Deletion of ADORA2B from myeloid cells dampens lung fibrosis and pulmonary hypertension
about
Attenuation of Pulmonary Ischemia-Reperfusion Injury by Adenosine A2B Receptor AntagonismAdenosine Attenuates Human Coronary Artery Smooth Muscle Cell Proliferation by Inhibiting Multiple Signaling Pathways That Converge on Cyclin D.Purinergic signaling in scarring.Extracellular adenosine levels are associated with the progression and exacerbation of pulmonary fibrosis.Prolyl-4 Hydroxylase 2 (PHD2) Deficiency in Endothelial Cells and Hematopoietic Cells Induces Obliterative Vascular Remodeling and Severe Pulmonary Arterial Hypertension in Mice and Humans Through Hypoxia-Inducible Factor-2α.Sphingosine-1-phosphate promotes erythrocyte glycolysis and oxygen release for adaptation to high-altitude hypoxia.Integrated analyses of gene expression and genetic association studies in a founder population.Erythrocytes retain hypoxic adenosine response for faster acclimatization upon re-ascentPulmonary Hypertension Associated with Idiopathic Pulmonary Fibrosis: Current and Future Perspectives.Modeling DNA damage-induced pneumopathy in mice: insight from danger signaling cascades.Pulmonary hypertension associated with lung diseases and hypoxemia.Pathological overproduction: the bad side of adenosine.Purinergic signalling links mechanical breath profile and alveolar mechanics with the pro-inflammatory innate immune response causing ventilation-induced lung injury.Plexin C1 deficiency permits synaptotagmin 7-mediated macrophage migration and enhances mammalian lung fibrosis.A novel mouse model of conditional IRAK-M deficiency in myeloid cells: application in lung Pseudomonas aeruginosa infection.Netrin-1 Regulates Fibrocyte Accumulation in the Decellularized Fibrotic Sclerodermatous Lung Microenvironment and in Bleomycin-Induced Pulmonary Fibrosis.Adenosine Receptors As Drug Targets for Treatment of Pulmonary Arterial Hypertension.HIF1A up-regulates the ADORA2B receptor on alternatively activated macrophages and contributes to pulmonary fibrosis.Adenosine metabolism, immunity and joint health.Adenosine Receptor-Mediated Cardioprotection-Current Limitations and Future Directions.Switching-Off Adora2b in Vascular Smooth Muscle Cells Halts the Development of Pulmonary Hypertension.Macrophages: friend or foe in idiopathic pulmonary fibrosis?
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P2860
Deletion of ADORA2B from myeloid cells dampens lung fibrosis and pulmonary hypertension
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
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2014年论文
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2014年论文
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name
Deletion of ADORA2B from myeloid cells dampens lung fibrosis and pulmonary hypertension
@ast
Deletion of ADORA2B from myeloid cells dampens lung fibrosis and pulmonary hypertension
@en
type
label
Deletion of ADORA2B from myeloid cells dampens lung fibrosis and pulmonary hypertension
@ast
Deletion of ADORA2B from myeloid cells dampens lung fibrosis and pulmonary hypertension
@en
prefLabel
Deletion of ADORA2B from myeloid cells dampens lung fibrosis and pulmonary hypertension
@ast
Deletion of ADORA2B from myeloid cells dampens lung fibrosis and pulmonary hypertension
@en
P2093
P2860
P50
P356
P1433
P1476
Deletion of ADORA2B from myeloid cells dampens lung fibrosis and pulmonary hypertension
@en
P2093
Fayong Luo
Holger K Eltzschig
Isabelle Bunge
Jonathan Davies
Jose G Molina
Kelly A Volcik
Luis F Acero
Ngoc-Bao Le
Ning-Yuan Chen
Richard A Johnston
P2860
P356
10.1096/FJ.14-260182
P407
P577
2014-10-15T00:00:00Z