Hematopoietic stem cell and progenitor cell mechanisms in myelodysplastic syndromes. - Wikidata - wikimedia - TriplyDB

Hematopoietic stem cell and progenitor cell mechanisms in myelodysplastic syndromes.

Hematopoietic stem cell and progenitor cell mechanisms in myelodysplastic syndromes.

http://www.wikidata.org/entity/Q36637556

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Somatic CALR mutations in myeloproliferative neoplasms with nonmutated JAK2 Bone Marrow Immunity and Myelodysplasia Mesenchymal Stem and Progenitor Cells in Normal and Dysplastic Hematopoiesis-Masters of Survival and Clonality?Revisiting the case for genetically engineered mouse models in human myelodysplastic syndrome research Deregulation of innate immune and inflammatory signaling in myelodysplastic syndromes MDS prognostic scoring systems – past, present, and future Deletion of Asxl1 results in myelodysplasia and severe developmental defects in vivo Stem cells are units of natural selection for tissue formation, for germline development, and in cancer development Stress-induced hematopoietic failure in the absence of immediate early response gene X-1 (IEX-1, IER3)Eltrombopag for the treatment of thrombocytopenia in patients with malignant and non-malignant hematologic disorders.Anti-CD47 Antibody As a Targeted Therapeutic Agent for Human Lung Cancer and Cancer Stem Cells.Quiescent hematopoietic stem cells accumulate DNA damage during aging that is repaired upon entry into cell cycle A humanized bone marrow ossicle xenotransplantation model enables improved engraftment of healthy and leukemic human hematopoietic cells.An MDS xenograft model utilizing a patient-derived cell line.Clonal evolution in myelodysplastic syndromes The Role of Efferocytosis in Atherosclerosis.Notch signaling in the malignant bone marrow microenvironment: implications for a niche-based model of oncogenesis.Feedback signals in myelodysplastic syndromes: increased self-renewal of the malignant clone suppresses normal hematopoiesis.Mutant U2AF1 Expression Alters Hematopoiesis and Pre-mRNA Splicing In Vivo.IL8-CXCR2 pathway inhibition as a therapeutic strategy against MDS and AML stem cells BMI1 reprogrammes histone acetylation and enhances c-fos pathway via directly binding to Zmym3 in malignant myeloid progression Prospective isolation of human erythroid lineage-committed progenitors.Alkylator-Induced and Patient-Derived Xenograft Mouse Models of Therapy-Related Myeloid Neoplasms Model Clinical Disease and Suggest the Presence of Multiple Cell Subpopulations with Leukemia Stem Cell Activity Normal Hematopoietic Progenitor Subsets Have Distinct Reactive Oxygen Species, BCL2 and Cell-Cycle Profiles That Are Decoupled from Maturation in Acute Myeloid Leukemia Apoptosis-Related Gene Expression Profiling in Hematopoietic Cell Fractions of MDS Patients.SF3B1 mutant MDS-initiating cells may arise from the haematopoietic stem cell compartment.Histiocytoid Sweet's syndrome in a patient with myelodsyplastic syndrome: report and review of the literature CD47-blocking immunotherapies stimulate macrophage-mediated destruction of small-cell lung cancer.Gene Expression and Methylation Pattern in HRK Apoptotic Gene in Myelodysplastic Syndrome CD47-blocking antibodies restore phagocytosis and prevent atherosclerosis.PUMA promotes apoptosis of hematopoietic progenitors driving leukemic progression in a mouse model of myelodysplasia.Myeloid Cell Origins, Differentiation, and Clinical Implications.B-myb is an essential regulator of hematopoietic stem cell and myeloid progenitor cell development.Downregulation of MMP1 in MDS-derived mesenchymal stromal cells reduces the capacity to restrict MDS cell proliferation.The bone marrow niche, stem cells, and leukemia: impact of drugs, chemicals, and the environment.Standardizing the initial evaluation for myelodysplastic syndromes.Stem cell origin of myelodysplastic syndromes.Higher Risk Myelodysplastic Syndromes in Patients with Well-Controlled HIV Infection: Clinical Features, Treatment, and Outcome.Ezatiostat hydrochloride for the treatment of myelodysplastic syndromes.Molecular mechanisms of the progression of myelodysplastic syndrome to secondary acute myeloid leukaemia and implication for therapy.

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Hematopoietic stem cell and progenitor cell mechanisms in myelodysplastic syndromes.

http://www.wikidata.org/entity/Q36637556

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Hematopoietic stem cell and progenitor cell mechanisms in myelodysplastic syndromes.

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Hematopoietic stem cell and progenitor cell mechanisms in myelodysplastic syndromes.

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Hematopoietic stem cell and progenitor cell mechanisms in myelodysplastic syndromes.

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Hematopoietic stem cell and progenitor cell mechanisms in myelodysplastic syndromes.

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Hematopoietic stem cell and progenitor cell mechanisms in myelodysplastic syndromes.

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Hematopoietic stem cell and progenitor cell mechanisms in myelodysplastic syndromes.

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Proceedings of the National Academy of Sciences of the United States of America

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Hematopoietic stem cell and progenitor cell mechanisms in myelodysplastic syndromes.

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Christopher Y Park

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Daniel A Arber

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Elizabeth A Price

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Irving L Weissman

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John V Pluvinage

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Kunju Sridhar

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Peter L Greenberg

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Stanley L Schrier

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Wendy W Pang

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P2860

Calreticulin is the dominant pro-phagocytic signal on multiple human cancers and is counterbalanced by CD47

Identification of a hierarchy of multipotent hematopoietic progenitors in human cord blood

Identification, molecular characterization, clinical prognosis, and therapeutic targeting of human bladder tumor-initiating cells

CD47 is upregulated on circulating hematopoietic stem cells and leukemia cells to avoid phagocytosis

Development of functional human blood and immune systems in NOD/SCID/IL2 receptor {gamma} chain(null) mice

Low density lipoprotein receptor-related protein is a calreticulin coreceptor that signals focal adhesion disassembly

AML1/ETO-expressing nonleukemic stem cells in acute myelogenous leukemia with 8;21 chromosomal translocation

Stem cells, cancer, and cancer stem cells

International scoring system for evaluating prognosis in myelodysplastic syndromes

Granulocyte-macrophage progenitors as candidate leukemic stem cells in blast-crisis CML

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