Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype. - Wikidata - wikimedia - TriplyDB

Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype.

Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype.

http://www.wikidata.org/entity/Q36568015

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Mitophagy plays a central role in mitochondrial ageing Therapeutic Manipulation of Ageing: Repurposing Old Dogs and Discovering New Tricks HMGB2 orchestrates the chromatin landscape of senescence-associated secretory phenotype gene loci Mitochondria in the spotlight of aging and idiopathic pulmonary fibrosis Rapamycin inhibits the secretory phenotype of senescent cells by a Nrf2-independent mechanism Senescence gives insights into the morphogenetic evolution of anamniotes.Context-dependent effects of cellular senescence in cancer development.From Ancient Pathways to Aging Cells-Connecting Metabolism and Cellular Senescence.Apoptotic transition of senescent cells accompanied with mitochondrial hyper-function.Whole Chromosome Instability induces senescence and promotes SASP.The Citrus Flavanone Naringenin Protects Myocardial Cells against Age-Associated Damage.Disease drivers of aging.mTOR Inhibition Rejuvenates the Aging Gingival Fibroblasts through Alleviating Oxidative Stress.Analysis of individual cells identifies cell-to-cell variability following induction of cellular senescence.Targeting the SASP to combat ageing: Mitochondria as possible intracellular allies?Mitochondrial dysfunction in inflammatory responses and cellular senescence: pathogenesis and pharmacological targets for chronic lung diseases.AMPK and HIF signaling pathways regulate both longevity and cancer growth: the good news and the bad news about survival mechanisms.Mitochondria in Cell Senescence: Is Mitophagy the Weakest Link?Mitochondrial redox system, dynamics, and dysfunction in lung inflammaging and COPD.Aging of the immune system: Focus on inflammation and vaccination.HIV-associated cellular senescence: A contributor to accelerated aging.Molecular pathogenesis in chronic obstructive pulmonary disease and therapeutic potential by targeting AMP-activated protein kinase.The female reproduction and senescence nexus.Mitophagy in neurodegeneration and aging.Successful and Maladaptive T Cell Aging.Cellular Senescence: A Translational Perspective.Fueling Inflamm-Aging through Mitochondrial Dysfunction: Mechanisms and Molecular Targets.Lipid (per) oxidation in mitochondria: an emerging target in the ageing process?Demystifying the role of mitochondria in senescence.Mitochondria and senescence: new actors for an old play.Techniques to Induce and Quantify Cellular Senescence.Controlling secretion to limit chemoresistance.Senescence in the aging process.Aging: Molecular Pathways and Implications on the Cardiovascular System.Structural and pharmacological basis for the induction of mitochondrial biogenesis by formoterol but not clenbuterol.Senescence-Inflammatory Regulation of Reparative Cellular Reprogramming in Aging and Cancer.CCN2 induces cellular senescence in fibroblasts.Replicatively senescent human fibroblasts reveal a distinct intracellular metabolic profile with alterations in NAD+ and nicotinamide metabolism.Autophagy Is Pro-Senescence When Seen in Close-Up, but Anti-Senescence in Long-Shot.Programmed cell senescence in skeleton during late puberty.

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Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype.

http://www.wikidata.org/entity/Q36568015

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2015 nî lūn-bûn

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype.

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Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype.

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Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype.

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J.CMET.2015.11.011

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Cell Metabolism

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Mitochondrial Dysfunction Induces Senescence with a Distinct Secretory Phenotype.

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Adam Freund

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Akos A Gerencser

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Arvind Ramanathan

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Christopher D Wiley

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Ethan A Sarnoski

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Hyung W Lim

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Judith Campisi

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Kotaro Shirakawa

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Michael C Velarde

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Pacome Lecot

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P2860

Oncogene-Induced Senescence: Putting the Brakes on Tumor Development

Premature ageing in mice expressing defective mitochondrial DNA polymerase

Feedback between p21 and reactive oxygen production is necessary for cell senescence

Reciprocal regulation of p53 and malic enzymes modulates metabolism and senescence

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

SIRT3 is a mitochondria-localized tumor suppressor required for maintenance of mitochondrial integrity and metabolism during stress

SIRT6 regulates TNF-α secretion through hydrolysis of long-chain fatty acyl lysine

Involvement of mortalin in cellular senescence from the perspective of its mitochondrial import, chaperone, and oxidative stress management functions

Declining NAD(+) induces a pseudohypoxic state disrupting nuclear-mitochondrial communication during aging

A versatile viral system for expression and depletion of proteins in mammalian cells

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2

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