IgM contributes to glomerular injury in FSGS - Wikidata - wikimedia - TriplyDB

IgM contributes to glomerular injury in FSGS

IgM contributes to glomerular injury in FSGS

http://www.wikidata.org/entity/Q36638347

about

Immunologic Changes Implicated in the Pathogenesis of Focal Segmental Glomerulosclerosis Histological changes of kidney in diabetic nephropathy Molecules Great and Small: The Complement System.Remission of aHUS neurological damage with eculizumab Serum C3 and Renal Outcome in Patients with Primary Focal Segmental Glomerulosclerosis.Classical Complement Pathway Activation in the Kidneys of Women With Preeclampsia.Complement Activation in Patients with Focal Segmental Glomerulosclerosis.Plasma Levels of Risk-Variant APOL1 Do Not Associate with Renal Disease in a Population-Based Cohort IgM exacerbates glomerular disease progression in complement-induced glomerulopathy Significance of the urokinase-type plasminogen activator and its receptor in the progression of focal segmental glomerulosclerosis in clinical and mouse models.Focal segmental glomerulosclerosis lagged behind the onset of rheumatoid arthritis by 7 years: A case report and literature review.The pathogenesis of focal segmental glomerulosclerosis.IgM as a novel predictor of disease progression in secondary focal segmental glomerulosclerosis IgM nephropathy: is it closer to minimal change disease or to focal segmental glomerulosclerosis?Focal Segmental Glomerulosclerosis.Association of apoptosis inhibitor of macrophage (AIM) expression with urinary protein and kidney dysfunction.World Small Animal Veterinary Association Renal Pathology Initiative: Classification of Glomerular Diseases in Dogs.Complement in Non-Antibody-Mediated Kidney Diseases.Clinical Significance of IgM and C3 Glomerular Deposition in Primary Focal Segmental Glomerulosclerosis IgM in the kidney: a multiple personality disorder.The impact of IgM deposits on the outcome of Nephrotic syndrome in children.IgM nephropathy: timely response to a call for action.Differentiating Primary, Genetic, and Secondary FSGS in Adults: A Clinicopathologic Approach.Complement factor H protects mice from ischemic acute kidney injury but is not critical for controlling complement activation by glomerular IgM.Complete B Cell Deficiency Reduces Allograft Inflammation and Intragraft Macrophages in a Rat Kidney Transplant Model.Investigational drugs in development for focal segmental glomerulosclerosis.Complement Activation in Patients With Diabetic Nephropathy.

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P2860

IgM contributes to glomerular injury in FSGS

http://www.wikidata.org/entity/Q36638347

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

2013年论文

@zh

2013年论文

@zh-cn

name

IgM contributes to glomerular injury in FSGS

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IgM contributes to glomerular injury in FSGS

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type

label

IgM contributes to glomerular injury in FSGS

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IgM contributes to glomerular injury in FSGS

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prefLabel

IgM contributes to glomerular injury in FSGS

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IgM contributes to glomerular injury in FSGS

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P1433

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P1433

Journal of the American Society of Nephrology

P1476

IgM contributes to glomerular injury in FSGS

@en

P2093

Brandon Renner

http://www.w3.org/2001/XMLSchema#string

Danica Ljubanović

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Derek Strassheim

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Joshua M Thurman

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Liudmila Kulik

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Richard Fuquay

http://www.w3.org/2001/XMLSchema#string

Sarah Panzer

http://www.w3.org/2001/XMLSchema#string

V Michael Holers

http://www.w3.org/2001/XMLSchema#string

P2860

Progenitors for Ly-1 B cells are distinct from progenitors for other B cells

Rituximab targets podocytes in recurrent focal segmental glomerulosclerosis.

Progressive adriamycin nephropathy in mice: sequence of histologic and immunohistochemical events.

Depletion of CD4(+) T cells aggravates glomerular and interstitial injury in murine adriamycin nephropathy.

Pathogenic natural antibodies recognizing annexin IV are required to develop intestinal ischemia-reperfusion injury.

IgM associated primary diffuse mesangial proliferative glomerulonephritis.

The actin cytoskeleton of kidney podocytes is a direct target of the antiproteinuric effect of cyclosporine A.

Blockade of self-reactive IgM significantly reduces injury in a murine model of acute myocardial infarction

The double life of a B-1 cell: self-reactivity selects for protective effector functions.

Efficacy and safety of treatment with rituximab for difficult steroid-resistant and -dependent nephrotic syndrome: multicentric report.

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393-406

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scholarly article

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10.1681/ASN.2012020187

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3

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24

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2013-02-07T00:00:00Z

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23393315

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3582199

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