Loss of Gli3 enhances the viability of embryonic telencephalic cells in vitro.
about
Mouse hitchhiker mutants have spina bifida, dorso-ventral patterning defects and polydactyly: identification of Tulp3 as a novel negative regulator of the Sonic hedgehog pathwayThe transcription factor Foxg1 regulates the competence of telencephalic cells to adopt subpallial fates in mice.Wnt/β-catenin signaling is disrupted in the extra-toes (Gli3(Xt/Xt) ) mutant from early stages of forebrain development, concomitant with anterior neural plate patterning defects.
P2860
Loss of Gli3 enhances the viability of embryonic telencephalic cells in vitro.
description
2005 nî lūn-bûn
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2005年の論文
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name
Loss of Gli3 enhances the viability of embryonic telencephalic cells in vitro.
@ast
Loss of Gli3 enhances the viability of embryonic telencephalic cells in vitro.
@en
type
label
Loss of Gli3 enhances the viability of embryonic telencephalic cells in vitro.
@ast
Loss of Gli3 enhances the viability of embryonic telencephalic cells in vitro.
@en
prefLabel
Loss of Gli3 enhances the viability of embryonic telencephalic cells in vitro.
@ast
Loss of Gli3 enhances the viability of embryonic telencephalic cells in vitro.
@en
P2093
P2860
P1476
Loss of Gli3 enhances the viability of embryonic telencephalic cells in vitro
@en
P2093
Ben Martynoga
Paulette A Zaki
Vassiliki Fotaki
P2860
P304
P356
10.1111/J.1460-9568.2005.04323.X
P407
P577
2005-09-01T00:00:00Z