TGF-β inhibition enhances chemotherapy action against triple-negative breast cancer. - Wikidata - wikimedia - TriplyDB

TGF-β inhibition enhances chemotherapy action against triple-negative breast cancer.

TGF-β inhibition enhances chemotherapy action against triple-negative breast cancer.

http://www.wikidata.org/entity/Q36638173

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Validation of the effects of TGF-β1 on tumor recurrence and prognosis through tumor retrieval and cell mechanical properties Anti-Cancer Stem-like Cell Compounds in Clinical Development - An Overview and Critical Appraisal Cancer Stem Cell Plasticity Drives Therapeutic Resistance New Findings on Breast Cancer Stem Cells: A Review The fate of chemoresistance in triple negative breast cancer (TNBC)Common stemness regulators of embryonic and cancer stem cells Aldehyde dehydrogenase as a marker and functional mediator of metastasis in solid tumors Clinical development of galunisertib (LY2157299 monohydrate), a small molecule inhibitor of transforming growth factor-beta signaling pathway Increased Oxidative Stress as a Selective Anticancer Therapy Identifying and targeting tumor-initiating cells in the treatment of breast cancer Can nanomedicines kill cancer stem cells?Wound healing and cancer stem cells: inflammation as a driver of treatment resistance in breast cancer In vitro models of cancer stem cells and clinical applications Transforming growth factor β as regulator of cancer stemness and metastasis Therapeutic targets of triple-negative breast cancer: a review Epigenetic coordination of signaling pathways during the epithelial-mesenchymal transition Combining antibody-directed presentation of IL-15 and 4-1BBL in a trifunctional fusion protein for cancer immunotherapy.The miR-424(322)/503 cluster orchestrates remodeling of the epithelium in the involuting mammary gland.Aurora-A mitotic kinase induces endocrine resistance through down-regulation of ERα expression in initially ERα+ breast cancer cells.Two faces of TGF-beta1 in breast cancer.Tumour suppressor EP300, a modulator of paclitaxel resistance and stemness, is downregulated in metaplastic breast cancer.Triple-negative breast cancer: challenges and opportunities of a heterogeneous disease.7-difluoromethoxyl-5,4'-di-n-octyl genistein inhibits ovarian cancer stem cell characteristics through the downregulation of FOXM1.SHOX2 is a direct miR-375 target and a novel epithelial-to-mesenchymal transition inducer in breast cancer cells Oncostatin M promotes cancer cell plasticity through cooperative STAT3-SMAD3 signaling.Concise review: breast cancer stem cells: regulatory networks, stem cell niches, and disease relevance NF-κB affects proliferation and invasiveness of breast cancer cells by regulating CD44 expression.Novel clinical therapeutics targeting the epithelial to mesenchymal transition TGFβ induces "BRCAness" and sensitivity to PARP inhibition in breast cancer by regulating DNA-repair genes Tackling the cancer stem cells - what challenges do they pose?Binding of a Smad4/Ets-1 complex to a novel intragenic regulatory element in exon12 of FPGS underlies decreased gene expression and antifolate resistance in leukemia Hypoxia-inducible factors are required for chemotherapy resistance of breast cancer stem cells Melanoma-initiating cells exploit M2 macrophage TGFβ and arginase pathway for survival and proliferation.Metformin inhibits the IL-6-induced epithelial-mesenchymal transition and lung adenocarcinoma growth and metastasis.The repressive effect of miR-148a on TGF beta-SMADs signal pathway is involved in the glabridin-induced inhibition of the cancer stem cells-like properties in hepatocellular carcinoma cells The anticancer effect of (1S,2S,3E,7E,11E)-3,7,11, 15-cembratetraen-17,2-olide(LS-1) through the activation of TGF-β signaling in SNU-C5/5-FU, fluorouracil-resistant human colon cancer cells.Keratoconus in vitro and the key players of the TGF-β pathway.Blockage of TGFβ-SMAD2 by demethylation-activated miR-148a is involved in caffeic acid-induced inhibition of cancer stem cell-like properties in vitro and in vivo.Nuclear basic fibroblast growth factor regulates triple-negative breast cancer chemo-resistance Heat shock factor 1 induces cancer stem cell phenotype in breast cancer cell lines

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P2860

TGF-β inhibition enhances chemotherapy action against triple-negative breast cancer.

http://www.wikidata.org/entity/Q36638173

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2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

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name

TGF-β inhibition enhances chemotherapy action against triple-negative breast cancer.

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TGF-β inhibition enhances chemotherapy action against triple-negative breast cancer.

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type

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TGF-β inhibition enhances chemotherapy action against triple-negative breast cancer.

@ast

TGF-β inhibition enhances chemotherapy action against triple-negative breast cancer.

@en

prefLabel

TGF-β inhibition enhances chemotherapy action against triple-negative breast cancer.

@ast

TGF-β inhibition enhances chemotherapy action against triple-negative breast cancer.

@en

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Journal of Clinical Investigation

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TGF-β inhibition enhances chemotherapy action against triple-negative breast cancer.

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Carlos L Arteaga

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Jamie Stanford

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María Gabriela Kuba

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Melinda Sanders

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Neil E Bhola

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Rebecca S Cook

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Teresa C Dugger

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Violeta Sánchez

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P2860

SB-431542 is a potent and specific inhibitor of transforming growth factor-beta superfamily type I activin receptor-like kinase (ALK) receptors ALK4, ALK5, and ALK7

Breast cancer bone metastasis mediated by the Smad tumor suppressor pathway

CXCR1 blockade selectively targets human breast cancer stem cells in vitro and in xenografts

A collection of breast cancer cell lines for the study of functionally distinct cancer subtypes

The epithelial-mesenchymal transition generates cells with properties of stem cells

ALDH1 is a marker of normal and malignant human mammary stem cells and a predictor of poor clinical outcome

Prospective identification of tumorigenic breast cancer cells

TGFbeta primes breast tumors for lung metastasis seeding through angiopoietin-like 4

Molecular definition of breast tumor heterogeneity

Cancer stem cells in solid tumours: accumulating evidence and unresolved questions

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Justin M. Balko

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