Mifepristone Suppresses Basal Triple-Negative Breast Cancer Stem Cells by Down-regulating KLF5 Expression. - Wikidata - wikimedia - TriplyDB

Mifepristone Suppresses Basal Triple-Negative Breast Cancer Stem Cells by Down-regulating KLF5 Expression.

Mifepristone Suppresses Basal Triple-Negative Breast Cancer Stem Cells by Down-regulating KLF5 Expression.

http://www.wikidata.org/entity/Q36644338

about

Metformin suppresses triple-negative breast cancer stem cells by targeting KLF5 for degradation.miR-217 inhibits triple-negative breast cancer cell growth, migration, and invasion through targeting KLF5 Dexamethasone induces docetaxel and cisplatin resistance partially through up-regulating Krüppel-like factor 5 in triple-negative breast cancer.YD277 Suppresses Triple-Negative Breast Cancer Partially Through Activating the Endoplasmic Reticulum Stress Pathway.Circular RNA profile indicates circular RNA VRK1 is negatively related with breast cancer stem cells.Mithramycin A suppresses basal triple-negative breast cancer cell survival partially via down-regulating Krüppel-like factor 5 transcription by Sp1.Hypoxia induces miR-153 through the IRE1α-XBP1 pathway to fine tune the HIF1α/VEGFA axis in breast cancer angiogenesis.Involvement of breast cancer stem cells in tumor angiogenesis.HDAC-mediated deacetylation of KLF5 associates with its proteasomal degradation.STARD13-correlated ceRNA network-directed inhibition on YAP/TAZ activity suppresses stemness of breast cancer via co-regulating Hippo and Rho-GTPase/F-actin signaling.TTK promotes mesenchymal signaling via multiple mechanisms in triple negative breast cancer Mifepristone inhibits proliferation, migration and invasion of HUUA cells and promotes its apoptosis by regulation of FAK and PI3K/AKT signaling pathway

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Mifepristone Suppresses Basal Triple-Negative Breast Cancer Stem Cells by Down-regulating KLF5 Expression.

http://www.wikidata.org/entity/Q36644338

description

2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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name

Mifepristone Suppresses Basal ...... wn-regulating KLF5 Expression.

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Mifepristone Suppresses Basal ...... wn-regulating KLF5 Expression.

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Mifepristone Suppresses Basal ...... wn-regulating KLF5 Expression.

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Mifepristone Suppresses Basal ...... wn-regulating KLF5 Expression.

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Mifepristone Suppresses Basal ...... wn-regulating KLF5 Expression.

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Mifepristone Suppresses Basal ...... wn-regulating KLF5 Expression.

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Mifepristone Suppresses Basal ...... wn-regulating KLF5 Expression.

@en

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Ceshi Chen

http://www.w3.org/2001/XMLSchema#string

Chao Dong

http://www.w3.org/2001/XMLSchema#string

Haijun Chen

http://www.w3.org/2001/XMLSchema#string

Huichun Liang

http://www.w3.org/2001/XMLSchema#string

Peiguo Shi

http://www.w3.org/2001/XMLSchema#string

Rong Liu

http://www.w3.org/2001/XMLSchema#string

Runxiang Yang

http://www.w3.org/2001/XMLSchema#string

Suling Liu

http://www.w3.org/2001/XMLSchema#string

Wenlin Chen

http://www.w3.org/2001/XMLSchema#string

Zhi Nie

http://www.w3.org/2001/XMLSchema#string

P2860

The Hippo transducer TAZ confers cancer stem cell-related traits on breast cancer cells

The role of PTEN/Akt/PI3K signaling in the maintenance and viability of prostate cancer stem-like cell populations

ALDH1 is a marker of normal and malignant human mammary stem cells and a predictor of poor clinical outcome

Prospective identification of tumorigenic breast cancer cells

Risks and benefits of estrogen plus progestin in healthy postmenopausal women: principal results From the Women's Health Initiative randomized controlled trial

Antiandrogen effects of mifepristone on coactivator and corepressor interactions with the androgen receptor

Identification of selective inhibitors of cancer stem cells by high-throughput screening

An embryonic stem cell-like gene expression signature in poorly differentiated aggressive human tumors

Identification of human triple-negative breast cancer subtypes and preclinical models for selection of targeted therapies

The induction of KLF5 transcription factor by progesterone contributes to progesterone-induced breast cancer cell proliferation and dedifferentiation.

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533-544

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scholarly article

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10.7150/THNO.14315

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4

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6

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2016-02-13T00:00:00Z

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26941846

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4775863

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