The transcription factor T-bet regulates intestinal inflammation mediated by interleukin-7 receptor+ innate lymphoid cells. - Wikidata - wikimedia - TriplyDB

The transcription factor T-bet regulates intestinal inflammation mediated by interleukin-7 receptor+ innate lymphoid cells.

The transcription factor T-bet regulates intestinal inflammation mediated by interleukin-7 receptor+ innate lymphoid cells.

http://www.wikidata.org/entity/Q36518480

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Show and tell: disclosure and data sharing in experimental pathology The origin and role of innate lymphoid cells in the lung Modulation of liver tolerance by conventional and nonconventional antigen-presenting cells and regulatory immune cells Mechanisms of Microbe-Host Interaction in Crohn's Disease: Dysbiosis vs. Pathobiont Selection Breast Milk and Solid Food Shaping Intestinal Immunity Heterogeneity of the gut microbiome in mice: guidelines for optimizing experimental design Linking obesity with type 2 diabetes: the role of T-bet Inflammatory bowel disease as a model for translating the microbiome Deciphering the transcriptional switches of innate lymphoid cell programming: the right factors at the right time Evolving concepts: how diet and the intestinal microbiome act as modulators of breast malignancy Emerging insights on intestinal dysbiosis during bacterial infections Dysregulation of Group 3 Innate Lymphoid Cells in the Pathogenesis of Inflammatory Bowel Disease Crosstalk between Innate Lymphoid Cells and Other Immune Cells in the Tumor Microenvironment When Insult Is Added to Injury: Cross Talk between ILCs and Intestinal Epithelium in IBD Group 3 innate lymphoid cells (ILC3s): Origin, differentiation, and plasticity in humans and mice Developing in vitro expanded CD45RA+ regulatory T cells as an adoptive cell therapy for Crohn's disease Fecal microbiota transplantation: effectiveness, complexities, and lingering concerns.The transcription factor GATA3 is critical for the development of all IL-7Rα-expressing innate lymphoid cells.Role of the microbiota in immunity and inflammation.Mechanisms of innate lymphoid cell and natural killer T cell activation during mucosal inflammation.Beyond NK cells: the expanding universe of innate lymphoid cells.Gut microbiome composition and function in experimental colitis during active disease and treatment-induced remission.Nod/Ripk2 signaling in dendritic cells activates IL-17A-secreting innate lymphoid cells and drives colitis in T-bet-/-.Rag2-/- (TRUC) mice.Establishment and function of tissue-resident innate lymphoid cells in the skin.A new player on the psoriasis block: IL-17A- and IL-22-producing innate lymphoid cells.DOCK8 regulates protective immunity by controlling the function and survival of RORγt+ ILCs Immune and genetic gardening of the intestinal microbiome.IL-21 induces IL-22 production in CD4+ T cells.Rag2-deficient IL-1 Receptor Antagonist-deficient Mice Are a Novel Colitis Model in Which Innate Lymphoid Cell-derived IL-17 Is Involved in the Pathogenesis.Effects of baicalin in CD4 + CD29 + T cell subsets of ulcerative colitis patients.Impact of the gut microbiome on mucosal inflammation Aryl hydrocarbon receptor promotes RORγt⁺ group 3 ILCs and controls intestinal immunity and inflammation.Recent advances in inflammatory bowel disease: mucosal immune cells in intestinal inflammation.T cell contamination in flow cytometry gating approaches for analysis of innate lymphoid cells.Helicobacter cinaedi induced typhlocolitis in Rag-2-deficient mice Standardised animal models of host microbial mutualism.Distinct Commensals Induce Interleukin-1β via NLRP3 Inflammasome in Inflammatory Monocytes to Promote Intestinal Inflammation in Response to Injury Microbiota-mediated inflammation and antimicrobial defense in the intestine.Autoimmune host-microbiota interactions at barrier sites and beyond.Crotoxin from Crotalus durissus terrificus is able to down-modulate the acute intestinal inflammation in mice.

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The transcription factor T-bet regulates intestinal inflammation mediated by interleukin-7 receptor+ innate lymphoid cells.

http://www.wikidata.org/entity/Q36518480

description

2012 nî lūn-bûn

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2012年論文

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2012年论文

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2012年论文

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name

The transcription factor T-bet ...... ceptor+ innate lymphoid cells.

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The transcription factor T-bet ...... ceptor+ innate lymphoid cells.

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type

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The transcription factor T-bet ...... ceptor+ innate lymphoid cells.

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The transcription factor T-bet ...... ceptor+ innate lymphoid cells.

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prefLabel

The transcription factor T-bet ...... ceptor+ innate lymphoid cells.

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The transcription factor T-bet ...... ceptor+ innate lymphoid cells.

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J.IMMUNI.2012.09.008

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The transcription factor T-bet ...... eceptor+ innate lymphoid cells

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Ahmed Hashim

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Ellen Marks

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Emilie Stolarczyk

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Ian Jackson

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James B Canavan

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Jane K Howard

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Michael A Curtis

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Thomas T MacDonald

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P2860

Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease

A novel transcription factor, T-bet, directs Th1 lineage commitment

The transcription factors T-bet and GATA-3 control alternative pathways of T-cell differentiation through a shared set of target genes

Introducing mothur: open-source, platform-independent, community-supported software for describing and comparing microbial communities

Lymphoid tissue inducer-like cells are an innate source of IL-17 and IL-22

Communicable ulcerative colitis induced by T-bet deficiency in the innate immune system

SILVA: a comprehensive online resource for quality checked and aligned ribosomal RNA sequence data compatible with ARB

Salmonella enterica serovar typhimurium exploits inflammation to compete with the intestinal microbiota

Fontolizumab in moderate to severe Crohn's disease: a phase 2, randomized, double-blind, placebo-controlled, multiple-dose study

Anti-interleukin-12 antibody for active Crohn's disease

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674-684

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Julian Parkhill

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M Refik Gökmen

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232246240

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23063332

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