An increase in herpes simplex virus type 1 in the anterior segment of the eye is linked to a deficiency in NK cell infiltration in mice deficient in CXCR3
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Evidence of molecular evolution driven by recombination events influencing tropism in a novel human adenovirus that causes epidemic keratoconjunctivitisDistinct populations of innate CD8+ T cells revealed in a CXCR3 reporter mouse.Chemokine CXCL1/KC and its receptor CXCR2 are responsible for neutrophil chemotaxis in adenoviral keratitis.Herpes keratitis.Chemokine receptor CXCR3 promotes growth of glioma.Enhanced resistance of CXCR3 deficient mice to ocular HSV-1 infection is due to control of replication in the brain ependymaCirculating herpes simplex type 1 (HSV-1)-specific CD8+ T cells do not access HSV-1 latently infected trigeminal ganglia.Resident Corneal Cells Communicate with Neutrophils Leading to the Production of IP-10 during the Primary Inflammatory Response to HSV-1 Infection.CD8+ T cells suppress viral replication in the cornea but contribute to VEGF-C-induced lymphatic vessel genesis.Comparison of the host immune response to herpes simplex virus 1 (HSV-1) and HSV-2 at two different mucosal sites.Dysregulation of CXCR3 signaling due to CXCL10 deficiency impairs the antiviral response to herpes simplex virus 1 infection.Interferon γ-induced intratumoral expression of CXCL9 alters the local distribution of T cells following immunotherapy with Listeria monocytogenesHSV-1 targets lymphatic vessels in the eye and draining lymph node of mice leading to edema in the absence of a functional type I interferon response.Dynamic change in natural killer cell type in the human ocular mucosa in situ as means of immune evasion by adenovirus infection.CXCR3 deficiency enhances tumor progression by promoting macrophage M2 polarization in a murine breast cancer model.Natural killer cell dysfunction during acute infection with foot-and-mouth disease virus.Flagellin-induced expression of CXCL10 mediates direct fungal killing and recruitment of NK cells to the cornea in response to Candida albicans infection.Co-infection with Chikungunya virus alters trafficking of pathogenic CD8(+) T cells into the brain and prevents Plasmodium-induced neuropathology.Optimal CD4 T cell priming after LPS-based adjuvanticity with CD134 costimulation relies on CXCL9 production.
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P2860
An increase in herpes simplex virus type 1 in the anterior segment of the eye is linked to a deficiency in NK cell infiltration in mice deficient in CXCR3
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
An increase in herpes simplex ...... ion in mice deficient in CXCR3
@ast
An increase in herpes simplex ...... ion in mice deficient in CXCR3
@en
type
label
An increase in herpes simplex ...... ion in mice deficient in CXCR3
@ast
An increase in herpes simplex ...... ion in mice deficient in CXCR3
@en
prefLabel
An increase in herpes simplex ...... ion in mice deficient in CXCR3
@ast
An increase in herpes simplex ...... ion in mice deficient in CXCR3
@en
P2860
P356
P1476
An increase in herpes simplex ...... ion in mice deficient in CXCR3
@en
P2093
Daniel J J Carr
Todd Wuest
P2860
P304
P356
10.1089/JIR.2007.0110
P50
P577
2008-04-01T00:00:00Z