Hantaviruses direct endothelial cell permeability by sensitizing cells to the vascular permeability factor VEGF, while angiopoietin 1 and sphingosine 1-phosphate inhibit hantavirus-directed permeability.
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Identification of a putative Crimean-Congo hemorrhagic fever virus entry factorDepletion of Alveolar Macrophages Does Not Prevent Hantavirus Disease Pathogenesis in Golden Syrian HamstersWhat Do We Know about How Hantaviruses Interact with Their Different Hosts?Hemorrhagic Fever with Renal Syndrome: Pathogenesis and Clinical PictureProfile of time-dependent VEGF upregulation in human pulmonary endothelial cells, HPMEC-ST1.6R infected with DENV-1, -2, -3, and -4 virusesHantavirus protein interactions regulate cellular functions and signaling responses.Pathogenic hantaviruses direct the adherence of quiescent platelets to infected endothelial cells.Hantavirus infections for the clinician: from case presentation to diagnosis and treatment.Hypoxia induces permeability and giant cell responses of Andes virus-infected pulmonary endothelial cells by activating the mTOR-S6K signaling pathway.Vascular events in viral hemorrhagic fevers: a comparative study of dengue and hantaviruses.Endothelial cell dysfunction in viral hemorrhage and edema.Andes virus recognition of human and Syrian hamster beta3 integrins is determined by an L33P substitution in the PSI domain.Hantavirus interferon regulation and virulence determinantsHantavirusesZika Virus Persistently Infects and Is Basolaterally Released from Primary Human Brain Microvascular Endothelial Cells.Pathogenic hantaviruses Andes virus and Hantaan virus induce adherens junction disassembly by directing vascular endothelial cadherin internalization in human endothelial cellsHantaviruses induce cell type- and viral species-specific host microRNA expression signatures.Hantaviruses in the americas and their role as emerging pathogensAndes virus disrupts the endothelial cell barrier by induction of vascular endothelial growth factor and downregulation of VE-cadherin.Sangassou virus, the first hantavirus isolate from Africa, displays genetic and functional properties distinct from those of other murinae-associated hantaviruses.Andes virus regulation of cellular microRNAs contributes to hantavirus-induced endothelial cell permeability.Virus interactions with endothelial cell receptors: implications for viral pathogenesis.Hantavirus-infection confers resistance to cytotoxic lymphocyte-mediated apoptosis.VEGFR2 and Src kinase inhibitors suppress Andes virus-induced endothelial cell permeabilityCompartmentalized, functional role of angiogenin during spotted fever group rickettsia-induced endothelial barrier dysfunction: evidence of possible mediation by host tRNA-derived small noncoding RNAs.Endothelial cell permeability during hantavirus infection involves factor XII-dependent increased activation of the kallikrein-kinin system.The C-terminal 42 residues of the Tula virus Gn protein regulate interferon induction.Involvement of the Akt/NF-κB pathways in the HTNV-mediated increase of IL-6, CCL5, ICAM-1, and VCAM-1 in HUVECs.Elevated cytokines, thrombin and PAI-1 in severe HCPS patients due to Sin Nombre virus.Productive dengue virus infection of human endothelial cells is directed by heparan sulfate-containing proteoglycan receptors.Endothelial Nitric Oxide Synthase G894T Polymorphism Associates with Disease Severity in Puumala Hantavirus Infection.Vascular endothelial hyperpermeability induces the clinical symptoms of Clarkson disease (the systemic capillary leak syndrome).Andes virus infection of lymphatic endothelial cells causes giant cell and enhanced permeability responses that are rapamycin and vascular endothelial growth factor C sensitive.Hantavirus regulation of type I interferon responses.Elevated VEGF Levels in Pulmonary Edema Fluid and PBMCs from Patients with Acute Hantavirus Pulmonary Syndrome.Dengue Virus-Induced Inflammation of the Endothelium and the Potential Roles of Sphingosine Kinase-1 and MicroRNAs.Lysophosphatidic Acid and Sphingosine-1-Phosphate: A Concise Review of Biological Function and Applications for Tissue Engineering.Sustained high level of serum VEGF at convalescent stage contributes to the renal recovery after HTNV infection in patients with hemorrhagic fever with renal syndrome.Seoul virus-infected rat lung endothelial cells and alveolar macrophages differ in their ability to support virus replication and induce regulatory T cell phenotypes.Gene control of tyrosine kinase TIE2 and vascular manifestations of infections.
P2860
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P2860
Hantaviruses direct endothelial cell permeability by sensitizing cells to the vascular permeability factor VEGF, while angiopoietin 1 and sphingosine 1-phosphate inhibit hantavirus-directed permeability.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Hantaviruses direct endothelia ...... tavirus-directed permeability.
@ast
Hantaviruses direct endothelia ...... tavirus-directed permeability.
@en
type
label
Hantaviruses direct endothelia ...... tavirus-directed permeability.
@ast
Hantaviruses direct endothelia ...... tavirus-directed permeability.
@en
prefLabel
Hantaviruses direct endothelia ...... tavirus-directed permeability.
@ast
Hantaviruses direct endothelia ...... tavirus-directed permeability.
@en
P2093
P2860
P356
P1433
P1476
Hantaviruses direct endothelia ...... tavirus-directed permeability.
@en
P2093
Elena E Gorbunova
Erich R Mackow
Irina N Gavrilovskaya
Natalie A Mackow
P2860
P304
P356
10.1128/JVI.02397-07
P407
P577
2008-03-26T00:00:00Z