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Parsing the Interferon Transcriptional Network and Its Disease Associations

Parsing the Interferon Transcriptional Network and Its Disease Associations

http://www.wikidata.org/entity/Q36550657

about

Respiratory viral infections and host responses; insights from genomics RNA-Seq analysis of chikungunya virus infection and identification of granzyme A as a major promoter of arthritic inflammation Network pharmacology of JAK inhibitors The Ox40/Ox40 Ligand Pathway Promotes Pathogenic Th Cell Responses, Plasmablast Accumulation, and Lupus Nephritis in NZB/W F1 Mice.Type 1 Interferon in the Human Intestine-A Co-ordinator of the Immune Response to the Microbiota.Single cell RNA sequencing to dissect the molecular heterogeneity in lupus nephritis.Understanding Human Autoimmunity and Autoinflammation Through Transcriptomics.Canonical and Non-Canonical Aspects of JAK-STAT Signaling: Lessons from Interferons for Cytokine Responses.Goose Mx and OASL Play Vital Roles in the Antiviral Effects of Type I, II, and III Interferon against Newly Emerging Avian Flavivirus.The Transcription Factor T-bet Limits Amplification of Type I IFN Transcriptome and Circuitry in T Helper 1 Cells.MxA Is A Positive Regulator of Type I IFN Signaling in HCV Infection.Protein synthesis inhibition and GADD34 control IFN-β heterogeneous expression in response to dsRNA.Innate Viral Receptor Signaling Determines Type 1 Diabetes Onset.Response to: 'JAK inhibition in STING-associated interferonopathy' by Crow et al.Multiplexed droplet single-cell RNA-sequencing using natural genetic variation.The IFN Response in Bats Displays Distinctive IFN-Stimulated Gene Expression Kinetics with Atypical RNASEL Induction.Pharmacokinetics, Pharmacodynamics, and Proposed Dosing of the Oral JAK1 and JAK2 Inhibitor Baricitinib in Pediatric and Young Adult CANDLE and SAVI Patients.Type I interferon-regulated gene expression and signaling in murine mixed glial cells lacking signal transducers and activators of transcription 1 or 2 or interferon regulatory factor 9.Chromatin dependencies in cancer and inflammation.Restoration of the type I IFN-IL-1 balance through targeted blockade of PTGER4 inhibits autoimmunity in NOD mice.Inactivation of Interferon Receptor Promotes the Establishment of Immune Privileged Tumor Microenvironment.Treg cells limit IFN-γ production to control macrophage accrual and phenotype during skeletal muscle regeneration.Interfering with transmission.JAK1/2 inhibition with baricitinib in the treatment of autoinflammatory interferonopathies.JAK inhibition in STING-associated interferonopathy Molecular dissection of plasmacytoid dendritic cell activation during a viral infection

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P2860

Parsing the Interferon Transcriptional Network and Its Disease Associations

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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Parsing the Interferon Transcriptional Network and Its Disease Associations

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Parsing the Interferon Transcriptional Network and Its Disease Associations

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Parsing the Interferon Transcriptional Network and Its Disease Associations

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Christophe Benoist

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Chun Jimmie Ye

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Devapregasan Moodley

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Diane Mathis

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Hideyuki Yoshida

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Hugo LeBoité

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Immunological Genome Project Consortium

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James D Clark

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Jean-Baptiste Telliez

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Katherine Rothamel

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P2860

Functional role of type I and type II interferons in antiviral defense

Unbiased reconstruction of a mammalian transcriptional network mediating pathogen responses

c-Myc regulates transcriptional pause release

A natural mutation in the Tyk2 pseudokinase domain underlies altered susceptibility of B10.Q/J mice to infection and autoimmunity

Regulation of type I interferon responses

Structural Linkage between Ligand Discrimination and Receptor Activation by Type I Interferons

Constitutive type I interferon modulates homeostatic balance through tonic signaling

Multiple functions of the IKK-related kinase IKKepsilon in interferon-mediated antiviral immunity

Meta-analysis of inter-species liver co-expression networks elucidates traits associated with common human diseases

Structural basis of a unique interferon-β signaling axis mediated via the receptor IFNAR1

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Jean-Laurent Casanova

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