Respiratory virus infection of mice provokes a permanent humoral immune response.
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Role of different lymphoid tissues in the initiation and maintenance of DNA-raised antibody responses to the influenza virus H1 glycoproteinHow specific is too specific? B-cell responses to viral infections reveal the importance of breadth over depthPulmonary infection with influenza A virus induces site-specific germinal center and T follicular helper cell responsesHeterologous Prime-Boost HIV-1 Vaccination Regimens in Pre-Clinical and Clinical TrialsHeterologous prime-boost vaccination with MF59-adjuvanted H5 vaccines promotes antibody affinity maturation towards the hemagglutinin HA1 domain and broad H5N1 cross-clade neutralizationOral priming with replicating adenovirus serotype 4 followed by subunit H5N1 vaccine boost promotes antibody affinity maturation and expands H5N1 cross-clade neutralization.Protective antiviral antibody responses in a mouse model of influenza virus infection require TACIDifferential impact of ageing on cellular and humoral immunity to a persistent murine gamma-herpesvirusDissecting the host response to a gamma-herpesvirus.Role of interleukin-12 in primary influenza virus infectionNasal-associated lymphoid tissue is a site of long-term virus-specific antibody production following respiratory virus infection of mice.The extent of affinity maturation differs between the memory and antibody-forming cell compartments in the primary immune response.Quantitative analysis of the CD8+ T-cell response to readily eliminated and persistent virusesRobust IgA and IgG-producing antibody forming cells in the diffuse-NALT and lungs of Sendai virus-vaccinated cotton rats associate with rapid protection against human parainfluenza virus-type 1Coinfection with Streptococcus pneumoniae modulates the B cell response to influenza virusClonally related CD8+ T cells responsible for rapid population of both diffuse nasal-associated lymphoid tissue and lung after respiratory virus infectionProtective B cell responses to flu--no fluke!Contributions of humoral and cellular immunity to vaccine-induced protection in humans.Sendai virus-based RSV vaccine protects African green monkeys from RSV infection.Quantitative analysis of herpes simplex virus type 1-specific memory B cells generated by different routes of infection.Host response to Sendai virus in mice lacking class II major histocompatibility complex glycoproteins.Bone marrow is a major site of long-term antibody production after acute viral infectionSelection of a single amino acid substitution in the hemagglutinin molecule by chicken eggs can render influenza A virus (H3) candidate vaccine ineffectiveThe B-cell response in lymphoid tissue of mice immunized with various antigenic forms of the influenza virus hemagglutinin.Differential antigen burden modulates the gamma interferon but not the immunoglobulin response in mice that vary in susceptibility to Sendai virus pneumoniaAntibody-forming cell response to virus challenge in mice immunized with DNA encoding the influenza virus hemagglutininImmune CD4+ T cells promote the clearance of influenza virus from major histocompatibility complex class II -/- respiratory epitheliumAntigenic Fingerprinting following Primary RSV Infection in Young Children Identifies Novel Antigenic Sites and Reveals Unlinked Evolution of Human Antibody Repertoires to Fusion and Attachment Glycoproteins.Broad dispersion and lung localization of virus-specific memory B cells induced by influenza pneumonia.Inability to evoke a long-lasting protective immune response to respiratory syncytial virus infection in mice correlates with ineffective nasal antibody responsesDifferential localization and function of antibody-forming cells responsive to inactivated or live-attenuated influenza virus vaccinesSendai virus recombinant vaccine expressing hPIV-3 HN or F elicits protective immunity and combines with a second recombinant to prevent hPIV-1, hPIV-3 and RSV infectionsHuman PIV-2 recombinant Sendai virus (rSeV) elicits durable immunity and combines with two additional rSeVs to protect against hPIV-1, hPIV-2, hPIV-3, and RSV.Local and systemic antibody responses in mice immunized intranasally with native and detergent-extracted outer membrane vesicles from Neisseria meningitidisAntibody-secreting cells in respiratory tract tissues in the absence of eosinophils as supportive partnersMyeloid depression follows infection of susceptible newborn mice with the parvovirus minute virus of mice (strain i).The cytotoxic T-lymphocyte response to Sendai virus is unimpaired in the absence of gamma interferon.Multi-Envelope HIV-1 Vaccine Development: Two Targeted Immune Pathways, One Desired Protective Outcome.Antibody response to influenza infection of mice: different patterns for glycoprotein and nucleocapsid antigens.Respiratory syncytial virus vaccine development
P2860
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P2860
Respiratory virus infection of mice provokes a permanent humoral immune response.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
Respiratory virus infection of mice provokes a permanent humoral immune response.
@ast
Respiratory virus infection of mice provokes a permanent humoral immune response.
@en
type
label
Respiratory virus infection of mice provokes a permanent humoral immune response.
@ast
Respiratory virus infection of mice provokes a permanent humoral immune response.
@en
prefLabel
Respiratory virus infection of mice provokes a permanent humoral immune response.
@ast
Respiratory virus infection of mice provokes a permanent humoral immune response.
@en
P2093
P2860
P1433
P1476
Respiratory virus infection of mice provokes a permanent humoral immune response.
@en
P2093
P2860
P304
P407
P577
1994-09-01T00:00:00Z