The FoxO3a gene is a key negative target of canonical Notch signalling in the keratinocyte UVB response.
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The impact of UVB exposure and differentiation state of primary keratinocytes on their interaction with quantum dotsNotch gain of function in mouse periocular mesenchyme downregulates FoxL2 and impairs eyelid levator muscle formation, leading to congenital blepharophimosis.Noncanonical NOTCH signaling limits self-renewal of human epithelial and induced pluripotent stem cells through ROCK activation.Small GTPase RhoE/Rnd3 is a critical regulator of Notch1 signaling.Differential control of Notch1 gene transcription by Klf4 and Sp3 transcription factors in normal versus cancer-derived keratinocytes.The inhibition of NOTCH2 reduces UVB-induced damage in retinal pigment epithelium cells.Nrf2 establishes a glutathione-mediated gradient of UVB cytoprotection in the epidermis.Effects of Korean ginseng berry on skin antipigmentation and antiaging via FoxO3a activation.DDR1 receptor tyrosine kinase promotes prosurvival pathway through Notch1 activation.Resveratrol prevents oxidative stress-induced senescence and proliferative dysfunction by activating the AMPK-FOXO3 cascade in cultured primary human keratinocytes.Notch Cooperates with Survivin to Maintain Stemness and to Stimulate Proliferation in Human Keratinocytes during Ageing.Multifocal epithelial tumors and field cancerization from loss of mesenchymal CSL signaling.Squamous Cell Cancers: A Unified Perspective on Biology and GeneticsManganese superoxide dismutase promotes anoikis resistance and tumor metastasisA miR-34a-SIRT6 axis in the squamous cell differentiation networkDevelopmental SHP2 dysfunction underlies cardiac hypertrophy in Noonan syndrome with multiple lentigines.Nitric oxide-releasing sulindac is a novel skin cancer chemopreventive agent for UVB-induced photocarcinogenesis.Notch tumor suppressor functionFOXO3a regulates oxygen-responsive expression of tumor necrosis factor receptor 2 in human dermal microvascular endothelial cells.EGFR signalling as a negative regulator of Notch1 gene transcription and function in proliferating keratinocytes and cancer.Cisplatin selects for stem-like cells in osteosarcoma by activating Notch signaling.A positive FGFR3/FOXN1 feedback loop underlies benign skin keratosis versus squamous cell carcinoma formation in humans.Developmental pathways in colon cancer: crosstalk between WNT, BMP, Hedgehog and Notch.Does therapeutic intervention in atopic dermatitis normalize epidermal Notch deficiency?Opposing functions of Fbxw7 in keratinocyte growth, differentiation and skin tumorigenesis mediated through negative regulation of c-Myc and Notch.
P2860
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P2860
The FoxO3a gene is a key negative target of canonical Notch signalling in the keratinocyte UVB response.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
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2008年论文
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2008年论文
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name
The FoxO3a gene is a key negat ...... the keratinocyte UVB response.
@ast
The FoxO3a gene is a key negat ...... the keratinocyte UVB response.
@en
type
label
The FoxO3a gene is a key negat ...... the keratinocyte UVB response.
@ast
The FoxO3a gene is a key negat ...... the keratinocyte UVB response.
@en
prefLabel
The FoxO3a gene is a key negat ...... the keratinocyte UVB response.
@ast
The FoxO3a gene is a key negat ...... the keratinocyte UVB response.
@en
P2093
P2860
P356
P1433
P1476
The FoxO3a gene is a key negat ...... the keratinocyte UVB response
@en
P2093
Anna Mandinova
G Paolo Dotto
Haruhi Iwaki
Jotaro Nakanishi
Karine Lefort
Paola Ostano
Wesley Stonely
P2860
P304
P356
10.1038/EMBOJ.2008.45
P407
P577
2008-04-03T00:00:00Z