Deleterious effect of the IL-23/IL-17A axis and γδT cells on left ventricular remodeling after myocardial infarction.
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CXCL10 Is a Circulating Inflammatory Marker in Patients with Advanced Heart Failure: a Pilot Study.MicroRNA-150 protects the mouse heart from ischaemic injury by regulating cell death.Interleukin-17A contributes to the development of post-operative atrial fibrillation by regulating inflammation and fibrosis in rats with sterile pericarditis.Leukemia Inhibitory Factor Enhances Endogenous Cardiomyocyte Regeneration after Myocardial Infarction.Interleukin-2/Anti-Interleukin-2 Immune Complex Attenuates Cardiac Remodeling after Myocardial Infarction through Expansion of Regulatory T Cells.The role of antioxidation and immunomodulation in postnatal multipotent stem cell-mediated cardiac repair.Naoxintong attenuates Ischaemia/reperfusion Injury through inhibiting NLRP3 inflammasome activation.Cyclosporin A in left ventricular remodeling after myocardial infarction.Vγ1⁺ γδT Cells Are Correlated With Increasing Expression of Eosinophil Cationic Protein and Metalloproteinase-7 in Chronic Rhinosinusitis With Nasal Polyps Inducing the Formation of Edema.Mesenchymal stem cell therapy for cardiac inflammation: immunomodulatory properties and the influence of toll-like receptors.The Biological Basis for Cardiac Repair After Myocardial Infarction: From Inflammation to Fibrosis.Cytokine Effects on Mechano-Induced Electrical Activity in Atrial Myocardium.The proton-sensing G protein-coupled receptor T-cell death-associated gene 8 (TDAG8) shows cardioprotective effects against myocardial infarction.RORγt-expressing cells attenuate cardiac remodeling after myocardial infarction.A carvedilol-responsive microRNA, miR-125b-5p protects the heart from acute myocardial infarction by repressing pro-apoptotic bak1 and klf13 in cardiomyocytes.Anti-inflammatory therapies in myocardial infarction: failures, hopes, and challenges.The adaptive immune response to cardiac injury-the true roadblock to effective regenerative therapies?T-cells in myocardial infarction: Culprit instigators or mere effectors?Liver-heart crosstalk controls IL-22 activity in cardiac protection after myocardial infarctionAssociation of levels of interleukin 17 and T-helper 17 count with symptom severity and etiology of chronic heart failure: a case-control study
P2860
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P2860
Deleterious effect of the IL-23/IL-17A axis and γδT cells on left ventricular remodeling after myocardial infarction.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Deleterious effect of the IL-2 ...... g after myocardial infarction.
@ast
Deleterious effect of the IL-2 ...... g after myocardial infarction.
@en
type
label
Deleterious effect of the IL-2 ...... g after myocardial infarction.
@ast
Deleterious effect of the IL-2 ...... g after myocardial infarction.
@en
prefLabel
Deleterious effect of the IL-2 ...... g after myocardial infarction.
@ast
Deleterious effect of the IL-2 ...... g after myocardial infarction.
@en
P2093
P2860
P356
P1476
Deleterious effect of the IL-2 ...... g after myocardial infarction.
@en
P2093
Akihiko Yoshimura
Atsushi Anzai
Hideyuki Ito
Jun Fujita
Keiichi Fukuda
Ken Shinmura
Kensuke Kimura
Kentaro Ito
Motoaki Sano
P2860
P304
P356
10.1161/JAHA.112.004408
P577
2012-10-25T00:00:00Z