A prospective analysis of lymphocyte phenotype and function over the course of acute sepsis. - Wikidata - wikimedia - TriplyDB

A prospective analysis of lymphocyte phenotype and function over the course of acute sepsis.

A prospective analysis of lymphocyte phenotype and function over the course of acute sepsis.

http://www.wikidata.org/entity/Q36633756

about

Monitoring the immune response in sepsis: a rational approach to administration of immunoadjuvant therapies Immunosuppression after sepsis: systemic inflammation and sepsis induce a loss of naïve T-cells but no enduring cell-autonomous defects in T-cell function Increased CD4(+) T cell co-inhibitory immune receptor CEACAM1 in neonatal sepsis and soluble-CEACAM1 in meningococcal sepsis: a role in sepsis-associated immune suppression?Early alterations of B cells in patients with septic shock: another piece in the complex puzzle of the immune response in sepsis T-cell Co-inhibitory Molecules in Sepsis-induced Immunosuppression: From Bench to Bedside.Early alterations of B cells in patients with septic shock.Cutting edge: critical role for C5aRs in the development of septic lymphopenia in mice.Full activation of CD4+ T cells early during sepsis requires specific antigen.Altered immune status of circulating T lymphocytes during sepsis: children also.Altered T Lymphocyte Proliferation upon Lipopolysaccharide Challenge Ex Vivo.The pediatric sepsis biomarker risk model: potential implications for sepsis therapy and biology.Soluble B and T Lymphocyte Attenuator Correlates to Disease Severity in Sepsis and High Levels Are Associated with an Increased Risk of Mortality Increased PD-1 Expression and Altered T Cell Repertoire Diversity Predict Mortality in Patients with Septic Shock: A Preliminary Study.A rapidly progressing lymphocyte exhaustion after severe sepsis.Sepsis-induced long-term immune paralysis--results of a descriptive, explorative study.The role of natural killer cells in the pathogenesis of sepsis: the ongoing enigma.Immunosuppression in sepsis: a novel understanding of the disorder and a new therapeutic approach Traumatic brain injury-induced alterations in peripheral immunity The changing immune system in sepsis: is individualized immuno-modulatory therapy the answer?Heterogeneity among septic shock patients in a set of immunoregulatory markers.Persistent lymphopenia is a risk factor for ICU-acquired infections and for death in ICU patients with sustained hypotension at admission.Pathways mediating resolution of inflammation: when enough is too much.Pathophysiologic mechanisms in septic shock.Sepsis-induced immune alterations monitoring by flow cytometry as a promising tool for individualized therapy.Persistent lymphopenia is an independent predictor of mortality in critically ill emergency general surgical patients.Human CD56bright NK Cells: An Update.Chronic Critical Illness from Sepsis Is Associated with an Enhanced TCR Response.Immunotherapy: A promising approach to reverse sepsis-induced immunosuppression.Functional characterization of T-cells from palatine tonsils in patients with chronic tonsillitis.Increased Expression of Cytotoxic T-Lymphocyte-Associated Protein 4 by T Cells, Induced by B7 in Sera, Reduces Adaptive Immunity in Patients With Acute Liver Failure.Frontline Science: Defects in immune function in patients with sepsis are associated with PD-1 or PD-L1 expression and can be restored by antibodies targeting PD-1 or PD-L1.The effect of tadalafil therapy on kidney damage caused by sepsis in a polymicrobial septic model induced in rats: a biochemical and histopathological study Sepsis in PD-1 light.BTLA as a biomarker and mediator of sepsis-induced immunosuppression.Inflammation-Induced Changes in Circulating T-Cell Subsets and Cytokine Production During Human Endotoxemia.[Epigenetic regulation in sepsis : current state of knowledge].IL-7 Restores T Lymphocyte Immunometabolic Failure in Septic Shock Patients through mTOR Activation.[Interleukin-7, a new immunoadjuvant for the treatment of septic shock].Targeting Immune Cell Checkpoints during Sepsis.Ex vivo Stimulation of Lymphocytes with IL-10 Mimics Sepsis-Induced Intrinsic T-Cell Alterations.

