Protective role of nuclear factor erythroid 2-related factor 2 in the hemorrhagic shock-induced inflammatory response

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Preconditioning by Low Dose LPS Prevents Subsequent LPS-Induced Severe Liver Injury via Nrf2 Activation in Mice.Haematoma scavenging in intracerebral haemorrhage: from mechanisms to the clinic.The -Antioxidant Response Element Signaling Pathway Controls Fibrosis and Autoimmunity in Scleroderma

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Protective role of nuclear factor erythroid 2-related factor 2 in the hemorrhagic shock-induced inflammatory response

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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Protective role of nuclear fac ...... -induced inflammatory response

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Protective role of nuclear fac ...... -induced inflammatory response

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Protective role of nuclear fac ...... -induced inflammatory response

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Protective role of nuclear fac ...... -induced inflammatory response

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Protective role of nuclear fac ...... -induced inflammatory response

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Protective role of nuclear fac ...... -induced inflammatory response

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Protective role of nuclear fac ...... -induced inflammatory response

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Haige Zhao

http://www.w3.org/2001/XMLSchema#string

Hongfei Xu

http://www.w3.org/2001/XMLSchema#string

Liang Ma

http://www.w3.org/2001/XMLSchema#string

Luyang Yu

http://www.w3.org/2001/XMLSchema#string

Sijing Hao

http://www.w3.org/2001/XMLSchema#string

Yiming Ni

http://www.w3.org/2001/XMLSchema#string

Zheng Zhang

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Clinical review: hemorrhagic shock

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

HMG-1 as a late mediator of endotoxin lethality in mice

HMGB1 as a DNA-binding cytokine

An Nrf2/small Maf heterodimer mediates the induction of phase II detoxifying enzyme genes through antioxidant response elements

Cell survival responses to environmental stresses via the Keap1-Nrf2-ARE pathway

Organ dysfunction following hemorrhage and sepsis: mechanisms and therapeutic approaches (Review).

Systemic inflammation and liver injury following hemorrhagic shock and peripheral tissue trauma involve functional TLR9 signaling on bone marrow-derived cells and parenchymal cells.

Nrf2 is a critical regulator of the innate immune response and survival during experimental sepsis.

Reversing established sepsis with antagonists of endogenous high-mobility group box 1.

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1014-1022

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scholarly article

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10.3892/IJMM.2016.2507

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2016-02-25T00:00:00Z

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297590173

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26935388

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4790708

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Protective role of nuclear factor erythroid 2-related factor 2 in the hemorrhagic shock-induced inflammatory response

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Protective role of nuclear factor erythroid 2-related factor 2 in the hemorrhagic shock-induced inflammatory response

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