The IL-33/ST2 pathway contributes to intestinal tumorigenesis in humans and mice. - Wikidata - wikimedia - TriplyDB

The IL-33/ST2 pathway contributes to intestinal tumorigenesis in humans and mice.

The IL-33/ST2 pathway contributes to intestinal tumorigenesis in humans and mice.

http://www.wikidata.org/entity/Q36597522

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Cytokine-Induced Modulation of Colorectal Cancer The Role of IL-33-Dependent Inflammation in the Tumor Microenvironment.ILC2s regulate adaptive Th2 cell functions via PD-L1 checkpoint control.Epithelial-derived IL-33 promotes intestinal tumorigenesis in Apc Min/+ mice.The ESRP1-GPR137 axis contributes to intestinal pathogenesis.Interleukins 17 and 23 in patients with gastric neoplasms.Unique invariant natural killer T cells promote intestinal polyps by suppressing TH1 immunity and promoting regulatory T cells.Editorial: Danger Signals Triggering Immune Response and Inflammation.Transcriptomic Landscape of Treatment - Naïve Ulcerative Colitis.TREM-1 promotes intestinal tumorigenesis.Gram-negative bacteria facilitate tumor progression through TLR4/IL-33 pathway in patients with non-small-cell lung cancer.The Pro-tumorigenic IL-33 Involved in Antitumor Immunity: A Yin and Yang Cytokine The Role of IL-33/ST2 Pathway in Tumorigenesis IL-33 facilitates proliferation of colorectal cancer dependent on COX2/PGE

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The IL-33/ST2 pathway contributes to intestinal tumorigenesis in humans and mice.

http://www.wikidata.org/entity/Q36597522

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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The IL-33/ST2 pathway contributes to intestinal tumorigenesis in humans and mice.

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The IL-33/ST2 pathway contributes to intestinal tumorigenesis in humans and mice.

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The IL-33/ST2 pathway contributes to intestinal tumorigenesis in humans and mice.

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The IL-33/ST2 pathway contributes to intestinal tumorigenesis in humans and mice.

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The IL-33/ST2 pathway contributes to intestinal tumorigenesis in humans and mice.

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The IL-33/ST2 pathway contributes to intestinal tumorigenesis in humans and mice.

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The IL-33/ST2 pathway contributes to intestinal tumorigenesis in humans and mice.

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Achim Weber

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Alessandro Lugli

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Gieri Cathomas

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Giulia Ruzzante

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Holger Moch

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Jenna R Murdoch

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Kirsten D Mertz

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Marie-Hélène Wasmer

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Stefanie Joller

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Thomas Brümmendorf

http://www.w3.org/2001/XMLSchema#string

P2860

Immunity, Inflammation, and Cancer

Type, density, and location of immune cells within human colorectal tumors predict clinical outcome

The dual function cytokine IL-33 interacts with the transcription factor NF-κB to dampen NF-κB-stimulated gene transcription

Endogenous IL-33 is highly expressed in mouse epithelial barrier tissues, lymphoid organs, brain, embryos, and inflamed tissues: in situ analysis using a novel Il-33-LacZ gene trap reporter strain

Genetic alterations during colorectal-tumor development

IL-33, a potent inducer of adaptive immunity to intestinal nematodes

IL-6 and Stat3 are required for survival of intestinal epithelial cells and development of colitis-associated cancer

Tissue microarrays for high-throughput molecular profiling of tumor specimens

Epithelial-derived IL-33 and its receptor ST2 are dysregulated in ulcerative colitis and in experimental Th1/Th2 driven enteritis

IL-33 is a crucial amplifier of innate rather than acquired immunity

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1

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Viktor H. Koelzer

Lukas Franz Mager

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2015-06-26T00:00:00Z

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26942077

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