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Clinicopathological Analysis and Multipronged Quantitative Proteomics Reveal Oxidative Stress and Cytoskeletal Proteins as Possible Markers for Severe Vivax Malaria.Structure and non-essential function of glycerol kinase inPlasmodium falciparumblood stagesCopper Homeostasis for the Developmental Progression of Intraerythrocytic Malarial ParasiteEvolutionary patterning: a novel approach to the identification of potential drug target sites in Plasmodium falciparumHost reticulocytes provide metabolic reservoirs that can be exploited by malaria parasitesPlasmodium falciparum Choline Kinase Inhibition Leads to a Major Decrease in Phosphatidylethanolamine Causing Parasite DeathPlasmodium falciparum phospholipase C hydrolyzing sphingomyelin and lysocholinephospholipids is a possible target for malaria chemotherapyThe neutral lipid composition present in the digestive vacuole of Plasmodium falciparum concentrates heme and mediates β-hematin formation with an unusually low activation energy.A systematic approach to understand the mechanism of action of the bisthiazolium compound T4 on the human malaria parasite, Plasmodium falciparumBiochemistry of Plasmodium (malarial parasites).Serum lipids and lipoproteins in malaria--a systematic review and meta-analysisQuaternary ammonium compounds efficiently inhibit Plasmodium falciparum growth in vitro by impairment of choline transportInward cholesterol gradient of the membrane system in P. falciparum-infected erythrocytes involves a dilution effect from parasite-produced lipids.Targeting the Lipid Metabolic Pathways for the Treatment of Malaria.Pharmacological properties of a new antimalarial bisthiazolium salt, T3, and a corresponding prodrug, TE3.A type II pathway for fatty acid biosynthesis presents drug targets in Plasmodium falciparum.Kinetic modelling of phospholipid synthesis in Plasmodium knowlesi unravels crucial steps and relative importance of multiple pathways.Liver Metabolic Alterations and Changes in Host Intercompartmental Metabolic Correlation during Progression of MalariaPotent inhibitors of Plasmodium phospholipid metabolism with a broad spectrum of in vitro antimalarial activities.Emerging role of lipid droplets in host/pathogen interactions.3-Hydroxy-3-methylglutaryl coenzyme A reductase inhibitors lovastatin and simvastatin inhibit in vitro development of Plasmodium falciparum and Babesia divergens in human erythrocytes.Jammed traffic impedes parasite growthCyclosporin A in Membrane Lipids Environment: Implications for Antimalarial Activity of the Drug--The Langmuir Monolayer Studies.Glycosylphosphatidylinositol anchors of Plasmodium falciparum: molecular characterization and naturally elicited antibody response that may provide immunity to malaria pathogenesis.Lipid traffic between high density lipoproteins and Plasmodium falciparum-infected red blood cells.Changes in lipid composition during sexual development of the malaria parasite Plasmodium falciparum.Disruption of the Plasmodium falciparum PfPMT gene results in a complete loss of phosphatidylcholine biosynthesis via the serine-decarboxylase-phosphoethanolamine-methyltransferase pathway and severe growth and survival defects.Comparative biology of intracellular parasitism.Rodent and nonrodent malaria parasites differ in their phospholipid metabolic pathways.Exploring Drug Targets in Isoprenoid Biosynthetic Pathway for Plasmodium falciparum.Isoprenoid biosynthesis in Plasmodium falciparum.In vivo antimalarial activities of mono- and bis quaternary ammonium salts interfering with Plasmodium phospholipid metabolismThe antimalarial drug mefloquine binds to membrane phospholipids.Parasite-regulated membrane transport processes and metabolic control in malaria-infected erythrocytes.Phospholipid metabolism of serine in Plasmodium-infected erythrocytes involves phosphatidylserine and direct serine decarboxylationAn intracellular simian malarial parasite (Plasmodium knowlesi) induces stage-dependent alterations in membrane phospholipid organization of its host erythrocyteNew insight into the mechanism of accumulation and intraerythrocytic compartmentation of albitiazolium, a new type of antimalarial.Toxoplasma gondii acyl-lipid metabolism: de novo synthesis from apicoplast-generated fatty acids versus scavenging of host cell precursors.Fatty acid synthesis and pyruvate metabolism pathways remain active in dihydroartemisinin-induced dormant ring stages of Plasmodium falciparumIncreased choline transport in erythrocytes from mice infected with the malaria parasite Plasmodium vinckei vinckei.
P2860
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P2860
description
1977 nî lūn-bûn
@nan
1977年の論文
@ja
1977年論文
@yue
1977年論文
@zh-hant
1977年論文
@zh-hk
1977年論文
@zh-mo
1977年論文
@zh-tw
1977年论文
@wuu
1977年论文
@zh
1977年论文
@zh-cn
name
Lipids and the malarial parasite.
@ast
Lipids and the malarial parasite.
@en
type
label
Lipids and the malarial parasite.
@ast
Lipids and the malarial parasite.
@en
prefLabel
Lipids and the malarial parasite.
@ast
Lipids and the malarial parasite.
@en
P2860
P1476
Lipids and the malarial parasite.
@en
P2093
Holz GG Jr
P2860
P304
P407
P577
1977-01-01T00:00:00Z