20Abnormal calcium homeostasis and mitochondrial polarization in a human encephalomyopathy.Internal Ca2+ mobilization is altered in fibroblasts from patients with Alzheimer diseaseRNA polymerase III drives alternative splicing of the potassium channel-interacting protein contributing to brain complexity and neurodegenerationSystemic administration of substance P recovers beta amyloid-induced cognitive deficits in rat: involvement of Kv potassium channels.Natural distribution of environmental radon daughters in the different brain areas of an Alzheimer disease victim.Transcriptional response of Saccharomyces cerevisiae to potassium starvation.Alzheimer's-specific effects of soluble beta-amyloid on protein kinase C-alpha and -gamma degradation in human fibroblasts.Involvement of the nitric oxide pathway in synaptic dysfunction following amyloid elevation in Alzheimer's disease.Alterations of some membrane transport proteins in Alzheimer's disease: role of amyloid beta-peptide.A cellular model of Alzheimer's disease therapeutic efficacy: PKC activation reverses Abeta-induced biomarker abnormality on cultured fibroblastsPotassium channels: structures, diseases, and modulators.Defective protein kinase C alpha leads to impaired secretion of soluble beta-amyloid precursor protein from Alzheimer's disease fibroblasts.Fibroblast circadian rhythms of PER2 expression depend on membrane potential and intracellular calciumBradykinin-induced amyloid precursor protein secretion: a protein kinase C-independent mechanism that is not altered in fibroblasts from patients with sporadic Alzheimer's disease.Drp1/Fis1 interaction mediates mitochondrial dysfunction, bioenergetic failure and cognitive decline in Alzheimer's disease.Structures and dynamics of β-barrel oligomer intermediates of amyloid-beta16-22 aggregation.
P2860
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P2860
Potassium channel dysfunction in fibroblasts identifies patients with Alzheimer disease
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Potassium channel dysfunction in fibroblasts identifies patients with Alzheimer disease
@ast
Potassium channel dysfunction in fibroblasts identifies patients with Alzheimer disease
@en
type
label
Potassium channel dysfunction in fibroblasts identifies patients with Alzheimer disease
@ast
Potassium channel dysfunction in fibroblasts identifies patients with Alzheimer disease
@en
prefLabel
Potassium channel dysfunction in fibroblasts identifies patients with Alzheimer disease
@ast
Potassium channel dysfunction in fibroblasts identifies patients with Alzheimer disease
@en
P2093
P2860
P356
P1476
Potassium channel dysfunction in fibroblasts identifies patients with Alzheimer disease
@en
P2093
P2860
P304
P356
10.1073/PNAS.90.17.8209
P407
P577
1993-09-01T00:00:00Z