Reperfusion-induced myocardial dysfunction is prevented by endogenous annexin-A1 and its N-terminal-derived peptide Ac-ANX-A1(2-26).

Reperfusion-induced myocardial dysfunction is prevented by endogenous annexin-A1 and its N-terminal-derived peptide Ac-ANX-A1(2-26).

Item
http://www.wikidata.org/entity/Q36603227
Antagonism of human formyl peptide receptor 1 (FPR1) by chromones and related isoflavones.Antenatal hypoxia induces epigenetic repression of glucocorticoid receptor and promotes ischemic-sensitive phenotype in the developing heart.Inhibition of formyl peptide receptor 1 reduces the efficacy of anticancer chemotherapy against carcinogen-induced breast cancer.PR65A phosphorylation regulates PP2A complex signaling.Small-molecule-biased formyl peptide receptor agonist compound 17b protects against myocardial ischaemia-reperfusion injury in mice.Annexin A1: potential for glucocorticoid sparing in RA.A fragmented form of annexin A1 is secreted from C2C12 myotubes by electric pulse-induced contraction.The advantageous role of annexin A1 in cardiovascular disease.Ac2-26, an Annexin A1 Peptide, Attenuates Ischemia-Reperfusion-Induced Acute Lung Injury.Neuroprotective Effects of Annexin A1 Tripeptide after Deep Hypothermic Circulatory Arrest in Rats.HNO/cGMP-dependent antihypertrophic actions of isopropylamine-NONOate in neonatal rat cardiomyocytes: potential therapeutic advantages of HNO over NO.Endogenous Annexin-A1 Regulates Haematopoietic Stem Cell Mobilisation and Inflammatory Response Post Myocardial Infarction in Mice In Vivo.Biomarkers identification for acute myocardial infarction detection via weighted gene co-expression network analysis.Annexin A1 attenuates microvascular complications through restoration of Akt signalling in a murine model of type 1 diabetes.Formyl Peptide Receptor 2 Is Involved in Cardiac Repair After Myocardial Infarction Through Mobilization of Circulating Angiogenic Cells.Recent novel approaches to limit oxidative stress and inflammation in diabetic complications.Therapeutic Potential of Annexin A1 in Ischemia Reperfusion Injury.
P2860

Reperfusion-induced myocardial dysfunction is prevented by endogenous annexin-A1 and its N-terminal-derived peptide Ac-ANX-A1(2-26).

Item
http://www.wikidata.org/entity/Q36603227
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Reperfusion-induced myocardial ...... rived peptide Ac-ANX-A1(2-26).
@ast
Reperfusion-induced myocardial ...... rived peptide Ac-ANX-A1(2-26).
@en
type
label
Reperfusion-induced myocardial ...... rived peptide Ac-ANX-A1(2-26).
@ast
Reperfusion-induced myocardial ...... rived peptide Ac-ANX-A1(2-26).
@en
prefLabel
Reperfusion-induced myocardial ...... rived peptide Ac-ANX-A1(2-26).
@ast
Reperfusion-induced myocardial ...... rived peptide Ac-ANX-A1(2-26).
@en
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Reperfusion-induced myocardial ...... erived peptide Ac-ANX-A1(2-26)
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10.1111/J.1476-5381.2012.02176.X
P407
P433
P478
P50
P577
P5875
P698
P932