Immunopathogenesis of IBD: insufficient suppressor function in the gut? - Wikidata - wikimedia - TriplyDB

Immunopathogenesis of IBD: insufficient suppressor function in the gut?

Immunopathogenesis of IBD: insufficient suppressor function in the gut?

http://www.wikidata.org/entity/Q36626285

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Helminth infections and host immune regulation New insights into the role of fatty acids in the pathogenesis and resolution of inflammatory bowel disease Heme oxygenase-1 ameliorates dextran sulfate sodium-induced acute murine colitis by regulating Th17/Treg cell balance.Progress in the diagnosis and treatment of inflammatory bowel disease.Lactobacillus paracasei reduces intestinal inflammation in adoptive transfer mouse model of experimental colitis.RNA regulators of host immunity and pathogen adaptive responses in the oral cavity Immunopathogenesis of IBD: current state of the art.Pharmacological modulation of adenosine system: novel options for treatment of inflammatory bowel diseases.Intestinal dendritic cells and epithelial barrier dysfunction in Crohn's disease.Anti-inflammatory effect of recreational exercise in TNBS-induced colitis in rats: role of NOS/HO/MPO system.Gene and cell therapy based treatment strategies for inflammatory bowel diseases.CCL2 (pM levels) as a therapeutic agent in Inflammatory Bowel Disease models in mice.Reciprocal changes of Foxp3 expression in blood and intestinal mucosa in IBD patients responding to infliximab.Oral administration of carbonic anhydrase I ameliorates murine experimental colitis induced by Foxp3-CD4+CD25- T cells.Interleukin-10 promoter polymorphisms influence susceptibility to ulcerative colitis in a gender-specific manner.Apoptosis resistance of mucosal lymphocytes and IL-10 deficiency in patients with steroid-refractory Crohn's disease.

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P2860

Immunopathogenesis of IBD: insufficient suppressor function in the gut?

http://www.wikidata.org/entity/Q36626285

description

2006 nî lūn-bûn

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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2006年论文

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2006年论文

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name

Immunopathogenesis of IBD: insufficient suppressor function in the gut?

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Immunopathogenesis of IBD: insufficient suppressor function in the gut?

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Immunopathogenesis of IBD: insufficient suppressor function in the gut?

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Immunopathogenesis of IBD: insufficient suppressor function in the gut?

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Immunopathogenesis of IBD: insufficient suppressor function in the gut?

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Immunopathogenesis of IBD: insufficient suppressor function in the gut?

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Immunopathogenesis of IBD: insufficient suppressor function in the gut?

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A U van Lent

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I L Huibregtse

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S J H van Deventer

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P2860

A frameshift mutation in NOD2 associated with susceptibility to Crohn's disease

Crucial role of FOXP3 in the development and function of human CD25+CD4+ regulatory T cells

Toll-like receptor 8-mediated reversal of CD4+ regulatory T cell function

Essential role for CD103 in the T cell-mediated regulation of experimental colitis

Conversion of peripheral CD4+CD25- naive T cells to CD4+CD25+ regulatory T cells by TGF-beta induction of transcription factor Foxp3

Treatment of experimental (Trinitrobenzene sulfonic acid) colitis by intranasal administration of transforming growth factor (TGF)-beta1 plasmid: TGF-beta1-mediated suppression of T helper cell type 1 response occurs by interleukin (IL)-10 induction

Control of regulatory T cell development by the transcription factor Foxp3

Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn's disease

Host recognition of bacterial muramyl dipeptide mediated through NOD2. Implications for Crohn's disease

Targeted disruption of the mouse transforming growth factor-beta 1 gene results in multifocal inflammatory disease

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