P2860

Q26852697-874EF389-EA28-472A-AC32-1B178E4DE70E Q27320494-A34B9A37-8511-44A6-8C60-1E2A3D27F967 Q31124179-3BDAA096-A9D5-4095-A08F-D0689C50E24A Q33356168-02218B2C-A766-4110-A488-07517CDB4830 Q33723244-EC1ADECA-CF44-4C6E-A0A9-FEC20F16B366 Q33752795-431AC64F-E795-4785-AC01-3C9CF59A3634 Q34980303-748C1C5C-7215-44F5-BC6B-ABEDF431FECC Q35013590-7C06DAD6-9DF9-4948-AE32-29636CECC6CD Q35580678-D676F8B0-49F5-426E-B1F6-CF548C40CD26 Q35862468-7AE1B98C-EC16-4CDD-A7E1-9E954EA68D7A Q36181988-2715E259-7E36-4B5C-BDA4-5C7FD5A8C4AF Q36240333-68ACE157-D6E1-48AA-9B74-0A202F3E7152 Q36245574-6EF52F0F-9375-4A01-9D36-969FFFA8FBF8 Q36633849-78D426E5-3955-4549-A561-8D60E57E5763 Q36774624-60501592-050E-423C-8920-0833BDC3CA5E Q36902795-689DD0AD-3F2C-41CC-94DB-FFAFDF57980B Q37233354-E15111AD-6B33-4F71-9C1B-13DB15CD5302 Q37257295-C9269E5F-87C0-4FA2-ACF4-1C854197CFD9 Q37563023-21A3EED3-1E98-47EE-8F4C-6BAE18583EED Q37585971-DE8990C0-CC7C-4389-BDC7-92D024A7114B Q37707179-DDE9CAD6-B3D3-4D26-B958-52691482423F Q38116494-96BA78F2-E40A-4274-A1DB-932A18F76FB3 Q38141738-A56ACB83-EDF1-412C-9821-2733E39475AA Q38541110-A79D4972-BBA8-4E05-8EA0-CB94A5269719 Q38616352-2F49A214-9AE6-48E5-A801-75BDFFF409EA Q38781384-F0ECB534-63FF-4048-9FAE-8C5946D14DFD Q38796965-B4C2AB8D-E3E7-428E-B732-BE3AC156DBF2 Q38836832-2C7D6EDB-828C-493F-B7B7-0B56C5D226ED Q40055705-1898B2FF-9BCD-48DC-9A37-B05742ED4BC3 Q40270051-5E30065F-3B92-4C64-BF7F-077B6805D8C5 Q40526734-1ED163CA-D9FB-43CD-A34A-DE4346B031E2 Q40542339-661AE0FA-83FC-4978-9927-AF1946E13BBF Q40625575-0175208F-1091-4F60-90DF-BA0FA13FE3F8 Q40763516-35F95954-EE90-4B23-A4E5-9AF4A1B6942A Q40988581-9D2B6099-97CD-491F-9A5D-3884FE5CBBAE Q41620454-9ABE76D4-F53D-4DCE-96A2-7B726E89FAEA Q42512567-FD434A54-E525-4380-A0F2-FE82AC6A9500 Q45241698-AFF185B3-00A7-4C4A-8847-D0A4D72386A8 Q47150101-C860B2B1-EC8B-44E6-8570-75BE362277C2 Q47226688-5E40E42E-8CB6-4645-A8EA-BA07E0964273

P2860

A prospective analysis of lymphocyte phenotype and function over the course of acute sepsis.

http://www.wikidata.org/entity/Q36633756

description

2012 nî lūn-bûn

@nan

2012年の論文

@ja

2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

2012年论文

@zh

2012年论文

@zh-cn

name

A prospective analysis of lymp ...... er the course of acute sepsis.

@ast

A prospective analysis of lymp ...... er the course of acute sepsis.

@en

type

label

A prospective analysis of lymp ...... er the course of acute sepsis.

@ast

A prospective analysis of lymp ...... er the course of acute sepsis.

@en

prefLabel

A prospective analysis of lymp ...... er the course of acute sepsis.

@ast

A prospective analysis of lymp ...... er the course of acute sepsis.

