Production of interleukin-8 by human neutrophils stimulated with Trichomonas vaginalis. - Wikidata - wikimedia - TriplyDB

Production of interleukin-8 by human neutrophils stimulated with Trichomonas vaginalis.

Production of interleukin-8 by human neutrophils stimulated with Trichomonas vaginalis.

http://www.wikidata.org/entity/Q36575104

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The Pathogenesis of Human Cervical Epithelium Cells Induced by Interacting with Trichomonas vaginalis Trichomoniasis immunity and the involvement of the purinergic signaling Trichomoniasis - are we giving the deserved attention to the most common non-viral sexually transmitted disease worldwide?Trichomonas vaginalis adherence mediates differential gene expression in human vaginal epithelial cells.The proteins secreted by Trichomonas vaginalis and vaginal epithelial cell response to secreted and episomally expressed AP65.Structural details and composition of Trichomonas vaginalis lipophosphoglycan in relevance to the epithelial immune function.Delayed human neutrophil apoptosis by Trichomonas vaginalis lysate Trichomonosis, a common curable STI, and prostate carcinogenesis--a proposed molecular mechanism.Trichomonas vaginalis lipophosphoglycan triggers a selective upregulation of cytokines by human female reproductive tract epithelial cells.Modulation of dendritic cell function by Trichomonas vaginalis-derived secretory products.NF-κB and CREB are involved in IL-8 production of human neutrophils induced by Trichomonas vaginalis-derived secretory products Involvement of purinergic signaling on nitric oxide production by neutrophils stimulated with Trichomonas vaginalis.Activation of MAPK Is Required for ROS Generation and Exocytosis in HMC-1 Cells Induced by Trichomonas vaginalis-Derived Secretory Products Trichomonas vaginalis and trichomoniasis in the Republic of Korea Membrane tubulovesicular extensions (cytonemes): secretory and adhesive cellular organelles Trichomonas vaginalis promotes apoptosis of human neutrophils by activating caspase-3 and reducing Mcl-1 expression.Superoxide anion production by human neutrophils activated by Trichomonas vaginalis.Proinflammatory cytokine and nitric oxide production by human macrophages stimulated with Trichomonas vaginalis Fifty years of the Korean Society for Parasitology.Impact of T. vaginalis infection on innate immune responses and reproductive outcome.SNAP23-Dependent Surface Translocation of Leukotriene B4 (LTB4) Receptor 1 Is Essential for NOX2-Mediated Exocytotic Degranulation in Human Mast Cells Induced by Trichomonas vaginalis-Secreted LTB4.IL-10 release by bovine epithelial cells cultured with Trichomonas vaginalis and Tritrichomonas foetus.Male infertility: a public health issue caused by sexually transmitted pathogens.Immunity in urogenital protozoa.Trichomonas vaginalis: Clinical relevance, pathogenicity and diagnosis.Inflammatory response of prostate epithelial cells to stimulation by Trichomonas vaginalis.Excretory-secretory products of Giardia lamblia induce interleukin-8 production in human colonic cells via activation of p38, ERK1/2, NF-κB and AP-1.Leukotriene B(4) receptors BLT1 and BLT2 are involved in interleukin-8 production in human neutrophils induced by Trichomonas vaginalis-derived secretory products.Suppressed production of pro-inflammatory cytokines by LPS-activated macrophages after treatment with Toxoplasma gondii lysate.Adenosine reduces reactive oxygen species and interleukin-8 production by Trichomonas vaginalis-stimulated neutrophils.Trichomonas vaginalis α-Actinin 2 Modulates Host Immune Responses by Inducing Tolerogenic Dendritic Cells via IL-10 Production from Regulatory T Cells Nitric oxide radicals in leucocytes and vaginal washes of Trichomonas vaginalis -infected symptomatic and asymptomatic women.The anti-Trichomonas vaginalis phloroglucinol derivative isoaustrobrasilol B modulates extracellular nucleotide hydrolysis.Streptococcus pneumoniae burden and nasopharyngeal inflammation during acute otitis media.Human fallopian tube neutrophils--a distinct phenotype from blood neutrophils.Involvement of mast cells in inflammation induced by Trichomonas vaginalis via crosstalk with vaginal epithelial cells.Trichomonas vaginalis Induces Production of Proinflammatory Cytokines in Mouse Macrophages Through Activation of MAPK and NF-κB Pathways Partially Mediated by TLR2.Susceptibility of Middle Adolescent Females to Sexually Transmitted infections: Impact of Hormone Contraception and Sexual Behaviors on Vaginal Immunity

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P2860

Production of interleukin-8 by human neutrophils stimulated with Trichomonas vaginalis.

http://www.wikidata.org/entity/Q36575104

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2004 nî lūn-bûn

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Production of interleukin-8 by human neutrophils stimulated with Trichomonas vaginalis.

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Production of interleukin-8 by human neutrophils stimulated with Trichomonas vaginalis.

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Production of interleukin-8 by human neutrophils stimulated with Trichomonas vaginalis.

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Production of interleukin-8 by human neutrophils stimulated with Trichomonas vaginalis.

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Production of interleukin-8 by human neutrophils stimulated with Trichomonas vaginalis.

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Production of interleukin-8 by human neutrophils stimulated with Trichomonas vaginalis.

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IAI.72.3.1326-1332.2004

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Infection and Immunity

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Production of interleukin-8 by human neutrophils stimulated with Trichomonas vaginalis

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Duk-Young Min

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Hyun Park

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Jae-Sook Ryu

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Ji-Hyun Kang

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Seung-Yong Jung

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P2860

CP30, a cysteine proteinase involved in Trichomonas vaginalis cytoadherence.

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

Contact-dependent cytopathogenic mechanisms of Trichomonas vaginalis.

Inflammatory mediators in dengue virus infection in children: interleukin-8 and its relationship to neutrophil degranulation

Involvement of mitogen-activated protein kinase pathways in interleukin-8 production by human monocytes and polymorphonuclear cells stimulated with lipopolysaccharide or Mycoplasma fermentans membrane lipoproteins.

Chemokine secretion of human cells in response to Toxoplasma gondii infection

Distinct proinflammatory host responses to Neisseria gonorrhoeae infection in immortalized human cervical and vaginal epithelial cells

Cytokine and chemokine production by human oral and vaginal epithelial cells in response to Candida albicans

The neutrophil as a cellular source of chemokines.

A distinct array of proinflammatory cytokines is expressed in human colon epithelial cells in response to bacterial invasion.

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1326-1332

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10.1128/IAI.72.3.1326-1332.2004

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3

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Myeong Heon Shin

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2004-03-01T00:00:00Z

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14977935

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Trichomonas vaginalis

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355987

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