Isolation of murine valve endothelial cells

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Growth and maturation of heart valves leads to changes in endothelial cell distribution, impaired function, decreased metabolism and reduced cell proliferation.Cross Talk between NOTCH Signaling and Biomechanics in Human Aortic Valve Disease Pathogenesis.

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P2860

Isolation of murine valve endothelial cells

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2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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2014年论文

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name

Isolation of murine valve endothelial cells

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Isolation of murine valve endothelial cells

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Isolation of murine valve endothelial cells

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Isolation of murine valve endothelial cells

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Isolation of murine valve endothelial cells

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Isolation of murine valve endothelial cells

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P1476

Isolation of murine valve endothelial cells

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P2093

Joy Lincoln

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Lindsey J Miller

http://www.w3.org/2001/XMLSchema#string

P2860

Endothelial deletion of murine Jag1 leads to valve calcification and congenital heart defects associated with Alagille syndrome

Scleraxis is required for cell lineage differentiation and extracellular matrix remodeling during murine heart valve formation in vivo

Nfatc1 coordinates valve endocardial cell lineage development required for heart valve formation.

Mmp15 is a direct target of Snai1 during endothelial to mesenchymal transformation and endocardial cushion development.

Loss of Gata5 in mice leads to bicuspid aortic valve.

Molecular and developmental mechanisms of congenital heart valve disease.

Heart valve development, maintenance, and disease: the role of endothelial cells.

Hemodynamic and cellular response feedback in calcific aortic valve disease.

Mitral valve endothelial cells with osteogenic differentiation potential.

Hemodynamic environments from opposing sides of human aortic valve leaflets evoke distinct endothelial phenotypes in vitro.

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scholarly article

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10.3791/51860

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2014-08-21T00:00:00Z

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4758760

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Isolation of murine valve endothelial cells

about

P2860

P2860

Isolation of murine valve endothelial cells

description

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P1476

P2093

P2860

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P2860

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