@en

P1433

Q36633756-B0E5079D-4089-4A47-AAE7-75FF63E09CF5

P1476

Q36633756-C878E783-FE10-44F3-AE5A-666663396DE4

P2093

Q36633756-2394AC7E-D435-4F2C-974C-7CCB22C0A1BB

Q36633756-52197C9D-B719-4B3B-AF1F-B8F280DCBAF7

Q36633756-F3B5B258-81CC-4012-B1B5-DB6D483DA58E

P2860

Q36633756-089120CA-E510-4572-A738-B38DFA9DEB49

Q36633756-0AF07971-73D7-4EC8-A34D-A22E3E919DED

Q36633756-1CB20B2E-9418-4D8B-8DA8-6B9F358FE89A

Q36633756-20C6533F-5C8F-488B-99FC-85CC9EC246E5

Q36633756-218A35BC-EDE2-4032-93E4-2865C4A64FB0

Q36633756-280C68A6-F908-4177-BF60-420F74FD7E58

Q36633756-2DD6F395-9FD4-49C4-94EE-899D7F6E9B28

Q36633756-33062B08-942B-4346-93CF-0325E7FC524B

Q36633756-3A16D502-E26D-459A-9C5E-0AED747A0102

Q36633756-3B541241-FFA7-4B79-8A30-AE73EC7DB82E

P2888

Q36633756-AF9C89AA-D8B7-434F-BCE0-3997DFB572A1

P304

Q36633756-630EADE3-B6B8-40E8-84CD-7DE0BE46570C

P31

Q36633756-73EE01C6-7549-4424-AC85-B80347208568

P356

Q36633756-C8FD52F8-0EEE-491C-95C2-A68527E517B0

P433

Q36633756-0960E710-77F8-440C-854F-D8759349E786

P478

Q36633756-B6D71882-1251-4578-A8FA-D3C983D0D97D

P50

Q36633756-A1BC44AC-0CB0-4ABF-8B66-557F7CB71830

P577

Q36633756-4D15587B-227F-4802-A1A1-D5780F4DB986

P5875

Q36633756-70277515-0C6A-4368-99B1-2C913EB05086

P6179

Q36633756-2B110B29-1403-4982-807C-DF25BD7E8936

P698

Q36633756-9F6F30AF-CE01-476C-BADB-A6B60BDF2228

P921

Q36633756-6034D703-11D5-4956-8B1B-25B3E35282BC

P932

Q36633756-EE6B7014-3970-4774-BB00-85645DC42FAB

P356

P1433

P1476

A prospective analysis of lymp ...... er the course of acute sepsis.

@en

P2093

Jennifer Shuherk-Shaffer

http://www.w3.org/2001/XMLSchema#string

Jonathan M Green

http://www.w3.org/2001/XMLSchema#string

Jonathan S Boomer

http://www.w3.org/2001/XMLSchema#string

P2860

Engagement of the PD-1 immunoinhibitory receptor by a novel B7 family member leads to negative regulation of lymphocyte activation

PD-1 Expression in Acute Hepatitis C Virus (HCV) Infection Is Associated with HCV-Specific CD8 Exhaustion

Liver-Infiltrating Lymphocytes in Chronic Human Hepatitis C Virus Infection Display an Exhausted Phenotype with High Levels of PD-1 and Low Levels of CD127 Expression

Myeloid-derived suppressor cells as regulators of the immune system

Restoring function in exhausted CD8 T cells during chronic viral infection

2001 SCCM/ESICM/ACCP/ATS/SIS International Sepsis Definitions Conference

PD-1 expression on HIV-specific T cells is associated with T-cell exhaustion and disease progression

T cell exhaustion

The immunopathogenesis of sepsis

Both percentage of gammadelta T lymphocytes and CD3 expression are reduced during septic shock.

P2888

P304

R112

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1186/CC11404

http://www.w3.org/2001/XMLSchema#string

P433

3

http://www.w3.org/2001/XMLSchema#string

P478

16

http://www.w3.org/2001/XMLSchema#string

P50

Richard S Hotchkiss

P577

2012-06-28T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

228086837

http://www.w3.org/2001/XMLSchema#string

P6179

1039561743

http://www.w3.org/2001/XMLSchema#string

P698

22742734

http://www.w3.org/2001/XMLSchema#string

P921

P932

3580670

http://www.w3.org/2001/XMLSchema#string