WIN 52035-2 inhibits both attachment and eclipse of human rhinovirus 14
about
Antibodies to the buried N terminus of rhinovirus VP4 exhibit cross-serotypic neutralizationInteraction of poliovirus with its receptor affords a high level of infectivity to the virion in poliovirus infections mediated by the Fc receptorDistribution of drug resistance mutations in type 3 poliovirus identifies three regions involved in uncoating functionsRelationship of pleconaril susceptibility and clinical outcomes in treatment of common colds caused by rhinoviruses.VP1 sequencing of all human rhinovirus serotypes: insights into genus phylogeny and susceptibility to antiviral capsid-binding compounds.Pocket factors are unlikely to play a major role in the life cycle of human rhinovirus.Mutations in the 2C region of poliovirus responsible for altered sensitivity to benzimidazole derivatives.WIN 52035-dependent human rhinovirus 16: assembly deficiency caused by mutations near the canyon surface.An RNA replication-center assay for high content image-based quantifications of human rhinovirus and coxsackievirus infectionsNectin-like interactions between poliovirus and its receptor trigger conformational changes associated with cell entry.The cardiovirulent phenotype of coxsackievirus B3 is determined at a single site in the genomic 5' nontranslated region.Viral cell recognition and entry.Potent antiviral agents fail to elicit genetically-stable resistance mutations in either enterovirus 71 or Coxsackievirus A16.Surveillance of community-acquired viral infections due to respiratory viruses in Rhone-Alpes (France) during winter 1994 to 1995.Increasing Type 1 Poliovirus Capsid Stability by Thermal Selection.Soluble receptor-resistant poliovirus mutants identify surface and internal capsid residues that control interaction with the cell receptorHuman rhinovirus type 16: mutant V1210A requires capsid-binding drug for assembly of pentamers to form virions during morphogenesis.The antiviral compound enviroxime targets the 3A coding region of rhinovirus and poliovirus.Poliovirus 2C region functions during encapsidation of viral RNA.Structural and virological studies of the stages of virus replication that are affected by antirhinovirus compounds.Human major group rhinoviruses downmodulate the accessory function of monocytes by inducing IL-10.VP4 protein from human rhinovirus 14 is released by pressure and locked in the capsid by the antiviral compound WIN.
P2860
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P2860
WIN 52035-2 inhibits both attachment and eclipse of human rhinovirus 14
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
WIN 52035-2 inhibits both attachment and eclipse of human rhinovirus 14
@ast
WIN 52035-2 inhibits both attachment and eclipse of human rhinovirus 14
@en
type
label
WIN 52035-2 inhibits both attachment and eclipse of human rhinovirus 14
@ast
WIN 52035-2 inhibits both attachment and eclipse of human rhinovirus 14
@en
prefLabel
WIN 52035-2 inhibits both attachment and eclipse of human rhinovirus 14
@ast
WIN 52035-2 inhibits both attachment and eclipse of human rhinovirus 14
@en
P2093
P2860
P1433
P1476
WIN 52035-2 inhibits both attachment and eclipse of human rhinovirus 14
@en
P2093
D A Shepard
R R Rueckert
P2860
P304
P407
P577
1993-04-01T00:00:00Z