about
Emergence of the Middle East respiratory syndrome coronavirusMolecular pathology of emerging coronavirus infectionsExacerbated innate host response to SARS-CoV in aged non-human primatesCoronavirus Infection Modulates the Unfolded Protein Response and Mediates Sustained Translational RepressionEarly Upregulation of Acute Respiratory Distress Syndrome-Associated Cytokines Promotes Lethal Disease in an Aged-Mouse Model of Severe Acute Respiratory Syndrome Coronavirus InfectionA new mouse-adapted strain of SARS-CoV as a lethal model for evaluating antiviral agents in vitro and in vivoMacaque proteome response to highly pathogenic avian influenza and 1918 reassortant influenza virus infectionsPLP2 of mouse hepatitis virus A59 (MHV-A59) targets TBK1 to negatively regulate cellular type I interferon signaling pathway.Avian influenza virus, Streptococcus suis serotype 2, severe acute respiratory syndrome-coronavirus and beyond: molecular epidemiology, ecology and the situation in China.Detection and evaluation of immunofunction of patients with severe fever with thrombocytopenia syndrome.Toll-like receptors, chemokine receptors and death receptor ligands responses in SARS coronavirus infected human monocyte derived dendritic cells.Evasion by stealth: inefficient immune activation underlies poor T cell response and severe disease in SARS-CoV-infected mice.Understanding PRRSV infection in porcine lung based on genome-wide transcriptome response identified by deep sequencingPorcine reproductive and respiratory syndrome virus modifies innate immunity and alters disease outcome in pigs subsequently infected with porcine respiratory coronavirus: implications for respiratory viral co-infections.Mechanisms of severe acute respiratory syndrome pathogenesis and innate immunomodulation.The spike protein of SARS-CoV--a target for vaccine and therapeutic development.Pathogenesis of murine coronavirus in the central nervous system.Accessory protein 5a is a major antagonist of the antiviral action of interferon against murine coronavirus.Porcine epidemic diarrhea virus nucleocapsid protein antagonizes beta interferon production by sequestering the interaction between IRF3 and TBK1.The SARS coronavirus papain like protease can inhibit IRF3 at a post activation step that requires deubiquitination activity.Interferon priming enables cells to partially overturn the SARS coronavirus-induced block in innate immune activationp53 degradation by a coronavirus papain-like protease suppresses type I interferon signaling.Distinct immune response in two MERS-CoV-infected patients: can we go from bench to bedside?Cell-type-specific type I interferon antagonism influences organ tropism of murine coronavirusApplications of the phytomedicine Echinacea purpurea (Purple Coneflower) in infectious diseases.Porcine epidemic diarrhea virus inhibits dsRNA-induced interferon-β production in porcine intestinal epithelial cells by blockade of the RIG-I-mediated pathwayProtective role of Toll-like Receptor 3-induced type I interferon in murine coronavirus infection of macrophages.SARS coronavirus accessory proteins.Severe acute respiratory syndrome (SARS) coronavirus-induced lung epithelial cytokines exacerbate SARS pathogenesis by modulating intrinsic functions of monocyte-derived macrophages and dendritic cellsSARS coronavirus spike protein-induced innate immune response occurs via activation of the NF-kappaB pathway in human monocyte macrophages in vitro.The replication of a mouse adapted SARS-CoV in a mouse cell line stably expressing the murine SARS-CoV receptor mACE2 efficiently induces the expression of proinflammatory cytokinesReverse genetic characterization of the natural genomic deletion in SARS-Coronavirus strain Frankfurt-1 open reading frame 7b reveals an attenuating function of the 7b protein in-vitro and in-vivo.Comparative study of synonymous codon usage variations between the nucleocapsid and spike genes of coronavirus, and C-type lectin domain genes of human and mouse.CAPIH: a Web interface for comparative analyses and visualization of host-HIV protein-protein interactions.Macrophage polarization and HIV-1 infection.Dendritic cells in viral bronchiolitis.The highly cited SARS research literature.The interaction between human enteroviruses and type I IFN signaling pathway.The ORF4b-encoded accessory proteins of Middle East respiratory syndrome coronavirus and two related bat coronaviruses localize to the nucleus and inhibit innate immune signalling.Persistent replication of severe acute respiratory syndrome coronavirus in human tubular kidney cells selects for adaptive mutations in the membrane protein.
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P2860
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
SARS coronavirus and innate immunity.
@ast
SARS coronavirus and innate immunity.
@en
type
label
SARS coronavirus and innate immunity.
@ast
SARS coronavirus and innate immunity.
@en
prefLabel
SARS coronavirus and innate immunity.
@ast
SARS coronavirus and innate immunity.
@en
P2860
P1433
P1476
SARS coronavirus and innate immunity.
@en
P2093
Matthew Frieman
P2860
P304
P356
10.1016/J.VIRUSRES.2007.03.015
P577
2007-04-23T00:00:00Z