CMTM7 knockdown increases tumorigenicity of human non-small cell lung cancer cells and EGFR-AKT signaling by reducing Rab5 activation
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Site-specific monoubiquitination downregulates Rab5 by disrupting effector binding and guanine nucleotide conversionCMTM5 is downregulated and suppresses tumour growth in hepatocellular carcinoma through regulating PI3K-AKT signalling.Intravesicular epidermal growth factor receptor subject to retrograde trafficking drives epidermal growth factor-dependent migration.Cyclic Guanosine Monophosphate (cGMP)-Dependent Protein Kinase II Blocks Epidermal Growth Factor (EGF)/Epidermal Growth Factor Receptor (EGFR)-Induced Biological Effects on Osteosarcoma Cells.
P2860
CMTM7 knockdown increases tumorigenicity of human non-small cell lung cancer cells and EGFR-AKT signaling by reducing Rab5 activation
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
CMTM7 knockdown increases tumo ...... ng by reducing Rab5 activation
@ast
CMTM7 knockdown increases tumo ...... ng by reducing Rab5 activation
@en
type
label
CMTM7 knockdown increases tumo ...... ng by reducing Rab5 activation
@ast
CMTM7 knockdown increases tumo ...... ng by reducing Rab5 activation
@en
prefLabel
CMTM7 knockdown increases tumo ...... ng by reducing Rab5 activation
@ast
CMTM7 knockdown increases tumo ...... ng by reducing Rab5 activation
@en
P2093
P2860
P356
P1433
P1476
CMTM7 knockdown increases tumo ...... ng by reducing Rab5 activation
@en
P2093
Baocai Liu
Wanqiong Yuan
Wenling Han
Xiaoning Mo
P2860
P304
41092-41107
P356
10.18632/ONCOTARGET.5732
P407
P577
2015-10-26T00:00:00Z