Notch gain of function inhibits chondrocyte differentiation via Rbpj-dependent suppression of Sox9. - Wikidata - wikimedia - TriplyDB

Notch gain of function inhibits chondrocyte differentiation via Rbpj-dependent suppression of Sox9.

Notch gain of function inhibits chondrocyte differentiation via Rbpj-dependent suppression of Sox9.

http://www.wikidata.org/entity/Q36542533

about

Genes that regulate morphogenesis and growth of the temporomandibular joint: a review Vascular patterning sets the stage for macro and micro hepatic architecture Molecular and cellular aspects of calcific aortic valve disease MiR-199b-5p inhibits osteogenic differentiation in ligamentum flavum cells by targeting JAG1 and modulating the Notch signalling pathway.RBP-Jκ-dependent Notch signaling is required for murine articular cartilage and joint maintenance.In Vivo Characterization of an AHR-Dependent Long Noncoding RNA Required for Proper Sox9b Expression.Hajdu-Cheney syndrome: a review.Repression of Sox9 by Jag1 is continuously required to suppress the default chondrogenic fate of vascular smooth muscle cells.Notch signaling in skeletal health and disease.A dual role for NOTCH signaling in joint cartilage maintenance and osteoarthritis Notch signaling controls chondrocyte hypertrophy via indirect regulation of Sox9.Canonical Notch activation in osteocytes causes osteopetrosis.Notch suppresses nuclear factor of activated T cells (NFAT) transactivation and Nfatc1 expression in chondrocytes Early pancreatic islet fate and maturation is controlled through RBP-Jκ Notch Signaling and the Skeleton.HES factors regulate specific aspects of chondrogenesis and chondrocyte hypertrophy during cartilage development Interleukin 6 mediates selected effects of Notch in chondrocytes.The Notch Intracellular Domain Has an RBPj-Independent Role during Mouse Hair Follicular Development Notch signaling in skeletal stem cells.An update of human mesenchymal stem cell biology and their clinical uses.SOX9 and the many facets of its regulation in the chondrocyte lineage.Signaling pathways regulating cartilage growth plate formation and activity.Notch Signaling in Development, Tissue Homeostasis, and Disease.C/EBPα is Regulated by the RANK Cytoplasmic IVVY535-538 Motif and Stimulates Osteoclastogenesis more Strongly than c-Fos.Differential tissue specific, temporal and spatial expression patterns of the Aggrecan gene is modulated by independent enhancer elements.Notch signaling pathways in human thoracic ossification of the ligamentum flavum.Notch Represses Transcription by PRC2 Recruitment to the Ternary Complex.

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P2860

Notch gain of function inhibits chondrocyte differentiation via Rbpj-dependent suppression of Sox9.

http://www.wikidata.org/entity/Q36542533

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

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name

Notch gain of function inhibit ...... dependent suppression of Sox9.

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Notch gain of function inhibit ...... dependent suppression of Sox9.

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type

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Notch gain of function inhibit ...... dependent suppression of Sox9.

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Notch gain of function inhibit ...... dependent suppression of Sox9.

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Notch gain of function inhibit ...... dependent suppression of Sox9.

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Notch gain of function inhibit ...... dependent suppression of Sox9.

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Journal of Bone and Mineral Research

P1476

Notch gain of function inhibit ...... dependent suppression of Sox9.

@en

P2093

Brendan Lee

http://www.w3.org/2001/XMLSchema#string

Jianning Tao

http://www.w3.org/2001/XMLSchema#string

Ming-Ming Jiang

http://www.w3.org/2001/XMLSchema#string

Shan Chen

http://www.w3.org/2001/XMLSchema#string

Tao Yang

http://www.w3.org/2001/XMLSchema#string

Terry Bertin

http://www.w3.org/2001/XMLSchema#string

Yangjin Bae

http://www.w3.org/2001/XMLSchema#string

Yuqing Chen

http://www.w3.org/2001/XMLSchema#string

P2860

Mutations in the human delta homologue, DLL3, cause axial skeletal defects in spondylocostal dysostosis

Sox9 sustains chondrocyte survival and hypertrophy in part through Pik3ca-Akt pathways

Truncating mutations in the last exon of NOTCH2 cause a rare skeletal disorder with osteoporosis

Notch signaling controls multiple steps of pancreatic differentiation

Activated Notch1 target genes during embryonic cell differentiation depend on the cellular context and include lineage determinants and inhibitors

Notch-deficient skin induces a lethal systemic B-lymphoproliferative disorder by secreting TSLP, a sentinel for epidermal integrity

HES and HERP families: multiple effectors of the Notch signaling pathway

Mutations in NOTCH2 in families with Hajdu-Cheney syndrome

E-selectin ligand-1 regulates growth plate homeostasis in mice by inhibiting the intracellular processing and secretion of mature TGF-beta

The transcription factor Sox9 has essential roles in successive steps of the chondrocyte differentiation pathway and is required for expression of Sox5 and Sox6

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649-659

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scholarly article

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10.1002/JBMR.1770

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3

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28

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2013-03-01T00:00:00Z

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230880401

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22991339

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3548081

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