Dysregulated Type I Interferon and Inflammatory Monocyte-Macrophage Responses Cause Lethal Pneumonia in SARS-CoV-Infected Mice.
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Type I IFN signaling facilitates the development of IL-10-producing effector CD8+ T cells during murine influenza virus infection.Type I IFN Signaling Is Dispensable during Secondary Viral InfectionEarly endonuclease-mediated evasion of RNA sensing ensures efficient coronavirus replicationRecognition of Lys48-Linked Di-ubiquitin and Deubiquitinating Activities of the SARS Coronavirus Papain-like Protease.The Conserved Coronavirus Macrodomain Promotes Virulence and Suppresses the Innate Immune Response during Severe Acute Respiratory Syndrome Coronavirus Infection.Type I Interferons as Regulators of Lung Inflammation.The Severe Acute Respiratory Syndrome Coronavirus Nucleocapsid Inhibits Type I Interferon Production by Interfering with TRIM25-Mediated RIG-I Ubiquitination.Sex-Based Differences in Susceptibility to Severe Acute Respiratory Syndrome Coronavirus Infection.SARS-CoV-Encoded Small RNAs Contribute to Infection-Associated Lung Pathology.Unravelling the networks dictating host resistance versus tolerance during pulmonary infections.The Impact of the Interferon/TNF-Related Apoptosis-Inducing Ligand Signaling Axis on Disease Progression in Respiratory Viral Infection and Beyond.Pathogenic human coronavirus infections: causes and consequences of cytokine storm and immunopathology.MERS-coronavirus: From discovery to intervention.The nucleocapsid proteins of mouse hepatitis virus and severe acute respiratory syndrome coronavirus share the same IFN-β antagonizing mechanism: attenuation of PACT-mediated RIG-I/ MDA5 activation.Coronavirus nonstructural protein 15 mediates evasion of dsRNA sensors and limits apoptosis in macrophages.Mouse-adapted MERS coronavirus causes lethal lung disease in human DPP4 knockin mice.Immune responses in influenza A virus and human coronavirus infections: an ongoing battle between the virus and host.Interferon Lambda Genetics and Biology in Regulation of Viral Control.Type I interferon receptor knockout mice as models for infection of highly pathogenic viruses with outbreak potential.Exacerbated apoptosis of cells infected with infectious bursal disease virus (IBDV) upon exposure to Interferon alpha (IFN-α).Porcine Deltacoronavirus Accessory Protein NS6 Antagonizes Interferon Beta Production by Interfering with the Binding of RIG-I/MDA5 to Double-Stranded RNAComplement Activation Contributes to Severe Acute Respiratory Syndrome Coronavirus PathogenesisSARS-Coronavirus Open Reading Frame-3a drives multimodal necrotic cell death
P2860
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P2860
Dysregulated Type I Interferon and Inflammatory Monocyte-Macrophage Responses Cause Lethal Pneumonia in SARS-CoV-Infected Mice.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Dysregulated Type I Interferon ...... nia in SARS-CoV-Infected Mice.
@ast
Dysregulated Type I Interferon ...... nia in SARS-CoV-Infected Mice.
@en
type
label
Dysregulated Type I Interferon ...... nia in SARS-CoV-Infected Mice.
@ast
Dysregulated Type I Interferon ...... nia in SARS-CoV-Infected Mice.
@en
prefLabel
Dysregulated Type I Interferon ...... nia in SARS-CoV-Infected Mice.
@ast
Dysregulated Type I Interferon ...... nia in SARS-CoV-Infected Mice.
@en
P2093
P2860
P50
P1433
P1476
Dysregulated Type I Interferon ...... onia in SARS-CoV-Infected Mice
@en
P2093
Jincun Zhao
Matthias Mack
Rahul Vijay
Rudragouda Channappanavar
P2860
P304
P356
10.1016/J.CHOM.2016.01.007
P577
2016-02-01T00:00:00Z