Clinical aspects and physiopathology of Brugada syndrome: review of current concepts.
about
Fluoxetine blocks Nav1.5 channels via a mechanism similar to that of class 1 antiarrhythmics.Gating pore current is a novel biophysical defect of Nav1.5 mutations associated with unusual cardiac arrhythmias and dilation.A New Cardiac Channelopathy: From Clinical Phenotypes to Molecular Mechanisms Associated With Na1.5 Gating Pores
P2860
Clinical aspects and physiopathology of Brugada syndrome: review of current concepts.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
2006年论文
@zh
2006年论文
@zh-cn
name
Clinical aspects and physiopathology of Brugada syndrome: review of current concepts.
@ast
Clinical aspects and physiopathology of Brugada syndrome: review of current concepts.
@en
type
label
Clinical aspects and physiopathology of Brugada syndrome: review of current concepts.
@ast
Clinical aspects and physiopathology of Brugada syndrome: review of current concepts.
@en
prefLabel
Clinical aspects and physiopathology of Brugada syndrome: review of current concepts.
@ast
Clinical aspects and physiopathology of Brugada syndrome: review of current concepts.
@en
P2860
P356
P1476
Clinical aspects and physiopathology of Brugada syndrome: review of current concepts.
@en
P2093
P2860
P304
P356
10.1139/Y06-038
P577
2006-08-01T00:00:00Z