YK-4-279 effectively antagonizes EWS-FLI1 induced leukemia in a transgenic mouse model.
about
The Effects and Mechanism of YK-4-279 in Combination with Docetaxel on Prostate Cancer.Structures of mithramycin analogues bound to DNA and implications for targeting transcription factor FLI1.RGD delivery of truncated coagulase to tumor vasculature affords local thrombotic activity to induce infarction of tumors in mice.Increased survival and cell cycle progression pathways are required for EWS/FLI1-induced malignant transformation.EWS-FLI1 and RNA helicase A interaction inhibitor YK-4-279 inhibits growth of neuroblastoma.NGR (Asn-Gly-Arg)-targeted delivery of coagulase to tumor vasculature arrests cancer cell growth.
P2860
YK-4-279 effectively antagonizes EWS-FLI1 induced leukemia in a transgenic mouse model.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
@zh-hant
2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
YK-4-279 effectively antagonizes EWS-FLI1 induced leukemia in a transgenic mouse model.
@ast
YK-4-279 effectively antagonizes EWS-FLI1 induced leukemia in a transgenic mouse model.
@en
type
label
YK-4-279 effectively antagonizes EWS-FLI1 induced leukemia in a transgenic mouse model.
@ast
YK-4-279 effectively antagonizes EWS-FLI1 induced leukemia in a transgenic mouse model.
@en
prefLabel
YK-4-279 effectively antagonizes EWS-FLI1 induced leukemia in a transgenic mouse model.
@ast
YK-4-279 effectively antagonizes EWS-FLI1 induced leukemia in a transgenic mouse model.
@en
P2093
P2860
P50
P356
P1433
P1476
YK-4-279 effectively antagonizes EWS-FLI1 induced leukemia in a transgenic mouse model.
@en
P2093
Aykut Üren
Hayriye Verda Erkizan
Idil Temel
Jeffrey Toretsky
Kristina M Mueller
Metin Özdemirli
Michaela Schlederer
Sung-Hyeok Hong
Tahereh Javaheri
P2860
P304
37678-37694
P356
10.18632/ONCOTARGET.5520
P407
P577
2015-10-08T00:00:00Z