Targeted ablation of the vitamin D receptor: an animal model of vitamin D-dependent rickets type II with alopecia. - Wikidata - wikimedia - TriplyDB

Targeted ablation of the vitamin D receptor: an animal model of vitamin D-dependent rickets type II with alopecia.

Targeted ablation of the vitamin D receptor: an animal model of vitamin D-dependent rickets type II with alopecia.

http://www.wikidata.org/entity/Q36576317

about

Mutation in KANK2, encoding a sequestering protein for steroid receptor coactivators, causes keratoderma and woolly hair Duodenal calcium absorption in vitamin D receptor-knockout mice: functional and molecular aspects Molecular link between vitamin D and cancer prevention Protective links between vitamin D, inflammatory bowel disease and colon cancer Pleiotropic Activities of Vitamin D Receptors - Adequate Activation for Multiple Health Outcomes Vitamin D receptor agonists: suitable candidates as novel therapeutic options in autoimmune inflammatory myopathy Nuclear receptors in bone physiology and diseases The role of nuclear hormone receptors in cutaneous wound repair Vitamin D activities for health outcomes Regulation of Long Bone Growth in Vertebrates; It Is Time to Catch Up Gene targeting by the vitamin D response element binding protein reveals a role for vitamin D in osteoblast mTOR signaling Metabolic and cellular analysis of alopecia in vitamin D receptor knockout mice Dominant-negative retinoic acid receptors elicit epidermal defects through a non-canonical pathway Genetic ablation of parathyroid glands reveals another source of parathyroid hormone Physical and functional interaction between the vitamin D receptor and hairless corepressor, two proteins required for hair cycling Isolation and characterization of mammalian homologs of the Drosophila gene glial cells missing Targeted inactivation of Npt2 in mice leads to severe renal phosphate wasting, hypercalciuria, and skeletal abnormalities Vitamin D and intestinal calcium transport after bariatric surgery.Snail1 controls bone mass by regulating Runx2 and VDR expression during osteoblast differentiation.Quantification of the vitamin D receptor-coregulator interaction Evolution of the interaction between Runx2 and VDR, two transcription factors involved in osteoblastogenesis.Mutations in the vitamin D receptor and hereditary vitamin D-resistant rickets.Vitamin D and VDR in cancer cachexia and muscle regeneration.Hairless is a nuclear receptor corepressor essential for skin function.The biology and pathology of vitamin D control in bone.Mouse and human BAC transgenes recapitulate tissue-specific expression of the vitamin D receptor in mice and rescue the VDR-null phenotype.Chlamydial infection in vitamin D receptor knockout mice is more intense and prolonged than in wild-type mice.Age-related changes in the epithelial and stromal compartments of the mammary gland in normocalcemic mice lacking the vitamin D3 receptor.Steroid hormone receptors: evolution, ligands, and molecular basis of biologic function.Genetic disorders and defects in vitamin d action.Hypophosphatemia leads to rickets by impairing caspase-mediated apoptosis of hypertrophic chondrocytes.1,25(OH)2D3 alters growth plate maturation and bone architecture in young rats with normal renal function.Vitamin D and the skin: Physiology and pathophysiology.Vitamin D receptor deficiency enhances Wnt/β-catenin signaling and tumor burden in colon cancer.The nuclear vitamin D receptor controls the expression of genes encoding factors which feed the "Fountain of Youth" to mediate healthful aging.Mechanisms involved in vitamin D mediated intestinal calcium absorption and in non-classical actions of vitamin D.Inactivation of the vitamin D receptor enhances susceptibility of murine skin to UV-induced tumorigenesis Vitamin D receptor negatively regulates bacterial-stimulated NF-kappaB activity in intestine.DLX3 regulates bone mass by targeting genes supporting osteoblast differentiation and mineral homeostasis in vivo Osteoblast-specific transcription factor Osterix increases vitamin D receptor gene expression in osteoblasts.

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P2860

Targeted ablation of the vitamin D receptor: an animal model of vitamin D-dependent rickets type II with alopecia.

http://www.wikidata.org/entity/Q36576317

description

1997 nî lūn-bûn

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1997年論文

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1997年論文

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1997年論文

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1997年論文

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1997年論文

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1997年论文

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1997年论文

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1997年论文

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Targeted ablation of the vitam ...... rickets type II with alopecia.

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Targeted ablation of the vitam ...... rickets type II with alopecia.

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Targeted ablation of the vitam ...... rickets type II with alopecia.

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Targeted ablation of the vitam ...... rickets type II with alopecia.

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Targeted ablation of the vitam ...... rickets type II with alopecia.

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Proceedings of the National Academy of Sciences of the United States of America

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Targeted ablation of the vitam ...... rickets type II with alopecia.

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A E Pirro

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P2860

An ochre mutation in the vitamin D receptor gene causes hereditary 1,25-dihydroxyvitamin D3-resistant rickets in three families

Long-term nocturnal calcium infusions can cure rickets and promote normal mineralization in hereditary resistance to 1,25-dihydroxyvitamin D.

Hereditary vitamin D resistant rickets caused by a novel mutation in the vitamin D receptor that results in decreased affinity for hormone and cellular hyporesponsiveness.

Regulation by vitamin D metabolites of messenger ribonucleic acid for preproparathyroid hormone in isolated bovine parathyroid cells.

Vitamin D, calcium, and epidermal differentiation.

Regulation by vitamin D metabolites of parathyroid hormone gene transcription in vivo in the rat.

Alterations in rat epidermis provoked by chronic vitamin D deficiency.

Target cells for 1,25-dihydroxyvitamin D3 in intestinal tract, stomach, kidney, skin, pituitary, and parathyroid.

Analysis of the relation between alopecia and resistance to 1,25-dihydroxyvitamin D.

Vitamin D is not directly necessary for bone growth and mineralization.

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