Increased expression of microRNA-146a decreases myocardial ischaemia/reperfusion injury. - Wikidata - wikimedia - TriplyDB

Increased expression of microRNA-146a decreases myocardial ischaemia/reperfusion injury.

Increased expression of microRNA-146a decreases myocardial ischaemia/reperfusion injury.

http://www.wikidata.org/entity/Q36596636

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Current understanding of molecular pathology and treatment of cardiomyopathy in duchenne muscular dystrophy MicroRNAs as potential signatures of environmental exposure or effect: a systematic review MicroRNA-141 regulates the expression level of ICAM-1 on endothelium to decrease myocardial ischemia-reperfusion injury.MicroRNA-125b protects against myocardial ischaemia/reperfusion injury via targeting p53-mediated apoptotic signalling and TRAF6.MicroRNA signatures differentiate preserved from reduced ejection fraction heart failure Inhibition of microRNA-497 ameliorates anoxia/reoxygenation injury in cardiomyocytes by suppressing cell apoptosis and enhancing autophagy Attenuation of Cardiac Dysfunction in Polymicrobial Sepsis by MicroRNA-146a Is Mediated via Targeting of IRAK1 and TRAF6 Expression.miR-146a targets Fos expression in human cardiac cells miR-146a regulates glucose induced upregulation of inflammatory cytokines extracellular matrix proteins in the retina and kidney in diabetes Attenuation of cardiac dysfunction and remodeling of myocardial infarction by microRNA-130a are mediated by suppression of PTEN and activation of PI3K dependent signaling.MicroRNA-128 inhibition attenuates myocardial ischemia/reperfusion injury-induced cardiomyocyte apoptosis by the targeted activation of peroxisome proliferator-activated receptor gamma.Combination of microRNA-21 and microRNA-146a Attenuates Cardiac Dysfunction and Apoptosis During Acute Myocardial Infarction in Mice Toll-like receptor 3 plays a role in myocardial infarction and ischemia/reperfusion injury.MicroRNA-214 protects against hypoxia/reoxygenation induced cell damage and myocardial ischemia/reperfusion injury via suppression of PTEN and Bim1 expression.Neuroprotection by astrocytes in brain ischemia: importance of microRNAs.Regulatory RNAs and paracrine networks in the heart.Identifying the role of microRNAs in spinal cord injury.MicroRNAs: Roles in Regulating Neuroinflammation.Enhanced Glycolytic Metabolism Contributes to Cardiac Dysfunction in Polymicrobial Sepsis.Exosomes as critical agents of cardiac regeneration triggered by cell therapy.MicroRNA-125b Prevents Cardiac Dysfunction in Polymicrobial Sepsis by Targeting TRAF6-Mediated Nuclear Factor κB Activation and p53-Mediated Apoptotic Signaling.IL-8 up-regulates proliferative angiogenesis in ischemic myocardium in rabbits through phosphorylation of Akt/GSK-3β(ser9) dependent pathways Neuroprotective effects of viral overexpression of microRNA-22 in rat and cell models of cerebral ischemia-reperfusion injury.Cardiovascular research as a forum for publications from China: present, past, and future.MiR-146a protects small intestine against ischemia/reperfusion injury by down-regulating TLR4/TRAF6/NF-κB pathway.TLR3 Mediates Repair and Regeneration of Damaged Neonatal Heart through Glycolysis Dependent YAP1 Regulated miR-152 Expression.MicroRNA-146a in Human and Experimental Ischemic AKI: CXCL8-Dependent Mechanism of Action.AT1 receptor blocker azilsartan medoxomil normalizes plasma miR-146a and miR-342-3p in a murine heart failure model.Antineoplastic Drug-Induced Cardiotoxicity: A Redox Perspective.Insights into innate immune signalling in controlling cardiac remodelling.Targeting regenerative exosomes to myocardial infarction using cardiac homing peptide.MicroRNA-22 targeting CBP protects against myocardial ischemia-reperfusion injury through anti-apoptosis in rats.GATA-4-expressing mouse bone marrow mesenchymal stem cells improve cardiac function after myocardial infarction via secreted exosomes.Inhibition of interleukin-1 receptor-associated kinase 1 (IRAK1) as a therapeutic strategy MiR-320 regulates cardiomyocyte apoptosis induced by ischemia-reperfusion injury by targeting AKIP1 miR-146a Attenuates Sepsis-Induced Myocardial Dysfunction by Suppressing IRAK1 and TRAF6 via Targeting ErbB4 Expression

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Increased expression of microRNA-146a decreases myocardial ischaemia/reperfusion injury.

http://www.wikidata.org/entity/Q36596636

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2012年論文

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2012年论文

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2012年论文

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name

Increased expression of microRNA-146a decreases myocardial ischaemia/reperfusion injury.

@ast

Increased expression of microRNA-146a decreases myocardial ischaemia/reperfusion injury.

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type

label

Increased expression of microRNA-146a decreases myocardial ischaemia/reperfusion injury.

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Increased expression of microRNA-146a decreases myocardial ischaemia/reperfusion injury.

@en

prefLabel

Increased expression of microRNA-146a decreases myocardial ischaemia/reperfusion injury.

@ast

Increased expression of microRNA-146a decreases myocardial ischaemia/reperfusion injury.

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Cardiovascular Research

P1476

Increased expression of microRNA-146a decreases myocardial ischaemia/reperfusion injury.

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P2093

Chuanfu Li

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David Williams

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Jianghuan Zou

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John Kalbfleisch

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Li Liu

http://www.w3.org/2001/XMLSchema#string

Tuanzhu Ha

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Xia Zhang

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Xiang Gao

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Xiaohui Wang

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P2860

MicroRNAs in NF-kappaB signaling

MicroRNA expression signature and the role of microRNA-21 in the early phase of acute myocardial infarction

MicroRNA-320 is involved in the regulation of cardiac ischemia/reperfusion injury by targeting heat-shock protein 20

NF-kappaB-dependent induction of microRNA miR-146, an inhibitor targeted to signaling proteins of innate immune responses

MicroRNA-92a controls angiogenesis and functional recovery of ischemic tissues in mice

A human homologue of the Drosophila Toll protein signals activation of adaptive immunity

MicroRNAs and immunity: novel players in the regulation of normal immune function and inflammation

Modulating Toll-like receptor mediated signaling by (1-->3)-beta-D-glucan rapidly induces cardioprotection

TLR2 ligands attenuate cardiac dysfunction in polymicrobial sepsis via a phosphoinositide 3-kinase-dependent mechanism.

Toll-like receptor-mediated NF-kappaB activation: a phylogenetically conserved paradigm in innate immunity.

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10.1093/CVR/CVS356

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3

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97

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reperfusion injury

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3567787

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