The estrogen receptor-α is required and sufficient to maintain physiological glucose uptake in the mouse heart.
about
An animal model with a cardiomyocyte-specific deletion of estrogen receptor alpha: functional, metabolic, and differential network analysisEstrogen receptors alpha (ERα) and beta (ERβ): subtype-selective ligands and clinical potential.Estrogen regulation of protein expression and signaling pathways in the heart.The Role of Estrogen and Estrogen Receptors on Cardiomyocytes: An Overview.The effects of oestrogens and their receptors on cardiometabolic health.Morphological aspects and expression of estrogen and progesterone receptors in the interdigital sinus in cyclic ewes.Reduced expression of CRHR2 and Sp-1 in myocardium of ovariectomized rats is improved by exercise training.Estrogenic Impact on Cardiac Ischemic/Reperfusion Injury.
P2860
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P2860
The estrogen receptor-α is required and sufficient to maintain physiological glucose uptake in the mouse heart.
description
2012 nî lūn-bûn
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年论文
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name
The estrogen receptor-α is req ...... ose uptake in the mouse heart.
@ast
The estrogen receptor-α is req ...... ose uptake in the mouse heart.
@en
type
label
The estrogen receptor-α is req ...... ose uptake in the mouse heart.
@ast
The estrogen receptor-α is req ...... ose uptake in the mouse heart.
@en
prefLabel
The estrogen receptor-α is req ...... ose uptake in the mouse heart.
@ast
The estrogen receptor-α is req ...... ose uptake in the mouse heart.
@en
P2093
P2860
P1433
P1476
The estrogen receptor-α is req ...... ose uptake in the mouse heart.
@en
P2093
Barbara Bayer
Franz R Kaiser
Ina Israel
Karl-Heinz Fritzemeier
Michael C Kreissl
Paula-Anahi Arias-Loza
Stefan Frantz
Susanne Kneitz
Theo Pelzer
P2860
P304
P356
10.1161/HYPERTENSIONAHA.111.190389
P407
P577
2012-08-14T00:00:00Z