Stem cell therapy: social recognition recovery in a FASD model.
about
Fetal Alcohol Spectrum Disorder (FASD) Associated Neural Defects: Complex Mechanisms and Potential Therapeutic TargetsStem cell therapy: a new approach to the treatment of refractory depression.Pre-administration of G9a/GLP inhibitor during synaptogenesis prevents postnatal ethanol-induced LTP deficits and neurobehavioral abnormalities in adult mice.An Update on Fetal Alcohol Syndrome-Pathogenesis, Risks, and Treatment.Distinct neurobehavioral dysfunction based on the timing of developmental binge-like alcohol exposure.Ethanol exposure induces neonatal neurodegeneration by enhancing CB1R Exon1 histone H4K8 acetylation and up-regulating CB1R function causing neurobehavioral abnormalities in adult mice.The impact of prenatal alcohol exposure on social, cognitive and affective behavioral domains: Insights from rodent models.Effects of atelocollagen on neural stem cell function and its migrating capacity into brain in psychiatric disease model.
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P2860
Stem cell therapy: social recognition recovery in a FASD model.
description
2012 nî lūn-bûn
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年论文
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2012年论文
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2012年论文
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name
Stem cell therapy: social recognition recovery in a FASD model.
@ast
Stem cell therapy: social recognition recovery in a FASD model.
@en
type
label
Stem cell therapy: social recognition recovery in a FASD model.
@ast
Stem cell therapy: social recognition recovery in a FASD model.
@en
prefLabel
Stem cell therapy: social recognition recovery in a FASD model.
@ast
Stem cell therapy: social recognition recovery in a FASD model.
@en
P2093
P2860
P356
P1476
Stem cell therapy: social recognition recovery in a FASD model.
@en
P2093
E Hashimoto
T Shirasaka
T Yoshinaga
P2860
P2888
P356
10.1038/TP.2012.111
P577
2012-11-13T00:00:00Z
P5875
P6179
1001095634