Part of the series: from dietary antioxidants to regulators in cellular signaling and gene regulation. Sulforaphane and selenium, partners in adaptive response and prevention of cancer. - Wikidata - wikimedia - TriplyDB

Part of the series: from dietary antioxidants to regulators in cellular signaling and gene regulation. Sulforaphane and selenium, partners in adaptive response and prevention of cancer.

Part of the series: from dietary antioxidants to regulators in cellular signaling and gene regulation. Sulforaphane and selenium, partners in adaptive response and prevention of cancer.

http://www.wikidata.org/entity/Q36610404

about

Oxidative stress and age-related changes in T cells: is thalassemia a model of accelerated immune system aging?Glutathione- and thioredoxin-related enzymes are modulated by sulfur-containing chemopreventive agents Molecular targets of isothiocyanates in cancer: recent advances Iron behaving badly: inappropriate iron chelation as a major contributor to the aetiology of vascular and other progressive inflammatory and degenerative diseases Feeding of selenium alone or in combination with glucoraphanin differentially affects intestinal and hepatic antioxidant and phase II enzymes in growing rats.NRF2, cancer and calorie restriction Glutathione peroxidase-2 and selenium decreased inflammation and tumors in a mouse model of inflammation-associated carcinogenesis whereas sulforaphane effects differed with selenium supply.Toward understanding success and failures in the use of selenium for cancer prevention.Nrf2 signaling: an adaptive response pathway for protection against environmental toxic insults.Thiol chemistry in peroxidase catalysis and redox signaling.Sulforaphane Prevents Testicular Damage in Kunming Mice Exposed to Cadmium via Activation of Nrf2/ARE Signaling Pathways.Oxidative stress and therapeutic opportunities: focus on the Ewing's sarcoma family of tumors.The potential role of nutritional genomics tools in validating high health foods for cancer control: broccoli as example.Epigenetic and antioxidant effects of dietary isothiocyanates and selenium: potential implications for cancer chemoprevention.Chemopreventive Properties of Genipin on AGS Cell Line via Induction of JNK/Nrf2/ARE Signaling Pathway.Breakdown products of neoglucobrassicin inhibit activation of Nrf2 target genes mediated by myrosinase-derived glucoraphanin hydrolysis products.Activation of the glutathione peroxidase 2 (GPx2) promoter by beta-catenin.The yin and yang of nrf2-regulated selenoproteins in carcinogenesis.3,3'-Diindolylmethane but not indole-3-carbinol activates Nrf2 and induces Nrf2 target gene expression in cultured murine fibroblasts.The differential NF-kB modulation by S-adenosyl-L-methionine, N-acetylcysteine and quercetin on the promotion stage of chemical hepatocarcinogenesis.Dietary selenium affects selenoprotein W gene expression in the liver of chicken.H2O2 preconditioning modulates phase II enzymes through p38 MAPK and PI3K/Akt activation.An Encapsulated Fruit and Vegetable Juice Concentrate Increases Skin Microcirculation in Healthy Women

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P2860

Part of the series: from dietary antioxidants to regulators in cellular signaling and gene regulation. Sulforaphane and selenium, partners in adaptive response and prevention of cancer.

http://www.wikidata.org/entity/Q36610404

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2006 nî lūn-bûn

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Regina Brigelius-Flohé

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P2860

T RANSPORT B ETWEEN THE C ELL N UCLEUS AND THE C YTOPLASM

Ubiquitination of Keap1, a BTB-Kelch substrate adaptor protein for Cul3, targets Keap1 for degradation by a proteasome-independent pathway

The GI-GPx gene is a target for Nrf2

How selenium has altered our understanding of the genetic code

Keap1 is a redox-regulated substrate adaptor protein for a Cul3-dependent ubiquitin ligase complex.

Oxidative stress sensor Keap1 functions as an adaptor for Cul3-based E3 ligase to regulate proteasomal degradation of Nrf2

Keap1 represses nuclear activation of antioxidant responsive elements by Nrf2 through binding to the amino-terminal Neh2 domain

Most effective colon cancer chemopreventive agents in rats: a systematic review of aberrant crypt foci and tumor data, ranked by potency

Characterization of mammalian selenoproteomes.

Blocking tumor cell eicosanoid synthesis by GP x 4 impedes tumor growth and malignancy.

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